Compounds > cuprizone

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cuprizone and Aging

cuprizone has been researched along with Aging in 13 studies

Research

Studies (13)

Timeframe	Studies, this research(%)	All Research%
pre-1990	3 (23.08)	18.7374
1990's	0 (0.00)	18.2507
2000's	4 (30.77)	29.6817
2010's	6 (46.15)	24.3611
2020's	0 (0.00)	2.80

Authors

Authors	Studies
Bauer, J; Binder, CJ; Bradl, M; Haider, L; Hametner, S; Lassmann, H; Liblau, RS; Mahad, D; Probert, L; Schuh, C; Smith, KJ; Van Dam, AM; Wimmer, I	1
Imai, S; Stein, LR	1
Colonna, M; Fontana, E; Gilfillan, S; Poliani, PL; Robinette, ML; Wang, Y; Yamanishi, Y	1
Brück, W; Nessler, S; Pfeifenbring, S; Stadelmann, C; Wegner, C	1
Baumgartner, W; Beyer, C; Braun, A; Clarner, T; Hieble, A; Johann, S; Kipp, M; Norkute, A	1
Casaccia-Bonnefil, P; Dupree, J; Franklin, RJ; Li, J; Sandoval, J; Shen, S; Swiss, VA	1
Doucette, JR; Jiao, R; Nazarali, AJ	1
Brück, W; Grønborg, M; Jürgens, T; Kreutzfeldt, M; Kuhlmann, T; Manrique-Hoyos, N; Merkler, D; Schedensack, M; Schrick, C; Simons, M; Urlaub, H	1
Blakemore, WF; Irvine, KA	1
Akita, M; Asou, H; Fukutake, M; Ishige, A; Sakai, R; Seiwa, C; Takeda, S; Tanaka, K; Ueki, T; Watanabe, K; Yagi, T; Yamamoto, M	1
Carey, EM; Freeman, NM	1
Ludwin, SK	1
Hoppel, CL; Tandler, B	1

Reviews

1 review(s) available for cuprizone and Aging

Article	Year
Studies on giant mitochondria. Annals of the New York Academy of Sciences, 1986, Volume: 488 Topics: Aging; Animals; Cuprizone; Ethanol; Hormones; Humans; Microscopy, Electron; Mitochondria; Mitochondria, Liver; Nutrition Disorders; Riboflavin Deficiency	1986

