Page last updated: 2024-10-26

ddt and Primary Ovarian Insufficiency

ddt has been researched along with Primary Ovarian Insufficiency in 1 studies

1,1-bis(p-chlorophenyl)-2,2,2-trichloroethane: structure in first source

Primary Ovarian Insufficiency: Cessation of ovarian function after MENARCHE but before the age of 40, without or with OVARIAN FOLLICLE depletion. It is characterized by the presence of OLIGOMENORRHEA or AMENORRHEA, elevated GONADOTROPINS, and low ESTRADIOL levels. It is a state of female HYPERGONADOTROPIC HYPOGONADISM. Etiologies include genetic defects, autoimmune processes, chemotherapy, radiation, and infections. The most commonly known genetic cause is the expansion of a CGG repeat to 55 to 199 copies in the 5' untranslated region in the X-linked FMR1 gene.

Research

Studies (1)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's0 (0.00)29.6817
2010's1 (100.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Nilsson, E1
Klukovich, R1
Sadler-Riggleman, I1
Beck, D1
Xie, Y1
Yan, W1
Skinner, MK1

Other Studies

1 other study available for ddt and Primary Ovarian Insufficiency

ArticleYear
Environmental toxicant induced epigenetic transgenerational inheritance of ovarian pathology and granulosa cell epigenome and transcriptome alterations: ancestral origins of polycystic ovarian syndrome and primary ovarian insufiency.
    Epigenetics, 2018, Volume: 13, Issue:8

    Topics: Animals; DDT; DNA Methylation; Environmental Pollutants; Epigenesis, Genetic; Female; Granulosa Cell

2018