cuprizone and Aging
cuprizone has been researched along with Aging in 13 studies
Research
Studies (13)
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 3 (23.08) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 4 (30.77) | 29.6817 |
| 2010's | 6 (46.15) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
Authors
| Authors | Studies |
|---|---|
| Bauer, J; Binder, CJ; Bradl, M; Haider, L; Hametner, S; Lassmann, H; Liblau, RS; Mahad, D; Probert, L; Schuh, C; Smith, KJ; Van Dam, AM; Wimmer, I | 1 |
| Imai, S; Stein, LR | 1 |
| Colonna, M; Fontana, E; Gilfillan, S; Poliani, PL; Robinette, ML; Wang, Y; Yamanishi, Y | 1 |
| Brück, W; Nessler, S; Pfeifenbring, S; Stadelmann, C; Wegner, C | 1 |
| Baumgartner, W; Beyer, C; Braun, A; Clarner, T; Hieble, A; Johann, S; Kipp, M; Norkute, A | 1 |
| Casaccia-Bonnefil, P; Dupree, J; Franklin, RJ; Li, J; Sandoval, J; Shen, S; Swiss, VA | 1 |
| Doucette, JR; Jiao, R; Nazarali, AJ | 1 |
| Brück, W; Grønborg, M; