cuprizone has been researched along with Encephalitis in 15 studies
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 0 (0.00) | 18.2507 |
2000's | 2 (13.33) | 29.6817 |
2010's | 12 (80.00) | 24.3611 |
2020's | 1 (6.67) | 2.80 |
Authors | Studies |
---|---|
Bando, Y; Geisler, JG | 1 |
Behrangi, N; Brendel, M; Jiangshan, Z; Kaye, J; Kipp, M; Nedelcu, J; Reinbach, C; Riedler, P; Rominger, A; Schmitz, C | 1 |
Bello, AM; Caprariello, AV; Chakka, SK; Dunn, SE; Kotra, LP; Muthuramu, CM; Sarswat, A; Stys, PK; Wasilewski, E | 1 |
DuBois, JC; Gruber, RC; Gulinello, ME; Guzik, HM; Kozakiewicz, L; Perumal, G; Raine, C; Ray, AK; Shafit-Zagardo, B; Williamson, J | 1 |
Caprariello, AV; Dunn, JF; Hoghooghi, V; Koebel, A; Kotra, LP; Morgan, ML; Ousman, SS; Plemel, JR; Rogers, JA; Stys, PK; Tsutsui, S; Wee Yong, V | 1 |
Tezuka, T | 1 |
Boilesen, SN; LeSueur-Maluf, L; Medalha, CC; Monteiro de Castro, G; Prado de França Carvalho, C; Serra-de-Oliveira, N; Spadari, RC; Zollner, Rde L | 1 |
Huang, Q; Kong, J; Peng, H; Shao, Y; Xu, H | 1 |
Barbosa, KP; da Cruz-Höfling, MA; Luna, RL; Nunes, AK; Peixoto, CA; Rapôso, C; Rocha, SW | 1 |
Eckermann, N; Fairless, R; Gudi, V; Pul, R; Skripuletz, T; Stangel, M; Sühs, KW | 1 |
Berneman, Z; Bigot, C; Daans, J; De Vocht, N; Goossens, H; Guglielmetti, C; Hendrix, S; Hens, N; Hoornaert, C; Kara, F; Le Blon, D; Mai, Z; Peerlings, J; Planas, AM; Ponsaerts, P; Praet, J; Salas-Perdomo, A; Santermans, E; Shah, D; van der Linden, A; Verhoye, M | 1 |
Awad, H; Berger, K; Beyer, C; Buschmann, JP; Clarner, T; Kipp, M | 1 |
Amor, S; Berger, K; Beyer, C; Clarner, T; Denecke, B; Diederichs, F; Gan, L; Kipp, M; van der Valk, P | 1 |
Arnett, HA; Plant, SR; Ting, JP | 1 |
Burkly, LC; Hahm, K; Iocca, HA; Lundsmith, ET; O'Connor, BP; Plant, SR; Runkel, L; Ting, JP; van Deventer, HW; Wang, Y | 1 |
1 review(s) available for cuprizone and Encephalitis
Article | Year |
---|---|
[Cuprizone short-term exposure: glial activation and psychosis-like behavior].
Topics: Animals; Brain; Cuprizone; Cytokines; Disease Models, Animal; Drug Discovery; Encephalitis; Humans; Inflammation Mediators; Mice; Schizophrenia | 2013 |
14 other study(ies) available for cuprizone and Encephalitis
Article | Year |
---|---|
Disease modifying mitochondrial uncouplers, MP101, and a slow release ProDrug, MP201, in models of Multiple Sclerosis.
Topics: 2,4-Dinitrophenol; Animals; Axons; Brain-Derived Neurotrophic Factor; Cuprizone; Cytokines; Delayed-Action Preparations; Demyelinating Diseases; Encephalitis; Encephalomyelitis, Autoimmune, Experimental; Female; Immunization; Mice; Mice, Inbred C57BL; Mitochondria; Multiple Sclerosis; Nerve Growth Factors; Paralysis; Prodrugs; Uncoupling Agents | 2019 |
Laquinimod ameliorates secondary brain inflammation.
Topics: Animals; Brain; Cuprizone; Disease Models, Animal; Encephalitis; Female; Gliosis; Mice, Inbred C57BL; Multiple Sclerosis; Quinolones | 2020 |
Inhibitors of protein arginine deiminases and their efficacy in animal models of multiple sclerosis.
Topics: Animals; Brain; Catalytic Domain; Cuprizone; Demyelinating Diseases; Encephalitis; Enzyme Inhibitors; Female; Half-Life; Humans; Hydantoins; Hydrolases; Imidazoles; Isoenzymes; Male; Mice, Inbred C57BL; Molecular Docking Simulation; Multiple Sclerosis; Myelitis; Neuroprotective Agents; Spinal Cord; Tetrazoles | 2017 |
Loss of Gas6 and Axl signaling results in extensive axonal damage, motor deficits, prolonged neuroinflammation, and less remyelination following cuprizone exposure.
Topics: Animals; Axl Receptor Tyrosine Kinase; Axons; Cuprizone; Demyelinating Diseases; Disease Models, Animal; Encephalitis; Gene Expression Regulation; Intercellular Signaling Peptides and Proteins; Mice; Mice, Inbred C57BL; Mice, Transgenic; Monoamine Oxidase Inhibitors; Movement Disorders; Myelin Sheath; Nerve Tissue Proteins; Proprioception; Proto-Oncogene Proteins; Psychomotor Performance; Receptor Protein-Tyrosine Kinases; Reflex, Righting; Remyelination; Signal Transduction | 2017 |
Biochemically altered myelin triggers autoimmune demyelination.
