diacetyldichlorofluorescein has been researched along with Amino Acid Metabolism Disorders, Inborn in 2 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 0 (0.00) | 29.6817 |
| 2010's | 2 (100.00) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Arend, J; Barbosa, S; do Nascimento, PS; Dos Santos, ARS; Fighera, MR; Furian, AF; Gabbi, P; Haupental, F; Nogueira, V; Oliveira, MS; Rodrigues, FS; Royes, LFF | 1 |
| Amaral, AU; de Souza, DO; Goodman, SI; Leipnitz, G; Rodrigues, MD; Seminotti, B; Wajner, M; Woontner, M | 1 |
2 other study(ies) available for diacetyldichlorofluorescein and Amino Acid Metabolism Disorders, Inborn
| Article | Year |
|---|---|
Ammonia role in glial dysfunction in methylmalonic acidemia.
Topics: Amino Acid Metabolism, Inborn Errors; Ammonia; Ammonium Chloride; Animals; Behavior, Animal; Brain; Cell Proliferation; Disease Models, Animal; Fluoresceins; Hyperammonemia; Interleukin-1beta; Male; Malonates; Maze Learning; Memory Disorders; Memory, Short-Term; Mice; Neuroglia; Quaternary Ammonium Compounds; Time Factors; Tumor Necrosis Factor-alpha | 2018 |
Experimental evidence that overexpression of NR2B glutamate receptor subunit is associated with brain vacuolation in adult glutaryl-CoA dehydrogenase deficient mice: A potential role for glutamatergic-induced excitotoxicity in GA I neuropathology.
Topics: Amino Acid Metabolism, Inborn Errors; Animals; Brain Diseases, Metabolic; Brain Injuries; Catalase; Disease Models, Animal; Excitatory Amino Acid Transporter 2; Fluoresceins; Gene Expression Regulation; Glucosephosphate Dehydrogenase; Glutaryl-CoA Dehydrogenase; Glutathione; Glutathione Peroxidase; Glutathione Reductase; Malondialdehyde; Mice; Mice, Transgenic; NAD; Receptors, N-Methyl-D-Aspartate; Sulfhydryl Compounds; Superoxide Dismutase | 2015 |