cyclosporine and Asymmetric Septal Hypertrophy, Familial

cyclosporine has been researched along with Asymmetric Septal Hypertrophy, Familial in 1 studies

Research

Studies (1)

Timeframe	Studies, this research(%)	All Research%
pre-1990	0 (0.00)	18.7374
1990's	0 (0.00)	18.2507
2000's	0 (0.00)	29.6817
2010's	1 (100.00)	24.3611
2020's	0 (0.00)	2.80

Authors

Authors	Studies
Blankenburg, R; Deenen, R; Hackert, K; Lohse, MJ; Schmitt, JP; Seidman, CE; Seidman, JG; Wurster, S	1

Other Studies

1 other study(ies) available for cyclosporine and Asymmetric Septal Hypertrophy, Familial

Article	Year
β-Myosin heavy chain variant Val606Met causes very mild hypertrophic cardiomyopathy in mice, but exacerbates HCM phenotypes in mice carrying other HCM mutations. Circulation research, 2014, Jul-07, Volume: 115, Issue:2 Topics: Amino Acid Substitution; Animals; Cardiac Myosins; Cardiomyopathy, Hypertrophic, Familial; Cyclosporine; Disease Models, Animal; Gene Knock-In Techniques; Genotype; Humans; Hypertrophy, Left Ventricular; Mice; Models, Molecular; Mutation, Missense; Myocardial Contraction; Myosin Heavy Chains; Phenotype; Point Mutation; Protein Conformation; Transcription, Genetic; Ultrasonography; Ventricular Myosins; Ventricular Remodeling	2014

Article

Year

β-Myosin heavy chain variant Val606Met causes very mild hypertrophic cardiomyopathy in mice, but exacerbates HCM phenotypes in mice carrying other HCM mutations.

Circulation research, 2014, Jul-07, Volume: 115, Issue:2

Topics: Amino Acid Substitution; Animals; Cardiac Myosins; Cardiomyopathy, Hypertrophic, Familial; Cyclosporine; Disease Models, Animal; Gene Knock-In Techniques; Genotype; Humans; Hypertrophy, Left Ventricular; Mice; Models, Molecular; Mutation, Missense; Myocardial Contraction; Myosin Heavy Chains; Phenotype; Point Mutation; Protein Conformation; Transcription, Genetic; Ultrasonography; Ventricular Myosins; Ventricular Remodeling

2014