Page last updated: 2024-08-23

deoxynivalenol and Innate Inflammatory Response

deoxynivalenol has been researched along with Innate Inflammatory Response in 58 studies

Research

Studies (58)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's1 (1.72)18.2507
2000's3 (5.17)29.6817
2010's24 (41.38)24.3611
2020's30 (51.72)2.80

Authors

AuthorsStudies
Antonissen, G; Bouziotis, D; Fegeros, K; Griela, E; Mountzouris, KC; Paraskeuas, V1
Bao, W; Fan, H; Rehman, ZU; Ren, Z; Sun, MA; Wang, H; Wu, S; Wu, Z; Xu, C1
Chen, X; Ge, L; Guo, J; Hou, L; Huang, K; Liu, D; Liu, S; Mao, X1
Bracarense, APFRL; Bruel, S; Hasuda, AL; Khoshal, AK; Oswald, IP; Person, E; Pinton, P; Puel, S1
Bai, Y; Li, J; Ma, K; Ren, Z; Shan, A; Zhang, J1
Chen, S; Deng, Y; Huang, Q; Jiang, J; Li, J; Mao, X; Mu, P; Xie, X1
Bao, W; Wang, H; Wang, J; Wu, S; Xiao, Y1
Chen, X; Du, H; Ge, L; Huang, K; Liu, S; Mao, X; Zhang, P1
Ahmed, AA; Bao, W; Cai, D; Gu, F; Gu, H; Hu, P; Liu, HY; Liu, Y; Zhao, Y; Zong, Q1
Chen, H; Chen, X; Ma, J1
Ji, X; Tang, Z; Wu, D; Wu, Y; Zhang, F; Zhou, F1
Bao, W; Cai, D; Hu, P; Li, K; Liu, HY; Qu, H; Wang, H; Wang, S; Wu, S; Xu, C; Zong, Q1
Chen, Y; Fu, Y; Hong, X; Jiang, M; Sun, Y; Wang, J; Wang, X; Wu, H; Yang, Z; Ye, Y; Zhou, E1
Bakker, W; Bouwmeester, H; de Haan, L; Wang, J1
Chen, B; Fan, Q; Fang, H; Fu, Y; Jin, Y; Li, R; Shen, J; Wang, J; Wang, R; Wu, S; Yu, H; Zhang, J; Zhao, Y; Zheng, K; Zhou, C1
Cao, L; Chen, XF; Chu, XY; Feng, SB; Huang, YY; Li, Y; Ur Rahman, S; Wang, XC; Wu, JJ; Zhang, YF; Zhu, DF; Zhu, L1
Amiri, KMA; Co, VA; El-Nezami, H; Turner, PC; Wan, MLY; Wang, MF1
Cao, L; Chen, X; Feng, S; Huang, Y; Li, Y; Rahman, SU; Wang, X; Wu, J; Zhang, Y; Zhao, J; Zhu, L1
Abeln, H; Abia, W; Beisl, J; Del Favero, G; Ehling-Schulz, M; Ezekiel, CN; Marko, D; Pahlke, G; Sulyok, M; Varga, E; Warth, B1
Deng, X; Wu, W; Zhang, H; Zhou, C1
Chen, X; Li, M; Li, Y; Liao, P; Tang, M; Xu, K; Yuan, D1
Guo, F; Liang, S; Long, F; Ren, Z; Yang, X1
Chaturvedi, S; Dewangan, J; Divakar, A; Kumar, S; Mandal, P; Mishra, S; Rath, SK; Srivastava, S; Tripathi, A; Wahajuddin, M1
Cao, Z; Huang, W; Li, Y; Sun, Y1
Cui, Z; Li, J; Liao, S; Qi, M; Sun, P; Tan, B; Tang, S; Tang, Y; Wang, Y; Yin, Y; Zha, A1
Bannert, E; Bühler, S; Dänicke, S; Frahm, J; Görs, S; Kahlert, S; Kersten, S; Kluess, J; Metges, CC; Rothkötter, HJ; Sauerwein, H; Tesch, T1
Cao, L; Jia, R; Liu, W; Shen, Z; Zhao, L1
Gan, F; Ge, L; Hou, L; Huang, K; Le, G; Lin, Z; Liu, D; Liu, S; Mao, X1
Chang, J; Li, M; Liu, C; Lu, F; Song, A; Wang, P; Xu, X; Yin, Q; Zhu, Q1
Gan, F; Ge, L; Hou, L; Huang, K; Le, G; Lin, Z; Liu, S; Mao, X; Wen, L1
Cheng, YH; Hsiao, FS; Lai, YH; Yu, YH1
Liao, P; Tang, M; Yuan, D1
Duan, N; Lin, X; Meng, X; Qi, S; Wang, Z; Wu, S; Yuan, W; Zhang, Y; Zhou, Y1
Bai, Y; Bi, C; Li, J; Ma, K; Shan, A1
Bannert, E; Barta-Böszörményi, A; Dänicke, S; Frahm, J; Kahlert, S; Kersten, S; Kluess, J; Renner, L; Rothkötter, HJ; Schönfeld, P; Tesch, T1
Adesso, S; Autore, G; Marzocco, S; Popolo, A; Quaroni, A; Severino, L1
Du, Z; Kim, J; Kim, KH; Moon, Y1
Li, Y; Wang, J; Wu, H; Yu, Q; Zhao, S1
Chen, L; Hou, W; Yang, W; Yu, M1
Dai, P; Kang, R; Li, C; Li, R; Li, Y; Li, Z1
Chiers, K; Croubels, S; De Backer, P; Ducatelle, R; Hautekiet, V; Osselaere, A; Santos, R1
Airault, C; Barbouche, R; Dallaporta, M; Djelloul, M; Gaigé, S; Guillebaud, F; Tardivel, C; Troadec, JD1
Armand, L; Di Pasquale, E; Graziani, F; Maresca, M; Nicoletti, C; Oswald, IP; Perrier, J; Pinton, P; Pujol, A1
Belosevic, M; Burkhardt-Holm, P; Garcia-Garcia, E; Katzenback, BA; Pietsch, C; Schulz, C1
Akbari, P; Braber, S; Fink-Gremmels, J; Folkerts, G; Garssen, J; Kraneveld, AD; Verheijden, KA; Willemsen, LE1
Bannert, E; Dänicke, S; Frahm, J; Kahlert, S; Kersten, S; Kluess, J; Renner, L; Rothkötter, HJ; Tesch, T1
Armstrong, C; Aziz, SA; Bondy, GS; Caldwell, D; Coady, L; Curran, I; Gannon, AM; Liston, V; Mehta, R; Nunnikhoven, A; Shenton, J1
Bannert, E; Breves, G; Dänicke, S; Frahm, J; Hüther, L; Kahlert, S; Kersten, S; Kluess, J; Renner, L; Rothkötter, HJ; Tesch, T1
Amuzie, CJ; Pestka, JJ1
During, A; Larondelle, Y; Piront, N; Schneider, YJ; Toussaint, O; Van De Walle, J1
Katsuda, K; Kubo, M; Mikami, O; Miyazaki, S; Muneta, Y; Murata, H; Nakajima, Y; Tanimura, N1
Benzaria, A; Di Scala, C; Guo, XJ; Maresca, M; Razafimanjato, H; Taïeb, N; Varini, K; Vidal, N1
Boyen, F; Croubels, S; De Backer, P; Goossens, J; Haesebrouck, F; Martel, A; Pasmans, F; Shearer, N; Thompson, A; Van Deun, K; Vandenbroucke, V; Verbrugghe, E1
Pestka, JJ1
Abrami, R; Cano, PM; Cognie, J; Guzylack-Piriou, L; Meurens, F; Oswald, IP; Seeboth, J1
Cornwell, P; Corton, JC; Jia, Q; Kinser, S; Laughter, A; Li, M; Pestka, J1
Boyron, M; Caporiccio, B; Fantini, J; Maresca, M; Yahi, N; Younès-Sakr, L1
Harkema, JR; Pestka, JJ; Yan, D; Zhou, HR1

