Page last updated: 2024-08-26

cv 3988 and Pulmonary Disease, Chronic Obstructive

cv 3988 has been researched along with Pulmonary Disease, Chronic Obstructive in 1 studies

Research

Studies (1)

Timeframe	Studies, this research(%)	All Research%
pre-1990	0 (0.00)	18.7374
1990's	0 (0.00)	18.2507
2000's	0 (0.00)	29.6817
2010's	1 (100.00)	24.3611
2020's	0 (0.00)	2.80

Authors

Authors	Studies
Marentette, JO; McHowat, J; Rastogi, P; Sharma, J; Turk, J; Young, DM	1

Other Studies

1 other study(ies) available for cv 3988 and Pulmonary Disease, Chronic Obstructive

Article	Year
Lung endothelial cell platelet-activating factor production and inflammatory cell adherence are increased in response to cigarette smoke component exposure. American journal of physiology. Lung cellular and molecular physiology, 2012, Jan-01, Volume: 302, Issue:1 Topics: 1-Alkyl-2-acetylglycerophosphocholine Esterase; Animals; Cell Adhesion; Cell Line; Endothelial Cells; Group VI Phospholipases A2; Humans; Inflammation; Lung; Mice; Mice, Knockout; Neutrophils; Phospholipid Ethers; Platelet Activating Factor; Platelet Membrane Glycoproteins; Pulmonary Disease, Chronic Obstructive; Receptors, G-Protein-Coupled; Tobacco Smoke Pollution	2012

Article

Year

Lung endothelial cell platelet-activating factor production and inflammatory cell adherence are increased in response to cigarette smoke component exposure.

American journal of physiology. Lung cellular and molecular physiology, 2012, Jan-01, Volume: 302, Issue:1

Topics: 1-Alkyl-2-acetylglycerophosphocholine Esterase; Animals; Cell Adhesion; Cell Line; Endothelial Cells; Group VI Phospholipases A2; Humans; Inflammation; Lung; Mice; Mice, Knockout; Neutrophils; Phospholipid Ethers; Platelet Activating Factor; Platelet Membrane Glycoproteins; Pulmonary Disease, Chronic Obstructive; Receptors, G-Protein-Coupled; Tobacco Smoke Pollution

2012