diallyl disulfide has been researched along with Carcinoma, Anaplastic in 3 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 2 (66.67) | 29.6817 |
| 2010's | 1 (33.33) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Cai, C; Feng, C; Jing, F; Li, X; Liang, R; Liu, D; Liu, X; Ma, H; Wang, J; Wang, X; Xue, X; Yin, X; Zhang, J; Zhuang, K | 1 |
| Kassie, F; Mersch-Sundermann, V; Wu, XJ | 1 |
| Li, GC; Talbot, SG; Wen, J; Xiao, JB; Xu, M; Zhang, YW | 1 |
3 other study(ies) available for diallyl disulfide and Carcinoma, Anaplastic
| Article | Year |
|---|---|
DADS suppresses human esophageal xenograft tumors through RAF/MEK/ERK and mitochondria-dependent pathways.
Topics: Allyl Compounds; Animals; Antineoplastic Agents; Apoptosis; bcl-2-Associated X Protein; Carcinoma; Caspase 3; Cell Line, Tumor; Disulfides; Esophageal Neoplasms; Humans; MAP Kinase Signaling System; Mice; Mice, Nude; Mitochondria; Proliferating Cell Nuclear Antigen; Proto-Oncogene Proteins c-bcl-2; Tumor Suppressor Protein p53; Xenograft Model Antitumor Assays | 2014 |
The role of reactive oxygen species (ROS) production on diallyl disulfide (DADS) induced apoptosis and cell cycle arrest in human A549 lung carcinoma cells.
Topics: Acetylcysteine; Allyl Compounds; Antineoplastic Agents; Antioxidants; Apoptosis; Carcinoma; Cell Cycle; Cell Proliferation; Cell Survival; Disulfides; Dose-Response Relationship, Drug; Humans; Lung Neoplasms; Reactive Oxygen Species; Tumor Cells, Cultured | 2005 |
Induction of apoptosis and transient increase of phosphorylated MAPKs by diallyl disulfide treatment in human nasopharyngeal carcinoma CNE2 cells.
Topics: Allyl Compounds; Apoptosis; Blotting, Western; Carcinoma; Cell Cycle; Cell Line, Tumor; Cell Survival; Disulfides; Garlic; Humans; Mitogen-Activated Protein Kinase 1; Nasopharyngeal Neoplasms; p38 Mitogen-Activated Protein Kinases; Phosphorylation; S Phase | 2006 |