Reviews

1 review(s) available for deoxynivalenol and Innate Inflammatory Response

ArticleYear
Deoxynivalenol-induced proinflammatory gene expression: mechanisms and pathological sequelae.
    Toxins, 2010, Volume: 2, Issue:6

    Topics: Animals; Endoplasmic Reticulum Chaperone BiP; Gene Expression Regulation; Humans; Immunity, Innate; Inflammation; Leukocytes; Ribosomes; Trichothecenes

2010

Trials

1 trial(s) available for deoxynivalenol and Innate Inflammatory Response

ArticleYear
The effects of acute exposure to deoxynivalenol on some inflammatory parameters in miniature pigs.
    The Journal of veterinary medical science, 2011, Volume: 73, Issue:5

    Topics: Acute-Phase Proteins; Animals; Cytokines; Gene Expression Regulation; Inflammation; Luminescence; Neutrophils; Swine; Swine Diseases; Swine, Miniature; Time Factors; Trichothecenes

2011

Other Studies

56 other study(ies) available for deoxynivalenol and Innate Inflammatory Response

ArticleYear
Effects of Deoxynivalenol and Fumonisins on Broiler Gut Cytoprotective Capacity.
    Toxins, 2021, 10-16, Volume: 13, Issue:10

    Topics: Animal Feed; Animals; Antioxidants; Biomarkers; Chickens; Cytochrome P-450 Enzyme System; Diet; Food Contamination; Fumonisins; Inflammation; Intestines; Male; Poultry Diseases; Receptors, Aryl Hydrocarbon; Stress, Physiological; Trichothecenes

