cuprizone has been researched along with Disease Exacerbation in 8 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 4 (50.00) | 29.6817 |
| 2010's | 2 (25.00) | 24.3611 |
| 2020's | 2 (25.00) | 2.80 |
| Authors | Studies |
|---|---|
| Almuslehi, MSM; Coorssen, JR; Mahns, DA; Sen, MK; Shortland, PJ | 1 |
| Buonvicino, D; Chiarugi, A; Ranieri, G | 1 |
| Amor, S; Hochstrasser, T; Kipp, M; Nyamoya, S | 1 |
| Gudi, V; Kotsiari, A; Koutsoudaki, PN; Moharregh-Khiabani, D; Skripuletz, T; Skuljec, J; Stangel, M; Trebst, C | 1 |
| Armstrong, RC; Budde, MD; Chen, CI; Cross, AH; McDaniel, DP; Song, SK; Tobin, JE; Trinkaus, K; Xie, M | 1 |
| Goldman, JE; Hostettler, JD; Mason, JL; Matsushima, GK; Morell, P; Suzuki, K; Toews, A | 1 |
| Chaplin, DD; Mason, JL; Matsushima, GK; Suzuki, K | 1 |
| Hostettler, J; Jurevics, H; Kleindienst, A; Largent, C; Matsushima, GK; Morell, P; Sammond, DW; Toews, AD | 1 |
2 review(s) available for cuprizone and Disease Exacerbation
| Article | Year |
|---|---|
Revisiting the Pathoetiology of Multiple Sclerosis: Has the Tail Been Wagging the Mouse?
Topics: Animals; Autoimmunity; Central Nervous System; Cuprizone; Demyelinating Diseases; Disease Models, Animal; Disease Progression; Encephalomyelitis, Autoimmune, Experimental; Humans; Mice; Multiple Sclerosis; Oligodendroglia | 2020 |
Multiple sclerosis animal models: a clinical and histopathological perspective.
Topics: Animals; Axons; Cuprizone; Demyelinating Diseases; Disease Models, Animal; Disease Progression; Encephalomyelitis, Autoimmune, Experimental; Humans; Leukoencephalopathies; Multiple Sclerosis; Neurodegenerative Diseases; Spinal Cord | 2017 |
6 other study(ies) available for cuprizone and Disease Exacerbation
| Article | Year |
|---|---|
Cuprizone-Dependent De/Remyelination Responses and Functional Correlates in Mouse Strains Adopted to Model Relapsing, Chronic and Progressive Experimental Autoimmune Encephalomyelitis.
Topics: Animals; Chelating Agents; Chronic Disease; Cuprizone; Demyelinating Diseases; Disease Models, Animal; Disease Progression; Encephalomyelitis, Autoimmune, Experimental; Female; Mice; Mice, Inbred C57BL; Mice, Inbred NOD; Myelin Sheath; Remyelination | 2021 |
Regional differences between grey and white matter in cuprizone induced demyelination.
Topics: Animals; Astrocytes; Brain; Cerebral Cortex; Chelating Agents; Corpus Callosum; Cuprizone; Demyelinating Diseases; Disease Models, Animal; Disease Progression; Gliosis; Male; Mice; Mice, Inbred C57BL; Microglia; Multiple Sclerosis; Nerve Fibers, Myelinated; Nerve Regeneration; Neurotoxins; Oligodendroglia; Stem Cells; Time Factors | 2009 |
Rostrocaudal analysis of corpus callosum demyelination and axon damage across disease stages refines diffusion tensor imaging correlations with pathological features.
Topics: Animals; CD11b Antigen; Corpus Callosum; Cuprizone; Demyelinating Diseases; Diffuse Axonal Injury; Diffusion Magnetic Resonance Imaging; Disease Models, Animal; Disease Progression; Glial Fibrillary Acidic Protein; Indoles; Luminescent Proteins; Macrophages; Male; Mice; Mice, Inbred C57BL; Mice, Transgenic; Microglia; Microscopy, Electron, Transmission; Myelin Proteins; Myelin-Associated Glycoprotein; Myelin-Oligodendrocyte Glycoprotein; Neurofilament Proteins; Statistics as Topic; Time Factors | 2010 |
Oligodendrocytes and progenitors become progressively depleted within chronically demyelinated lesions.
Topics: Animals; Antigens; Apoptosis; Axons; Cell Death; Cell Differentiation; Corpus Callosum; Cuprizone; Demyelinating Diseases; Disease Progression; Glutathione Transferase; Immunohistochemistry; Lectins; Macrophages; Male; Mice; Mice, Inbred C57BL; Mice, Transgenic; Microglia; Myelin Sheath; Oligodendroglia; Protein Isoforms; Proteoglycans; Recombinant Fusion Proteins; RNA; Stem Cells; Time Factors | 2004 |
Interleukin-1beta promotes repair of the CNS.
Topics: Animals; Antigens, Differentiation; Astrocytes; Cell Count; Central Nervous System; Chelating Agents; Corpus Callosum; Cuprizone; Demyelinating Diseases; Disease Progression; Glutathione S-Transferase pi; Glutathione Transferase; Insulin-Like Growth Factor I; Interleukin-1; Isoenzymes; Macrophages; Mice; Mice, Inbred C57BL; Mice, Knockout; Microglia; Myelin Sheath; Oligodendroglia; Regeneration; RNA, Messenger; Stem Cells; Up-Regulation | 2001 |
Alterations in metabolism and gene expression in brain regions during cuprizone-induced demyelination and remyelination.
Topics: Animals; Biomarkers; Brain; Brain Stem; Cerebellum; Cerebrosides; Chelating Agents; Cholesterol; Corpus Callosum; Cuprizone; Demyelinating Diseases; Disease Models, Animal; Disease Progression; Galactosyltransferases; Gene Expression Profiling; Gene Expression Regulation; Mice; Mice, Inbred C3H; Myelin Basic Protein; Myelin Sheath; N-Acylsphingosine Galactosyltransferase; Oligonucleotide Array Sequence Analysis; RNA, Messenger | 2002 |