Compounds > diallyl disulfide
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diallyl disulfide and Colorectal Neoplasms

diallyl disulfide has been researched along with Colorectal Neoplasms in 5 studies

Research

Studies (5)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's2 (40.00)29.6817
2010's3 (60.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Colburn, NH; Gray, Z; Kim, YS; Li, W; Matter, MS; Saud, SM; Young, MR1
Altonsy, MO; Andrews, SC1
Liao, Q; Shi, L; Su, J; Su, Q; Zhou, Y1
Alston-Mills, B; Bottone, FG; Eling, TE; Ishibashi, M; Kim, JS; Moon, Y1
Baek, SJ; Bottone, FG; Eling, TE; Nixon, JB1

Other Studies

5 other study(ies) available for diallyl disulfide and Colorectal Neoplasms

ArticleYear
Diallyl Disulfide (DADS), a Constituent of Garlic, Inactivates NF-κB and Prevents Colitis-Induced Colorectal Cancer by Inhibiting GSK-3β.
    Cancer prevention research (Philadelphia, Pa.), 2016, Volume: 9, Issue:7

    Topics: Allyl Compounds; Animals; Antineoplastic Agents, Phytogenic; Carcinogenesis; Cell Line, Tumor; Cell Proliferation; Colitis; Colorectal Neoplasms; Disulfides; Garlic; Glycogen Synthase Kinase 3 beta; Humans; Inflammation; Mice; NF-kappa B; Plant Extracts

2016
Diallyl disulphide, a beneficial component of garlic oil, causes a redistribution of cell-cycle growth phases, induces apoptosis, and enhances butyrate-induced apoptosis in colorectal adenocarcinoma cells (HT-29).
    Nutrition and cancer, 2011, Volume: 63, Issue:7

    Topics: Allyl Compounds; Anticarcinogenic Agents; Antineoplastic Combined Chemotherapy Protocols; Apoptosis; Blotting, Western; Butyrates; Caspase 3; Caspase 9; Cell Proliferation; Colorectal Neoplasms; Disulfides; DNA Fragmentation; Fatty Acids, Volatile; Garlic; HT29 Cells; Humans; Plant Oils; Polysaccharides

2011
DADS downregulates the Rac1-ROCK1/PAK1-LIMK1-ADF/cofilin signaling pathway, inhibiting cell migration and invasion.
    Oncology reports, 2013, Volume: 29, Issue:2

    Topics: Allyl Compounds; Cell Line, Tumor; Cell Movement; Cofilin 1; Colorectal Neoplasms; Destrin; Disulfides; Down-Regulation; Gene Expression Regulation, Neoplastic; Gene Silencing; Humans; Lim Kinases; MicroRNAs; Neoplasm Invasiveness; p21-Activated Kinases; rac1 GTP-Binding Protein; rho-Associated Kinases; RNA, Messenger; Signal Transduction

2013
The anti-invasive activity of cyclooxygenase inhibitors is regulated by the transcription factor ATF3 (activating transcription factor 3).
    Molecular cancer therapeutics, 2005, Volume: 4, Issue:5

    Topics: Activating Transcription Factor 3; Allyl Compounds; Animals; Anti-Inflammatory Agents, Non-Steroidal; Apoptosis; Chromans; Colorectal Neoplasms; Cyclooxygenase Inhibitors; Disulfides; Gene Expression Regulation, Enzymologic; Gene Expression Regulation, Neoplastic; HCT116 Cells; Humans; Male; Mice; Mice, Nude; Microarray Analysis; Neoplasm Invasiveness; Resveratrol; RNA, Messenger; Stilbenes; Sulindac; Thiazolidinediones; Transcription Factors; Transplantation, Heterologous; Troglitazone; Up-Regulation

2005
Diallyl disulfide (DADS) induces the antitumorigenic NSAID-activated gene (NAG-1) by a p53-dependent mechanism in human colorectal HCT 116 cells.
    The Journal of nutrition, 2002, Volume: 132, Issue:4

    Topics: Allyl Compounds; Antineoplastic Agents; Apoptosis; Colorectal Neoplasms; Cytokines; Disulfides; Growth Differentiation Factor 15; Humans; Tumor Cells, Cultured; Tumor Suppressor Protein p53

2002