2021
Chromatin Accessibility and Transcriptomic Alterations in Murine Ovarian Granulosa Cells upon Deoxynivalenol Exposure.
    Cells, 2021, 10-20, Volume: 10, Issue:11

    Topics: Animals; Cells, Cultured; Chromatin; Female; Gene Expression Profiling; Gene Expression Regulation; Genetic Loci; Granulosa Cells; Histone Code; Inflammation; MAP Kinase Signaling System; Mice, Inbred ICR; NF-kappa B; Signal Transduction; Transcription, Genetic; Transcriptome; Trichothecenes

2021
Low Dose of Deoxynivalenol Aggravates Intestinal Inflammation and Barrier Dysfunction Induced by Enterotoxigenic
    Journal of agricultural and food chemistry, 2022, Mar-09, Volume: 70, Issue:9

    Topics: Animals; Cell Line; Enterotoxigenic Escherichia coli; Escherichia coli Infections; Inflammasomes; Inflammation; Intestinal Mucosa; Macroautophagy; Mice; NLR Family, Pyrin Domain-Containing 3 Protein; Swine; Trichothecenes

2022
Deoxynivalenol induces apoptosis and inflammation in the liver: Analysis using precision-cut liver slices.
    Food and chemical toxicology : an international journal published for the British Industrial Biological Research Association, 2022, Volume: 163

    Topics: Animals; Apoptosis; Food Contamination; Inflammation; Liver; Mycotoxins; Swine; Trichothecenes

2022
Dihydroartemisinin alleviates deoxynivalenol induced liver apoptosis and inflammation in piglets.
    Ecotoxicology and environmental safety, 2022, Volume: 241

    Topics: Animals; Antioxidants; Apoptosis; Artemisinins; bcl-2-Associated X Protein; Caspase 3; Humans; Inflammation; Interleukin-2; Liver; NF-kappa B; NF-KappaB Inhibitor alpha; Receptors, Tumor Necrosis Factor, Type I; RNA, Messenger; Swine; Trichothecenes; Tumor Necrosis Factor-alpha

2022
Deoxynivalenol induces caspase-3/GSDME-dependent pyroptosis and inflammation in mouse liver and HepaRG cells.
    Archives of toxicology, 2022, Volume: 96, Issue:11

    Topics: Animals; Caspase 3; Humans; Inflammation; Interleukin-6; Lactate Dehydrogenases; Liver; Mice; Poly(ADP-ribose) Polymerase Inhibitors; Pyroptosis; Receptors, Estrogen; Trichothecenes

2022
Analysis of the roles of the Notch1 signalling pathway in modulating deoxynivalenol cytotoxicity.
    Ecotoxicology and environmental safety, 2022, Volume: 246

    Topics: Animals; Apoptosis; Humans; Inflammation; Oxidative Stress; Platelet Aggregation Inhibitors; Receptor, Notch1; Signal Transduction; Trichothecenes

2022
The combined effect of deoxynivalenol and Fumonisin B
    Toxicology letters, 2023, Jan-01, Volume: 372

    Topics: Animals; Caspases; Inflammation; Interleukin-18; Mice; Mycotoxins; NLR Family, Pyrin Domain-Containing 3 Protein; Pyroptosis

2023
Lactoferrin Attenuates Intestinal Barrier Dysfunction and Inflammation by Modulating the MAPK Pathway and Gut Microbes in Mice.
    The Journal of nutrition, 2022, Volume: 152, Issue:11

    Topics: Animals; Gastrointestinal Microbiome; Inflammation; Intestinal Diseases; Lactoferrin; Male; MAP Kinase Signaling System; Mice; Occludin; RNA, Messenger; Trichothecenes

2022
The mitigation mechanism of hesperidin on deoxynivalenol toxicity in grass carp hepatocytes via decreasing ROS accumulation and inhibiting JNK phosphorylation.
    Fish & shellfish immunology, 2023, Volume: 134

    Topics: Animals; Apoptosis; Carps; Hepatocytes; Hesperidin; Inflammation; Oxidative Stress; Phosphorylation; Reactive Oxygen Species

2023
Dietary taurine supplementation counteracts deoxynivalenol-induced liver injury via alleviating oxidative stress, mitochondrial dysfunction, apoptosis, and inflammation in piglets.
    Ecotoxicology and environmental safety, 2023, Mar-15, Volume: 253

    Topics: Animal Feed; Animals; Antioxidants; Apoptosis; Chemical and Drug Induced Liver Injury, Chronic; Dietary Supplements; Humans; Inflammation; Liver; Mitochondria; Oxidative Stress; Swine; Taurine

