Page last updated: 2024-08-22

diallyl disulfide and Esophageal Neoplasms

diallyl disulfide has been researched along with Esophageal Neoplasms in 4 studies

Research

Studies (4)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's1 (25.00)29.6817
2010's3 (75.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Feng, C; Gao, X; Han, L; Jia, L; Jiao, Y; Jing, F; Ma, Y; Wang, J; Yin, X; Zhang, J; Zhang, Y1
Cai, C; Feng, C; Jing, F; Li, X; Liang, R; Liu, D; Liu, X; Ma, H; Wang, J; Wang, X; Xue, X; Yin, X; Zhang, J; Zhuang, K1
Feng, C; Li, Z; Liu, D; Liu, X; Ma, H; Wang, X; Xu, K; Yin, X; Zhang, J; Zhang, R; Zhang, S1
Chen, SC; Chung, JG; Lai, WW; Lin, JP; Yu, CS; Yu, FS1

Other Studies

4 other study(ies) available for diallyl disulfide and Esophageal Neoplasms

ArticleYear
Diallyl disulfide inhibits the metastasis of type Ⅱ esophageal‑gastric junction adenocarcinoma cells via NF-κB and PI3K/AKT signaling pathways in vitro.
    Oncology reports, 2018, Volume: 39, Issue:2

    Topics: Adenocarcinoma; Allyl Compounds; Anticarcinogenic Agents; Cell Line, Tumor; Cell Movement; Cell Survival; Disulfides; Esophageal Neoplasms; Esophagogastric Junction; Gene Expression Regulation, Neoplastic; Humans; In Vitro Techniques; Matrix Metalloproteinases; Neoplasm Metastasis; NF-kappa B; Phosphatidylinositol 3-Kinases; Proto-Oncogene Proteins c-akt; Signal Transduction; Tissue Inhibitor of Metalloproteinases

2018
DADS suppresses human esophageal xenograft tumors through RAF/MEK/ERK and mitochondria-dependent pathways.
    International journal of molecular sciences, 2014, Jul-14, Volume: 15, Issue:7

    Topics: Allyl Compounds; Animals; Antineoplastic Agents; Apoptosis; bcl-2-Associated X Protein; Carcinoma; Caspase 3; Cell Line, Tumor; Disulfides; Esophageal Neoplasms; Humans; MAP Kinase Signaling System; Mice; Mice, Nude; Mitochondria; Proliferating Cell Nuclear Antigen; Proto-Oncogene Proteins c-bcl-2; Tumor Suppressor Protein p53; Xenograft Model Antitumor Assays

2014
Diallyl disulfide induces G2/M arrest and promotes apoptosis through the p53/p21 and MEK-ERK pathways in human esophageal squamous cell carcinoma.
    Oncology reports, 2014, Volume: 32, Issue:4

    Topics: Allyl Compounds; Antineoplastic Agents; Apoptosis; Carcinoma, Squamous Cell; CDC2 Protein Kinase; cdc25 Phosphatases; Cell Line, Tumor; Cell Survival; Cyclin B1; Cyclin-Dependent Kinases; Disulfides; Drug Screening Assays, Antitumor; Esophageal Neoplasms; Esophageal Squamous Cell Carcinoma; G2 Phase Cell Cycle Checkpoints; Gene Expression Regulation, Neoplastic; Humans; MAP Kinase Signaling System; rho GTP-Binding Proteins; RNA, Messenger; Signal Transduction; Tumor Suppressor Protein p53

2014
Diallyl disulfide inhibits N-acetyltransferase activity and gene expression in human esophagus epidermoid carcinoma CE 81T/VGH cells.
    Food and chemical toxicology : an international journal published for the British Industrial Biological Research Association, 2005, Volume: 43, Issue:7

    Topics: Acetylation; Acetyltransferases; Allyl Compounds; Animals; Anticarcinogenic Agents; Carcinoma, Squamous Cell; Cell Line, Tumor; Disulfides; Dose-Response Relationship, Drug; Enzyme Inhibitors; Esophageal Neoplasms; Female; Flow Cytometry; Humans; Mice; Mice, Inbred BALB C; Oligonucleotide Array Sequence Analysis; Reverse Transcriptase Polymerase Chain Reaction; RNA, Messenger; RNA, Neoplasm

2005