Page last updated: 2024-08-21

cyclopentane and Bone Cancer

cyclopentane has been researched along with Bone Cancer in 5 studies

Research

Studies (5)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's0 (0.00)29.6817
2010's5 (100.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Chen, X; Jin, Y; Wang, Z; Zhao, Y; Zheng, J1
Huang, KY; Huang, TJ; Kuan, FC; Kuo, KL; Lee, CY; Lin, WC; Liu, SH; Shi, CS; Tang, CH; Wu, MH1
Li, GQ; Li, SS; Shi, CC; Zhang, HP; Zhang, Y1
Deng, Z; Jiang, F; Li, JD; Liu, H; Mao, W; Xia, Z; Zhang, J; Zhang, S1
de Álava, E; García-Domínguez, DJ; Ginel-Picardo, A; Mackintosh, C; Ordóñez, JL; Sacristán, MP; Smith, PG1

Other Studies

5 other study(ies) available for cyclopentane and Bone Cancer

ArticleYear
G-protein-coupled estrogen receptor suppresses the migration of osteosarcoma cells via post-translational regulation of Snail.
    Journal of cancer research and clinical oncology, 2019, Volume: 145, Issue:1

    Topics: Bone Neoplasms; Cell Line; Cell Line, Tumor; Cell Movement; Cyclopentanes; Disease Progression; Down-Regulation; Epithelial-Mesenchymal Transition; F-Box Proteins; Humans; Neoplasm Invasiveness; Neoplasm Metastasis; Osteosarcoma; Protein Processing, Post-Translational; Quinolines; Receptors, Estrogen; Receptors, G-Protein-Coupled; RNA, Messenger; Snail Family Transcription Factors; Survival Rate; Ubiquitin-Protein Ligase Complexes

2019
MLN4924, a Protein Neddylation Inhibitor, Suppresses the Growth of Human Chondrosarcoma through Inhibiting Cell Proliferation and Inducing Endoplasmic Reticulum Stress-Related Apoptosis.
    International journal of molecular sciences, 2018, Dec-24, Volume: 20, Issue:1

    Topics: Animals; Apoptosis; Bone Neoplasms; Caspase 3; Cell Line, Tumor; Cell Proliferation; Chondrosarcoma; Cyclopentanes; Endoplasmic Reticulum Chaperone BiP; Endoplasmic Reticulum Stress; Enzyme Inhibitors; G2 Phase Cell Cycle Checkpoints; Heat-Shock Proteins; Histones; Humans; Mice; Mice, Nude; NEDD8 Protein; Phosphorylation; Pyrimidines; Transcription Factor CHOP; Transplantation, Heterologous

2018
MLN4924 suppresses neddylation and induces cell cycle arrest, senescence, and apoptosis in human osteosarcoma.
    Oncotarget, 2016, Jul-19, Volume: 7, Issue:29

    Topics: Animals; Apoptosis; Bone Neoplasms; Cellular Senescence; Cullin Proteins; Cyclopentanes; DNA Damage; G2 Phase Cell Cycle Checkpoints; Humans; Mice; Osteosarcoma; Protein Processing, Post-Translational; Pyrimidines; Ubiquitin-Activating Enzymes; Xenograft Model Antitumor Assays

2016
Circadian clock components RORα and Bmal1 mediate the anti-proliferative effect of MLN4924 in osteosarcoma cells.
    Oncotarget, 2016, Oct-04, Volume: 7, Issue:40

    Topics: Animals; Apoptosis; ARNTL Transcription Factors; Biomarkers, Tumor; Bone Neoplasms; Cell Proliferation; Circadian Clocks; Cyclopentanes; Enzyme Inhibitors; Humans; Mice; Mice, Nude; Nuclear Receptor Subfamily 1, Group F, Member 1; Osteosarcoma; Pyrimidines; Tumor Cells, Cultured; Xenograft Model Antitumor Assays

2016
WEE1 accumulation and deregulation of S-phase proteins mediate MLN4924 potent inhibitory effect on Ewing sarcoma cells.
    Oncogene, 2013, Mar-14, Volume: 32, Issue:11

    Topics: Animals; Antineoplastic Agents; Bone Neoplasms; Cell Cycle Proteins; Cell Line, Tumor; Cyclopentanes; Female; Gene Expression Regulation, Neoplastic; Gene Knockdown Techniques; Humans; Mice; Mice, Inbred C57BL; Nuclear Proteins; Protein-Tyrosine Kinases; Pyrimidines; RNA, Small Interfering; S Phase; Sarcoma, Ewing; Xenograft Model Antitumor Assays

2013