2023
Sodium Butyrate Ameliorates Deoxynivalenol-Induced Oxidative Stress and Inflammation in the Porcine Liver via NR4A2-Mediated Histone Acetylation.
    Journal of agricultural and food chemistry, 2023, Jul-12, Volume: 71, Issue:27

    Topics: Acetylation; Animals; Butyric Acid; Chemical and Drug Induced Liver Injury, Chronic; Histones; Inflammation; Oxidative Stress; Reactive Oxygen Species; Swine; Tumor Necrosis Factor-alpha

2023
Quercetin Alleviates Deoxynivalenol-Induced Intestinal Damage by Suppressing Inflammation and Ferroptosis in Mice.
    Journal of agricultural and food chemistry, 2023, Jul-19, Volume: 71, Issue:28

    Topics: Animals; Ferroptosis; Humans; Inflammation; Mice; NF-kappa B; Quercetin; Toll-Like Receptor 4

2023
Deoxynivalenol increases pro-inflammatory cytokine secretion and reduces primary bile acid transport in an inflamed intestinal in vitro co-culture model.
    Food research international (Ottawa, Ont.), 2023, Volume: 173, Issue:Pt 1

    Topics: Bile Acids and Salts; Caco-2 Cells; Coculture Techniques; Cytokines; Humans; Inflammation; Intestines; Lipopolysaccharides

2023
Deoxynivalenol induces oxidative stress, inflammatory response and apoptosis in bovine mammary epithelial cells.
    Journal of animal physiology and animal nutrition, 2019, Volume: 103, Issue:6

    Topics: Animals; Annexin A5; Antioxidants; Apoptosis; ATP-Binding Cassette Transporters; Cattle; Cell Cycle; Cell Line; Cell Survival; Epithelial Cells; Female; Fluorescein-5-isothiocyanate; Gene Expression Regulation; Inflammation; Malondialdehyde; Mammary Glands, Animal; Oxidative Stress; Periplasmic Binding Proteins; Reactive Oxygen Species; RNA, Messenger; Superoxide Dismutase; Trichothecenes

2019
Deoxynivalenol Induces Intestinal Damage and Inflammatory Response through the Nuclear Factor-κB Signaling Pathway in Piglets.
    Toxins, 2019, 11-14, Volume: 11, Issue:11

    Topics: Animals; Epithelial Cells; Inflammation; Intestines; NF-kappa B; Signal Transduction; Swine; Trichothecenes

2019
Schisandrin A protects intestinal epithelial cells from deoxynivalenol-induced cytotoxicity, oxidative damage and inflammation.
    Scientific reports, 2019, 12-16, Volume: 9, Issue:1

    Topics: Catalase; Cell Cycle Checkpoints; Cell Death; Cell Nucleus; Cell Survival; Cyclooctanes; Cyclooxygenase 2; Cytoprotection; Dinoprostone; Enterocytes; Gene Expression Regulation; Glutathione Peroxidase; Heme Oxygenase-1; HT29 Cells; Humans; Inflammation; Inflammation Mediators; Interleukin-8; Lignans; Lipid Peroxidation; MAP Kinase Signaling System; Models, Biological; NF-E2-Related Factor 2; NF-kappa B; Nitrites; Oxidative Stress; Polycyclic Compounds; Reactive Oxygen Species; RNA, Messenger; Superoxide Dismutase; Trichothecenes

2019
Deoxynivalenol Induces Inflammatory Injury in IPEC-J2 Cells via NF-κB Signaling Pathway.
    Toxins, 2019, 12-16, Volume: 11, Issue:12

    Topics: Animals; Cell Line; Cell Survival; Cyclooxygenase 2; Cytokines; Epithelial Cells; I-kappa B Kinase; Inflammation; Intestines; Nitric Oxide Synthase Type II; Signal Transduction; Swine; Transcription Factor RelA; Trichothecenes

2019
Combinatory effects of cereulide and deoxynivalenol on in vitro cell viability and inflammation of human Caco-2 cells.
    Archives of toxicology, 2020, Volume: 94, Issue:3

    Topics: Caco-2 Cells; Cell Survival; Depsipeptides; Diet; Emetics; Food Contamination; Humans; Inflammation; Interleukin-1beta; Interleukin-8; Intestinal Mucosa; Intestines; Mycotoxins; Trichothecenes

2020
Deoxynivalenol Induces Inflammation in IPEC-J2 Cells by Activating P38 Mapk And Erk1/2.
    Toxins, 2020, 03-13, Volume: 12, Issue:3

    Topics: Animals; Cell Line; Cell Survival; Epithelial Cells; Inflammation; Interleukin-1; Interleukin-6; Intestinal Mucosa; MAP Kinase Signaling System; p38 Mitogen-Activated Protein Kinases; Swine; Transcriptome; Trichothecenes; Tumor Necrosis Factor-alpha

