deamino-arginine-vasopressin and Cystic-Fibrosis

Exercise-associated hyponatraemia (EAH) always involves a component of overhydration relative to available exchangeable sodium stores. In the majority of cases, this is purely due to excessive consumption of fluids during exercise. In a lesser number of cases, it is apparent that excessive sodium loss through sweat may play a role by decreasing the amount of acutely available exchangeable sodium. Two cases demonstrating the latter, one in an individual with cystic fibrosis (CF) and another in an endurance athlete without CF, demonstrate how elevated dermal sweat losses may contribute to a

Topics: Adult; Aftercare; Anticonvulsants; Antidiuretic Agents; Confusion; Cystic Fibrosis; Deamino Arginine Vasopressin; Diagnosis, Differential; Exercise; Female; Heat Exhaustion; Hospitalization; Humans; Hyponatremia; Iontophoresis; Lorazepam; Male; Nausea; Osmolar Concentration; Seizures; Sodium; Sweat; Treatment Outcome

Desmopressin and vasopressin were used to control massive haemoptysis in a patient with cystic fibrosis. After bolus doses a continuous infusion of vasopressin was maintained for 36 hours and haemoptysis stopped.

Topics: Adult; Cystic Fibrosis; Deamino Arginine Vasopressin; Drug Therapy, Combination; Hemoptysis; Humans; Male; Vasopressins

Topics: Administration, Intranasal; Adolescent; Coma; Cystic Fibrosis; Deamino Arginine Vasopressin; Female; Humans; Seizures