Page last updated: 2024-08-21

cyclopentane and Ventricular Dysfunction, Left

cyclopentane has been researched along with Ventricular Dysfunction, Left in 3 studies

Research

Studies (3)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's0 (0.00)29.6817
2010's3 (100.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Ahmad, S; da Silva, JS; Ferrario, CM; Groban, L; Sudo, RT; Sun, X; Varagic, J; Wang, H; Zapata-Sudo, G1
Hoshiga, M; Ishizaka, N; Ito, T; Kanzaki, Y; Morita, H; Nishida, Y; Shibata, K; Sohmiya, K; Tanaka, S; Tominaga, T; Ukimura, A; Wada, Y; Yoshimizu, N1
Chagas, C; Groban, L; Jessup, JA; Lin, MS; Lindsey, SH; Wang, H1

Other Studies

3 other study(ies) available for cyclopentane and Ventricular Dysfunction, Left

ArticleYear
G-Protein-Coupled Estrogen Receptor Agonist G1 Improves Diastolic Function and Attenuates Cardiac Renin-Angiotensin System Activation in Estrogen-Deficient Hypertensive Rats.
    Journal of cardiovascular pharmacology, 2019, Volume: 74, Issue:5

    Topics: Angiotensin II; Angiotensin-Converting Enzyme 2; Animals; Cyclopentanes; Diastole; Disease Models, Animal; Estrogens; Female; Hypertension; Myocardium; Ovariectomy; Peptidyl-Dipeptidase A; Proto-Oncogene Mas; Proto-Oncogene Proteins; Quinolines; Rats, Inbred SHR; Receptor, Angiotensin, Type 1; Receptors, G-Protein-Coupled; Renin-Angiotensin System; Signal Transduction; Ventricular Dysfunction, Left; Ventricular Function, Left

2019
Repetitive fulminant influenza myocarditis requiring the use of circulatory assist devices.
    Internal medicine (Tokyo, Japan), 2014, Volume: 53, Issue:2

    Topics: Acids, Carbocyclic; Adult; Antibodies, Viral; Antiviral Agents; Catecholamines; Cyclopentanes; Dyspnea; Electrocardiography; Guanidines; Heart-Assist Devices; Hemodynamics; Humans; Influenza A Virus, H3N2 Subtype; Influenza B virus; Influenza, Human; Intra-Aortic Balloon Pumping; Magnetic Resonance Imaging; Male; Myocarditis; Pericarditis; Recurrence; Ultrasonography; Ventricular Dysfunction, Left

2014
Activation of GPR30 attenuates diastolic dysfunction and left ventricle remodelling in oophorectomized mRen2.Lewis rats.
    Cardiovascular research, 2012, Apr-01, Volume: 94, Issue:1

    Topics: Angiotensin II; Animals; Atrial Natriuretic Factor; Blood Pressure; Calcium; Cardiotonic Agents; Cell Line; Collagen; Cyclopentanes; Echocardiography, Doppler; Estrogens; Female; Gene Expression Regulation; Genotype; Hypertrophy, Left Ventricular; Injections, Subcutaneous; Mice; Myocytes, Cardiac; NADPH Oxidase 4; NADPH Oxidases; Natriuretic Peptide, Brain; Ovariectomy; Phenotype; Quinolines; Rats; Rats, Inbred Lew; Rats, Transgenic; Receptors, G-Protein-Coupled; Renin; RNA, Messenger; Time Factors; Ventricular Dysfunction, Left; Ventricular Function, Left; Ventricular Remodeling

2012