diallyl disulfide has been researched along with ER-Negative PR-Negative HER2-Negative Breast Cancer in 4 studies
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 0 (0.00) | 18.2507 |
2000's | 0 (0.00) | 29.6817 |
2010's | 3 (75.00) | 24.3611 |
2020's | 1 (25.00) | 2.80 |
Authors | Studies |
---|---|
Chakraborti, A; Kundrapu, DB; Malla, R; Marni, R | 1 |
Chintamaneni, PK; Nandha Kumar, S; Pindiprolu, SKSS; Siddhartha, VT; Tummala, S | 1 |
Bauer, D; Darling-Reed, S; Mazzio, E; Oriaku, E; Soliman, KF; Taka, E; Womble, T | 1 |
Huang, J; Li, H; Peng, W; Qiu, Z; Ren, G; Wan, J; Xiang, T; Yang, B; Zhang, L | 1 |
4 other study(ies) available for diallyl disulfide and ER-Negative PR-Negative HER2-Negative Breast Cancer
Article | Year |
---|---|
Insight into drug sensitizing effect of diallyl disulfide and diallyl trisulfide from Allium sativum L. on paclitaxel-resistant triple-negative breast cancer cells.
Topics: Allyl Compounds; Antioxidants; Apoptosis; Cell Line, Tumor; Disulfides; Garlic; Humans; Paclitaxel; Reactive Oxygen Species; Sulfides; Triple Negative Breast Neoplasms | 2022 |
RAGE receptor targeted bioconjuguate lipid nanoparticles of diallyl disulfide for improved apoptotic activity in triple negative breast cancer: in vitro studies.
Topics: Allyl Compounds; Antineoplastic Agents; Apoptosis; Biological Transport; Cell Line, Tumor; Cell Survival; Disulfides; Drug Liberation; Gene Expression Regulation, Neoplastic; Humans; Lipids; Nanoparticles; Receptor for Advanced Glycation End Products; Triple Negative Breast Neoplasms | 2018 |
Diallyl disulfide inhibits TNFα-induced CCL2 release by MDA-MB-231 cells.
Topics: Adipokines; Allyl Compounds; Antineoplastic Agents; Apoptosis; Biomarkers, Tumor; Cell Proliferation; Chemokine CCL2; Disulfides; Enzyme-Linked Immunosorbent Assay; Female; Garlic; Humans; Protein Array Analysis; Triple Negative Breast Neoplasms; Tumor Cells, Cultured; Tumor Necrosis Factor-alpha | 2014 |
Diallyl disulfide inhibits growth and metastatic potential of human triple-negative breast cancer cells through inactivation of the β-catenin signaling pathway.
Topics: Allyl Compounds; Animals; Antineoplastic Agents; Apoptosis; bcl-2-Associated X Protein; beta Catenin; Cell Line, Tumor; Cell Proliferation; Cell Survival; Disulfides; Down-Regulation; Epithelial-Mesenchymal Transition; Female; Garlic; Gene Expression Regulation, Neoplastic; Humans; Matrix Metalloproteinase 9; Mice; Signal Transduction; Triple Negative Breast Neoplasms; Xenograft Model Antitumor Assays | 2015 |