2020
Baicalin alleviates deoxynivalenol-induced intestinal inflammation and oxidative stress damage by inhibiting NF-κB and increasing mTOR signaling pathways in piglets.
    Food and chemical toxicology : an international journal published for the British Industrial Biological Research Association, 2020, Volume: 140

    Topics: Animals; Flavonoids; Inflammation; Intestines; NF-kappa B; Signal Transduction; Swine; TOR Serine-Threonine Kinases; Trichothecenes

2020
Gut microbiota mediates the protective role of Lactobacillus plantarum in ameliorating deoxynivalenol-induced apoptosis and intestinal inflammation of broiler chickens.
    Poultry science, 2020, Volume: 99, Issue:5

    Topics: Animals; Antioxidants; Apoptosis; Chickens; Gastrointestinal Microbiome; Immunity, Mucosal; Immunologic Factors; In Vitro Techniques; Inflammation; Intestinal Diseases; Intestinal Mucosa; Lactobacillus plantarum; Lipopolysaccharides; Male; Poultry Diseases; Protective Agents; Spleen; Trichothecenes

2020
Celecoxib reduces Deoxynivalenol induced proliferation, inflammation and protein kinase C translocation via modulating downstream targets in mouse skin.
    Chemico-biological interactions, 2020, Aug-01, Volume: 326

    Topics: Animals; Celecoxib; Cell Proliferation; Cyclooxygenase 2; Cyclooxygenase 2 Inhibitors; Female; Inflammation; Mice; Protein Kinase C; Signal Transduction; Skin; Skin Diseases; Trichothecenes

2020
Deoxynivalenol induced spermatogenesis disorder by blood-testis barrier disruption associated with testosterone deficiency and inflammation in mice.
    Environmental pollution (Barking, Essex : 1987), 2020, Volume: 264

    Topics: Animals; Blood-Testis Barrier; Humans; Inflammation; Male; Mice; Spermatogenesis; Testis; Testosterone; Trichothecenes

2020
Chloroquine Improves Deoxynivalenol-Induced Inflammatory Response and Intestinal Mucosal Damage in Piglets.
    Oxidative medicine and cellular longevity, 2020, Volume: 2020

    Topics: Amine Oxidase (Copper-Containing); Animals; Antioxidants; Autophagy; Cadherins; Chloroquine; Cytokines; Diet; Inflammation; Integrins; Intestinal Mucosa; Lactic Acid; Occludin; Proliferating Cell Nuclear Antigen; RNA, Messenger; Sirolimus; Swine; Trichothecenes; Zonula Occludens-1 Protein

2020
Oral exposure of pigs to the mycotoxin deoxynivalenol does not modulate the hepatic albumin synthesis during a LPS-induced acute-phase reaction.
    Innate immunity, 2020, Volume: 26, Issue:8

    Topics: Acute-Phase Reaction; Administration, Oral; Albumins; Animal Feed; Animals; C-Reactive Protein; Dietary Supplements; Haptoglobins; Inflammation; Lipopolysaccharides; Liver; Mycotoxins; Serum Amyloid A Protein; Swine; Trichothecenes

2020
Low doses of individual and combined deoxynivalenol and zearalenone in naturally moldy diets impair intestinal functions via inducing inflammation and disrupting epithelial barrier in the intestine of piglets.
    Toxicology letters, 2020, Oct-15, Volume: 333

    Topics: Animal Feed; Animals; Body Weight; Cecum; Cytokines; Diet; Dose-Response Relationship, Drug; Drug Synergism; Female; Food Contamination; Fusarium; Gene Expression; Hordeum; Immunity, Mucosal; Inflammation; Intestinal Mucosa; Jejunum; Swine; Tight Junction Proteins; Trichothecenes; Zea mays; Zearalenone

2020
Nontoxic-dose deoxynivalenol aggravates lipopolysaccharides-induced inflammation and tight junction disorder in IPEC-J2 cells through activation of NF-κB and LC3B.
    Food and chemical toxicology : an international journal published for the British Industrial Biological Research Association, 2020, Volume: 145

    Topics: Animals; Cell Line; Epithelial Cells; Inflammation; Intestinal Mucosa; Lipopolysaccharides; Microtubule-Associated Proteins; NF-kappa B; NLR Family, Pyrin Domain-Containing 3 Protein; Signal Transduction; Tight Junctions; Trichothecenes

2020
Effect of chlorogenic acid on alleviating inflammation and apoptosis of IPEC-J2 cells induced by deoxyniyalenol.
    Ecotoxicology and environmental safety, 2020, Dec-01, Volume: 205

    Topics: Animals; Apoptosis; Caspase 3; Cell Count; Cell Line; Cell Survival; Chlorogenic Acid; Epithelial Cells; Inflammation; Intestines; Occludin; Protective Agents; Swine; Trichothecenes

