cyclosporine has been researched along with Anterior Horn Cell Disease in 4 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 2 (50.00) | 18.2507 |
| 2000's | 2 (50.00) | 29.6817 |
| 2010's | 0 (0.00) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Brown, RH; Kazantsev, AG; Maxwell, MM; Pasinelli, P | 1 |
| Anderson, GW; Even, C; Harty, JT; Palmer, GA; Plagemann, PG; Rowland, RR | 1 |
| Arechalde, A; Braun, R; French, LE | 1 |
| Akaike, A; Honda, K; Inoue, R; Kihara, T; Nakamizo, T; Sawada, H; Shimohama, S; Urushitani, M | 1 |
4 other study(ies) available for cyclosporine and Anterior Horn Cell Disease
| Article | Year |
|---|---|
RNA interference-mediated silencing of mutant superoxide dismutase rescues cyclosporin A-induced death in cultured neuroblastoma cells.
Topics: Base Sequence; Cell Death; Cell Survival; Cyclosporine; DNA Primers; DNA, Complementary; Gene Silencing; HeLa Cells; Humans; Motor Neuron Disease; Mutagenesis, Site-Directed; Neuroblastoma; RNA, Small Interfering; Superoxide Dismutase; Superoxide Dismutase-1; Transfection; Tumor Cells, Cultured | 2004 |
C58 and AKR mice of all ages develop motor neuron disease after lactate dehydrogenase-elevating virus infection but only if antiviral immune responses are blocked by chemical or genetic means or as a result of old age.
Topics: Aging; Animals; Antibodies, Viral; Arterivirus Infections; Blotting, Northern; Cyclosporine; Female; Immune System; Immunosuppressive Agents; Lactate dehydrogenase-elevating virus; Leukemia Virus, Murine; Male; Mice; Mice, Inbred AKR; Mice, Transgenic; Motor Neuron Disease; Poliomyelitis; RNA, Viral; Spinal Cord | 1995 |
Reversible ascending motor neuropathy as a side effect of systemic treatment with ciclosporine for nodular prurigo.
Topics: Aged; Aged, 80 and over; Cyclosporine; Humans; Immunosuppressive Agents; Motor Neuron Disease; Prurigo | 1999 |
N-methyl-D-aspartate receptor-mediated mitochondrial Ca(2+) overload in acute excitotoxic motor neuron death: a mechanism distinct from chronic neurotoxicity after Ca(2+) influx.
Topics: 2,4-Dinitrophenol; 6-Cyano-7-nitroquinoxaline-2,3-dione; alpha-Amino-3-hydroxy-5-methyl-4-isoxazolepropionic Acid; Animals; Calcium; Calcium Signaling; Calcium-Calmodulin-Dependent Protein Kinase Type 2; Calcium-Calmodulin-Dependent Protein Kinases; Carbonyl Cyanide p-Trifluoromethoxyphenylhydrazone; Cells, Cultured; Cyclosporine; Dibucaine; Dizocilpine Maleate; Enzyme Inhibitors; Excitatory Amino Acid Agonists; Excitatory Amino Acid Antagonists; Fluoresceins; Fluorescent Dyes; Glutamic Acid; Heterocyclic Compounds, 3-Ring; Imidazoles; Kainic Acid; Mitochondria; Motor Neuron Disease; Motor Neurons; N-Methylaspartate; Nerve Tissue Proteins; Neurons; Neurotoxins; Nitric Oxide Synthase; Nitric Oxide Synthase Type I; Oxidative Stress; Rats; Rats, Wistar; Reactive Oxygen Species; Receptors, AMPA; Receptors, Kainic Acid; Receptors, N-Methyl-D-Aspartate; Rhodamines; Spinal Cord; Superoxide Dismutase; Uncoupling Agents | 2001 |