Other Studies

12 other study(ies) available for cuprizone and Aging

Article	Year
Oxidative tissue injury in multiple sclerosis is only partly reflected in experimental disease models. Acta neuropathologica, 2014, Volume: 128, Issue:2 Topics: Aging; Animals; CD4 Antigens; CD8 Antigens; Coronavirus Infections; Cuprizone; Disease Models, Animal; Encephalomyelitis, Autoimmune, Experimental; Gene Expression; Iron; Lipopolysaccharides; Macrophages; Mice, Inbred C57BL; Microglia; Multiple Sclerosis; Myelin-Oligodendrocyte Glycoprotein; Oxidative Stress; Peptide Fragments; Rats; Rats, Inbred Lew; Respiratory Burst; T-Lymphocytes	2014
Specific ablation of Nampt in adult neural stem cells recapitulates their functional defects during aging. The EMBO journal, 2014, Jun-17, Volume: 33, Issue:12 Topics: Adult Stem Cells; Aging; Animals; Bromodeoxyuridine; Cell Culture Techniques; Cell Differentiation; Cell Proliferation; Crosses, Genetic; Cuprizone; Cytokines; Fluorescent Antibody Technique; Mice; Mice, Transgenic; Neural Stem Cells; Nicotinamide Mononucleotide; Nicotinamide Phosphoribosyltransferase	2014
TREM2 sustains microglial expansion during aging and response to demyelination. The Journal of clinical investigation, 2015, Volume: 125, Issue:5 Topics: Aging; Animals; Axons; Brain; Cell Count; Cuprizone; Demyelinating Diseases; Disease Models, Animal; Gene Expression Profiling; Genes, Reporter; Humans; Lipodystrophy; Membrane Glycoproteins; Mice; Mice, Knockout; Microglia; Myelin Sheath; Nerve Tissue Proteins; Osteochondrodysplasias; Phagocytosis; Receptors, Immunologic; Regeneration; Subacute Sclerosing Panencephalitis; Transcription, Genetic	2015
Remyelination After Cuprizone-Induced Demyelination Is Accelerated in Juvenile Mice. Journal of neuropathology and experimental neurology, 2015, Volume: 74, Issue:8 Topics: Aging; Animals; Brain; Cuprizone; Demyelinating Diseases; Disease Models, Animal; Immunohistochemistry; Mice; Mice, Inbred C57BL; Microscopy, Electron, Transmission; Monoamine Oxidase Inhibitors; Nerve Regeneration; Oligodendroglia	2015
Cuprizone treatment induces demyelination and astrocytosis in the mouse hippocampus. Journal of neuroscience research, 2009, May-01, Volume: 87, Issue:6 Topics: Aging; Animals; Astrocytes; Cuprizone; Demyelinating Diseases; Disease Models, Animal; Gliosis; Hippocampus; Immunohistochemistry; Indoles; Male; Mice; Mice, Inbred C57BL; Microglia; Microscopy, Electron; Multiple Sclerosis; Myelin Sheath; Oligodendroglia; Reverse Transcriptase Polymerase Chain Reaction	2009
Age-dependent epigenetic control of differentiation inhibitors is critical for remyelination efficiency. Nature neuroscience, 2008, Volume: 11, Issue:9 Topics: Aging; Animals; Animals, Newborn; Antigens, CD; Antigens, Differentiation, Myelomonocytic; Cell Differentiation; Cells, Cultured; Cerebral Cortex; Cuprizone; Demyelinating Diseases; Disease Models, Animal; Enzyme Inhibitors; Epigenesis, Genetic; Glial Fibrillary Acidic Protein; Histone Deacetylases; Mice; Mice, Inbred C57BL; Microglia; Microscopy, Electron, Transmission; Myelin Proteins; Neurosecretory Systems; Rats; Regeneration; Stem Cells; Time Factors; Transcription Factors; Transcription, Genetic; Valproic Acid	2008
Age-related and cuprizone-induced changes in myelin and transcription factor gene expression and in oligodendrocyte cell densities in the rostral corpus callosum of mice. Cellular and molecular neurobiology, 2010, Volume: 30, Issue:4 Topics: Aging; Animals; Behavior, Animal; Biomarkers; Cell Lineage; Chelating Agents; Corpus Callosum; Cuprizone; Diet; Gene Expression; Humans; Mice; Mice, Inbred C57BL; Myelin Sheath; Oligodendroglia; Stem Cells; Transcription Factors	2010
Late motor decline after accomplished remyelination: impact for progressive multiple sclerosis. Annals of neurology, 2012, Volume: 71, Issue:2 Topics: Aging; Animals; Axons; Cuprizone; Demyelinating Diseases; Disease Models, Animal; Humans; Mice; Mice, Inbred C57BL; Monoamine Oxidase Inhibitors; Motor Skills; Multiple Sclerosis, Chronic Progressive; Myelin Sheath; Time Factors	2012
Age increases axon loss associated with primary demyelination in cuprizone-induced demyelination in C57BL/6 mice. Journal of neuroimmunology, 2006, Volume: 175, Issue:1-2 Topics: Aging; Animals; Axons; Corpus Callosum; Cuprizone; Demyelinating Diseases; Female; Mice; Mice, Inbred C57BL	2006
Restoration of FcRgamma/Fyn signaling repairs central nervous system demyelination. Journal of neuroscience research, 2007, Volume: 85, Issue:5 Topics: Aging; Animals; Anti-Inflammatory Agents; Central Nervous System; Cuprizone; Demyelinating Diseases; Disease Models, Animal; Drugs, Chinese Herbal; Female; Male; Mice; Mice, Inbred C57BL; Mice, Knockout; Monoamine Oxidase Inhibitors; Multiple Sclerosis; Myelin Basic Protein; Myelin Sheath; Nerve Regeneration; Neuroprotective Agents; Neurotoxins; Proto-Oncogene Proteins c-fyn; Receptors, IgG; Recovery of Function; Signal Transduction	2007
Biochemical changes in Cuprizone-induced spongiform encephalopathy. I. Changes in the activities of 2',3'-cyclic nucleotide 3'-phosphohydrolase, oligodendroglial ceramide galactosyl transferase, and the hydrolysis of the alkenyl group of alkenyl, acyl-gly Neurochemical research, 1983, Volume: 8, Issue:8 Topics: 2',3'-Cyclic Nucleotide 3'-Phosphodiesterase; 2',3'-Cyclic-Nucleotide Phosphodiesterases; Aging; Animals; Brain; Cuprizone; Cyclohexanes; Demyelinating Diseases; Galactolipids; Galactosyltransferases; Glycolipids; Hydrolases; Male; Mice; N-Acylsphingosine Galactosyltransferase; Oligodendroglia; Phosphoric Diester Hydrolases; Plasmalogens	1983
Chronic demyelination inhibits remyelination in the central nervous system. An analysis of contributing factors. Laboratory investigation; a journal of technical methods and pathology, 1980, Volume: 43, Issue:4 Topics: Aging; Animals; Astrocytes; Axons; Central Nervous System; Cerebellum; Chronic Disease; Cuprizone; Demyelinating Diseases; Male; Mice; Microscopy, Electron; Myelin Sheath; Oligodendroglia	1980