Compounds > cyclic adp-ribose
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cyclic adp-ribose and Nerve Degeneration

cyclic adp-ribose has been researched along with Nerve Degeneration in 3 studies

Research

Studies (3)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's1 (33.33)29.6817
2010's0 (0.00)24.3611
2020's2 (66.67)2.80

Authors

AuthorsStudies
Avizonis, D; Bergholz, JS; Chen, CY; de Sá Tavares Russo, M; DiAntonio, A; Hsueh, YP; Jiang, T; Ko, KW; Li, Y; Milbrandt, J; Pazyra-Murphy, MF; Segal, RA; Tang, S; Zhao, JJ; Zhu, J1
Bosanac, T; Devraj, R; DiAntonio, A; Engber, TM; Figley, MD; Hughes, RO; Krauss, R; Milbrandt, J; Sasaki, Y; Wu, T1
Hu, S; Peterson, PK; Shideman, CR; Thayer, SA1

Other Studies

3 other study(ies) available for cyclic adp-ribose and Nerve Degeneration

ArticleYear
Sarm1 activation produces cADPR to increase intra-axonal Ca++ and promote axon degeneration in PIPN.
    The Journal of cell biology, 2022, 02-07, Volume: 221, Issue:2

    Topics: Animals; Armadillo Domain Proteins; Axons; Calcium; Calcium Channels; Cyclic ADP-Ribose; Cytoskeletal Proteins; Female; HEK293 Cells; Humans; Mice, Inbred C57BL; Nerve Degeneration; Paclitaxel; Peripheral Nervous System Diseases; Rats, Sprague-Dawley

2022
cADPR is a gene dosage-sensitive biomarker of SARM1 activity in healthy, compromised, and degenerating axons.
    Experimental neurology, 2020, Volume: 329

    Topics: Animals; Armadillo Domain Proteins; Axons; Biomarkers; Brain; Cells, Cultured; Cyclic ADP-Ribose; Cytoskeletal Proteins; Gene Dosage; Mice; Mice, Inbred C57BL; Mice, Knockout; Nerve Degeneration; Sciatic Nerve

2020
CCL5 evokes calcium signals in microglia through a kinase-, phosphoinositide-, and nucleotide-dependent mechanism.
    Journal of neuroscience research, 2006, Volume: 83, Issue:8

    Topics: Agammaglobulinaemia Tyrosine Kinase; Calcium; Calcium Channels; Calcium Signaling; Cells, Cultured; Chemokine CCL5; Chemokines, CC; Cyclic ADP-Ribose; Encephalitis; GTP-Binding Protein alpha Subunits, Gi-Go; Humans; Immunity, Innate; Immunologic Surveillance; Inositol 1,4,5-Trisphosphate; Janus Kinase 1; Microglia; NAD; Nerve Degeneration; Phosphatidylinositol 3-Kinases; Protein-Tyrosine Kinases; Receptors, CCR5; Signal Transduction; Type C Phospholipases

2006