2020
Nontoxic dose of Phenethyl isothiocyanate ameliorates deoxynivalenol-induced cytotoxicity and inflammation in IPEC-J2 cells.
    Research in veterinary science, 2021, Volume: 136

    Topics: Animals; Cell Line; Cell Survival; Cytokines; Epithelial Cells; Inflammation; Intestinal Mucosa; Isothiocyanates; NF-kappa B; Signal Transduction; Swine; Swine Diseases; Trichothecenes

2021
Effects of Deoxynivalenol and Mycotoxin Adsorbent Agents on Mitogen-Activated Protein Kinase Signaling Pathways and Inflammation-Associated Gene Expression in Porcine Intestinal Epithelial Cells.
    Toxins, 2021, 04-23, Volume: 13, Issue:5

    Topics: Animals; Gene Expression; Inflammation; Intestinal Mucosa; MAP Kinase Signaling System; Mycotoxins; Phosphorylation; RNA, Messenger; Swine; Tight Junctions; Trichothecenes

2021
Berberine improves intestinal barrier function and reduces inflammation, immunosuppression, and oxidative stress by regulating the NF-κB/MAPK signaling pathway in deoxynivalenol-challenged piglets.
    Environmental pollution (Barking, Essex : 1987), 2021, Nov-15, Volume: 289

    Topics: Animals; Berberine; Immunosuppression Therapy; Inflammation; NF-kappa B; Oxidative Stress; Signal Transduction; Swine; Trichothecenes

2021
Deoxynivalenol photocatalytic detoxification products alleviate intestinal barrier damage and gut flora disorder in BLAB/c mice.
    Food and chemical toxicology : an international journal published for the British Industrial Biological Research Association, 2021, Volume: 156

    Topics: Animals; Catalysis; Female; Food Contamination; Gastrointestinal Microbiome; Gene Expression Regulation; Inflammation; Intestinal Diseases; Intestines; Mice; Oxidative Stress; Photolysis; Random Allocation; Tight Junction Proteins; Trichothecenes

2021
Deoxynivalenol exposure induces liver damage in mice: Inflammation and immune responses, oxidative stress, and protective effects of Lactobacillus rhamnosus GG.
    Food and chemical toxicology : an international journal published for the British Industrial Biological Research Association, 2021, Volume: 156

    Topics: Animals; Chemical and Drug Induced Liver Injury; Cytokines; Gene Expression Regulation; Humans; Immunoglobulins; Inflammation; Lacticaseibacillus rhamnosus; Mice; Oxidative Stress; Probiotics; Random Allocation; RNA, Messenger; Trichothecenes

2021
Chronic DON exposure and acute LPS challenge: effects on porcine liver morphology and function.
    Mycotoxin research, 2017, Volume: 33, Issue:3

    Topics: Animal Feed; Animals; Diet; Food Contamination; Inflammation; Lipopolysaccharides; Liver; Mitochondria, Liver; Swine; Trichothecenes

2017
The Food Contaminants Nivalenol and Deoxynivalenol Induce Inflammation in Intestinal Epithelial Cells by Regulating Reactive Oxygen Species Release.
    Nutrients, 2017, Dec-11, Volume: 9, Issue:12

    Topics: Cell Line; Epithelial Cells; Food Contamination; Humans; Inflammation; Intestinal Mucosa; Reactive Oxygen Species; Trichothecenes

2017
Fungal Deoxynivalenol-Induced Enterocyte Distress Is Attenuated by Adulterated Adlay:
    Frontiers in immunology, 2018, Volume: 9

    Topics: Animals; Cell Line; Cell Proliferation; Chemokines; Diet; ELAV-Like Protein 1; Enterocytes; HT29 Cells; Humans; Inflammation; Interleukin-8; Plant Extracts; Poaceae; Protein Kinase C; rhoA GTP-Binding Protein; Signal Transduction; Swine; Trichothecenes

2018
Protecting intestinal epithelial cells against deoxynivalenol and E. coli damage by recombinant porcine IL-22.
    Veterinary microbiology, 2019, Volume: 231

    Topics: Animals; Antimicrobial Cationic Peptides; Apoptosis; Cell Line; Enterotoxigenic Escherichia coli; Epithelial Cells; Inflammation; Interleukin-22; Interleukins; Intestinal Mucosa; Recombinant Proteins; Signal Transduction; STAT3 Transcription Factor; Swine; Trichothecenes

2019
[Deoxynivalenol induced mRNA expressions of inflammation and apoptosis in BeWo cells].
    Wei sheng yan jiu = Journal of hygiene research, 2019, Volume: 48, Issue:1

    Topics: Apoptosis; Cytokines; Humans; Inflammation; RNA, Messenger; Trichothecenes

2019
Deoxynivalenol induced apoptosis and inflammation of IPEC-J2 cells by promoting ROS production.
    Environmental pollution (Barking, Essex : 1987), 2019, Volume: 251