Topics: Animals; Cuprizone; Demyelinating Diseases; Disease Models, Animal; Encephalitis; Hashimoto Disease; Humans; Hydrolases; Inflammation; Male; Mice; Mice, Inbred C57BL; Monoamine Oxidase Inhibitors; Multiple Sclerosis; Myelin Sheath | 2018 |
Behavioural changes observed in demyelination model shares similarities with white matter abnormalities in humans.
Topics: Animals; Anxiety; Behavior, Animal; Corpus Callosum; Cuprizone; Disease Models, Animal; Encephalitis; Humans; Male; Microglia; Motor Activity; Multiple Sclerosis; Myelin Sheath; Oligodendroglia; Rats; Rats, Inbred Lew; White Matter | 2015 |
Quetiapine mitigates the neuroinflammation and oligodendrocyte loss in the brain of C57BL/6 mouse following cuprizone exposure for one week.
Topics: Animals; Antipsychotic Agents; Behavior, Animal; Cuprizone; Disease Models, Animal; Encephalitis; Enzyme-Linked Immunosorbent Assay; Exploratory Behavior; Immunohistochemistry; Interleukin-6; Male; Maze Learning; Mice, Inbred C57BL; Motor Activity; Oligodendroglia; Quetiapine Fumarate; Tumor Necrosis Factor-alpha | 2015 |
Involvement of AMPK, IKβα-NFκB and eNOS in the sildenafil anti-inflammatory mechanism in a demyelination model.
Topics: AMP-Activated Protein Kinases; Animals; Anti-Inflammatory Agents; Chelating Agents; Cuprizone; Cytokines; Demyelinating Diseases; Disease Models, Animal; Encephalitis; Enzyme Inhibitors; Glial Fibrillary Acidic Protein; Male; Mice; Mice, Inbred C57BL; NF-kappa B; Nitric Oxide Synthase Type III; Sildenafil Citrate | 2015 |
Cytokine regulation by modulation of the NMDA receptor on astrocytes.
Topics: Animals; Astrocytes; Bone Morphogenetic Protein 4; Brain; Cells, Cultured; Cuprizone; Cytokines; Demyelinating Diseases; Encephalitis; Hippocampus; Lipopolysaccharides; Male; Mice; Mice, Inbred C57BL; Receptors, N-Methyl-D-Aspartate; RNA, Messenger | 2016 |
Interleukin-13 immune gene therapy prevents CNS inflammation and demyelination via alternative activation of microglia and macrophages.
Topics: Animals; Antigens, Differentiation; Bone Marrow Transplantation; Cuprizone; Cytokines; Demyelinating Diseases; Disease Models, Animal; Encephalitis; Genetic Therapy; Green Fluorescent Proteins; Interleukin-13; Macrophages; Mice; Mice, Inbred C57BL; Mice, Transgenic; Microglia; Monoamine Oxidase Inhibitors; Myelin Proteins; Nerve Tissue Proteins; Transduction, Genetic | 2016 |
Inflammatory response and chemokine expression in the white matter corpus callosum and gray matter cortex region during cuprizone-induced demyelination.
Topics: Animals; Cerebral Cortex; Chemokine CCL2; Chemokine CCL3; Corpus Callosum; Cuprizone; Demyelinating Diseases; Disease Models, Animal; Encephalitis; Gene Expression Regulation; Male; Mice; Mice, Inbred C57BL; Microglia; Monoamine Oxidase Inhibitors; Multiple Sclerosis; Oligodendroglia | 2012 |
Myelin debris regulates inflammatory responses in an experimental demyelination animal model and multiple sclerosis lesions.
Topics: Adult; Aged; Aged, 80 and over; Analysis of Variance; Animals; Calcium-Binding Proteins; Chelating Agents; Corpus Callosum; Cuprizone; Cytokines; Disease Models, Animal; Dose-Response Relationship, Drug; Encephalitis; Female; Gliosis; Histocompatibility Antigens Class II; Humans; Macrophages; Male; Mice; Mice, Inbred C57BL; Microfilament Proteins; Microglia; Middle Aged; Multiple Sclerosis; Myelin Proteolipid Protein; Myelin Sheath; Nerve Fibers, Myelinated; Nerve Tissue Proteins; RNA, Messenger | 2012 |
Astroglial-derived lymphotoxin-alpha exacerbates inflammation and demyelination, but not remyelination.
Topics: Animals; Astrocytes; Cell Count; Corpus Callosum; Cuprizone; Demyelinating Diseases; Disease Models, Animal; Encephalitis; Gliosis; Glutathione S-Transferase pi; Glutathione Transferase; Inflammation Mediators; Isoenzymes; Lymphotoxin-alpha; Male; Mice; Mice, Inbred C57BL; Mice, Knockout; Microglia; Nerve Regeneration; Oligodendroglia; Signal Transduction; Time Factors; Tumor Necrosis Factor-alpha | 2005 |
TNF superfamily member TWEAK exacerbates inflammation and demyelination in the cuprizone-induced model.
Topics: Animals; Astrocytes; Chelating Agents; Copper; Cuprizone; Cytokine TWEAK; Demyelinating Diseases; Double-Blind Method; Encephalitis; Male; Mice; Mice, Inbred C57BL; Mice, Knockout; Microglia; Myelin Sheath; Oligodendroglia; Receptors, Tumor Necrosis Factor; RNA, Messenger; Tumor Necrosis Factors; TWEAK Receptor | 2008 |