cyc 202 has been researched along with Kidney Failure, Chronic in 2 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 0 (0.00) | 29.6817 |
| 2010's | 2 (100.00) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Alkanderi, S; Filby, A; Henry, C; Miles, CG; Molinari, E; Ramsbottom, SA; Saunier, S; Sayer, JA; Srivastava, S; White, K | 1 |
| Beier, DR; Billot, K; Bukanov, NO; Carlson, G; Husson, H; Ibraghimov-Beskrovnaya, O; Lane, M; Ledbetter, SR; Meijer, L; Moreno, S; Pitstick, R; Russo, RJ; Sergeev, M; Shah, J; Smith, LA; Smith, MM; Zhang, K | 1 |
2 other study(ies) available for cyc 202 and Kidney Failure, Chronic
| Article | Year |
|---|---|
A human patient-derived cellular model of Joubert syndrome reveals ciliary defects which can be rescued with targeted therapies.
Topics: Abnormalities, Multiple; Antigens, Neoplasm; Cell Cycle Proteins; Cerebellum; Child; Child, Preschool; Cilia; Ciliopathies; Cyclin-Dependent Kinase 5; Cytoskeletal Proteins; Epithelial Cells; Eye Abnormalities; Humans; Kidney Diseases, Cystic; Kidney Failure, Chronic; Male; Morpholines; Mutation; Neoplasm Proteins; Pedigree; Polycystic Kidney Diseases; Primary Cell Culture; Purines; Retina; Roscovitine; Signal Transduction | 2017 |
Reduction of ciliary length through pharmacologic or genetic inhibition of CDK5 attenuates polycystic kidney disease in a model of nephronophthisis.
Topics: Animals; Cell Differentiation; Cilia; Cyclin-Dependent Kinase 5; Disease Models, Animal; Humans; Intercellular Signaling Peptides and Proteins; Kidney; Kidney Failure, Chronic; Mice; Nerve Tissue Proteins; Polycystic Kidney Diseases; Purines; Roscovitine; Tubulin | 2016 |