    Topics: Animals; Antioxidants; Apoptosis; Cell Line; Cell Survival; Inflammation; Intestinal Mucosa; Reactive Oxygen Species; Swine; Trichothecenes

2019
Deoxynivalenol impairs hepatic and intestinal gene expression of selected oxidative stress, tight junction and inflammation proteins in broiler chickens, but addition of an adsorbing agent shifts the effects to the distal parts of the small intestine.
    PloS one, 2013, Volume: 8, Issue:7

    Topics: Adsorption; Animals; Biomarkers; Chickens; Gene Expression Regulation; Genes, Essential; Ileum; Inflammation; Intestine, Small; Jejunum; Liver; Oxidative Stress; RNA, Messenger; Tight Junctions; Trichothecenes

2013
The food born mycotoxin deoxynivalenol induces low-grade inflammation in mice in the absence of observed-adverse effects.
    Toxicology letters, 2015, Feb-03, Volume: 232, Issue:3

    Topics: Animals; Biomarkers; Dose-Response Relationship, Drug; Inflammation; Interleukin-1beta; Male; Mice; Mice, Inbred C57BL; Trichothecenes

2015
The Food-Associated Ribotoxin Deoxynivalenol Modulates Inducible NO Synthase in Human Intestinal Cell Model.
    Toxicological sciences : an official journal of the Society of Toxicology, 2015, Volume: 145, Issue:2

    Topics: Caco-2 Cells; Cytokines; Dose-Response Relationship, Drug; Enzyme Stability; Epithelial Cells; Food Contamination; Gene Expression Regulation, Enzymologic; Humans; Inflammation; Intestinal Mucosa; Nitric Oxide; Nitric Oxide Synthase Type II; Proteasome Endopeptidase Complex; Proteolysis; RNA, Messenger; Signal Transduction; Time Factors; Trichothecenes; Ubiquitination

2015
Acute and subchronic effects on immune responses of carp (Cyprinus carpio L.) after exposure to deoxynivalenol (DON) in feed.
    Mycotoxin research, 2015, Volume: 31, Issue:3

    Topics: Animal Feed; Animals; Arginase; Blood Cell Count; Carps; Cell Survival; Food Contamination; Gene Expression; Immunity, Innate; Inflammation; Kidney; Liver; Nitric Oxide; RNA, Messenger; Spleen; Trichothecenes

2015
Inflammation-induced expression of the alarmin interleukin 33 can be suppressed by galacto-oligosaccharides.
    International archives of allergy and immunology, 2015, Volume: 167, Issue:2

    Topics: Alarmins; Animals; Antigens, Dermatophagoides; Bronchoalveolar Lavage Fluid; Diet; Galactosides; Immunity, Mucosal; Immunosuppressive Agents; Inflammation; Interleukin-1 Receptor-Like 1 Protein; Interleukin-33; Intestinal Mucosa; Male; Mice; Mice, Inbred BALB C; Oligosaccharides; Receptors, Interleukin; RNA, Messenger; Trichothecenes

2015
Metabolic and hematological consequences of dietary deoxynivalenol interacting with systemic Escherichia coli lipopolysaccharide.
    Toxins, 2015, Nov-16, Volume: 7, Issue:11

    Topics: Acid-Base Equilibrium; Animal Feed; Animals; Blood Gas Analysis; Blood Glucose; Carbon Dioxide; Diet; Erythrocyte Count; Escherichia coli; Food Contamination; Inflammation; Lipopolysaccharides; Male; Sus scrofa; Trichothecenes; Water-Electrolyte Balance

2015
Effects of chronic deoxynivalenol exposure on p53 heterozygous and p53 homozygous mice.
    Food and chemical toxicology : an international journal published for the British Industrial Biological Research Association, 2016, Volume: 96

    Topics: Animals; Body Weight; Cell Proliferation; Dose-Response Relationship, Drug; Eating; Flow Cytometry; Heterozygote; Homozygote; Immunoglobulins; Inflammation; Kidney; Liver; Male; Mice; Mice, Knockout; Real-Time Polymerase Chain Reaction; Reverse Transcriptase Polymerase Chain Reaction; RNA, Messenger; Trichothecenes; Tumor Suppressor Protein p53

2016
Relationships between body temperatures and inflammation indicators under physiological and pathophysiological conditions in pigs exposed to systemic lipopolysaccharide and dietary deoxynivalenol.
    Journal of animal physiology and animal nutrition, 2018, Volume: 102, Issue:1

    Topics: Animal Feed; Animals; Biomarkers; Body Temperature; Inflammation; Kynurenine; Lipopolysaccharides; Swine; Trichothecenes; Tryptophan

2018
Tissue distribution and proinflammatory cytokine gene expression following acute oral exposure to deoxynivalenol: comparison of weanling and adult mice.
    Food and chemical toxicology : an international journal published for the British Industrial Biological Research Association, 2008, Volume: 46, Issue:8

    Topics: Aging; Animals; Biomarkers; Cytokines; Female; Gene Expression; Inflammation; Interleukin-1beta; Interleukin-6; Mice; Mycotoxins; Pharmacokinetics; Reverse Transcriptase Polymerase Chain Reaction; RNA, Messenger; Spleen; Tissue Distribution; Trichothecenes; Tumor Necrosis Factor-alpha

2008
Physio-pathological parameters affect the activation of inflammatory pathways by deoxynivalenol in Caco-2 cells.
    Toxicology in vitro : an international journal published in association with BIBRA, 2010, Volume: 24, Issue:7

    Topics: Caco-2 Cells; Cell Differentiation; Cell Proliferation; Dinoprostone; Dose-Response Relationship, Drug; Humans; Inflammation; Inflammation Mediators; Interleukin-8; Intestinal Mucosa; Time Factors; Trichothecenes

2010
The ribotoxin deoxynivalenol affects the viability and functions of glial cells.
    Glia, 2011, Volume: 59, Issue:11

    Topics: Adenosine Triphosphate; Animals; Astrocytes; Blotting, Western; Cell Survival; Cells, Cultured; Cytokines; Glutamate-Ammonia Ligase; Glutamic Acid; Homeostasis; Inflammation; Lipopolysaccharides; Membrane Proteins; Microscopy, Fluorescence; Neuroglia; Nitric Oxide; Rats; Rats, Wistar; Trichothecenes; Tumor Necrosis Factor-alpha

2011
The mycotoxin deoxynivalenol potentiates intestinal inflammation by Salmonella typhimurium in porcine ileal loops.
    PloS one, 2011, Volume: 6, Issue:8

    Topics: Animals; Cell Differentiation; Cell Line; Disease Models, Animal; Epithelial Cells; Gene Expression Regulation, Bacterial; Humans; Ileum; Inflammation; Intestinal Mucosa; Mycotoxins; Salmonella typhimurium; Sus scrofa; Trichothecenes

2011
Deoxynivalenol as a new factor in the persistence of intestinal inflammatory diseases: an emerging hypothesis through possible modulation of Th17-mediated response.
    PloS one, 2013, Volume: 8, Issue:1

    Topics: Animals; Cell Line; Chemokines; Dendritic Cells; Gene Expression Profiling; Gene Expression Regulation; In Vitro Techniques; Inflammation; Inflammation Mediators; Intestinal Mucosa; Intestines; Jejunum; Models, Animal; RNA, Messenger; Sus scrofa; T-Lymphocytes, Regulatory; Th17 Cells; Trichothecenes

2013
Gene expression profiling in spleens of deoxynivalenol-exposed mice: immediate early genes as primary targets.
    Journal of toxicology and environmental health. Part A, 2004, Sep-24, Volume: 67, Issue:18

    Topics: Animals; Chemokines; Chemotaxis; Cytokines; Drug Evaluation, Preclinical; Gene Expression Profiling; Genes, Immediate-Early; Hydrolases; Inflammation; Linear Models; Mice; Mice, Inbred Strains; Oligonucleotide Array Sequence Analysis; Phylogeny; Polymerase Chain Reaction; Signal Transduction; Spleen; Toxicogenetics; Transcription Factors; Trichothecenes; Up-Regulation

2004
Both direct and indirect effects account for the pro-inflammatory activity of enteropathogenic mycotoxins on the human intestinal epithelium: stimulation of interleukin-8 secretion, potentiation of interleukin-1beta effect and increase in the transepithel
    Toxicology and applied pharmacology, 2008, Apr-01, Volume: 228, Issue:1

    Topics: Bacteria; Caco-2 Cells; Humans; Inflammation; Interleukin-1beta; Interleukin-8; Intestinal Mucosa; Mycotoxins; NF-kappa B; Ochratoxins; p38 Mitogen-Activated Protein Kinases; Patulin; Permeability; Reverse Transcriptase Polymerase Chain Reaction; RNA, Messenger; Signal Transduction; Trichothecenes

2008
Amplified proinflammatory cytokine expression and toxicity in mice coexposed to lipopolysaccharide and the trichothecene vomitoxin (deoxynivalenol).
    Journal of toxicology and environmental health. Part A, 1999, May-28, Volume: 57, Issue:2

    Topics: Animals; Blood Proteins; Cytokines; Gene Expression; Inflammation; Interleukin-6; Lipopolysaccharides; Lymphoid Tissue; Male; Mice; Mice, Inbred Strains; RNA, Messenger; Salmonella typhimurium; Trichothecenes; Tumor Necrosis Factor-alpha

1999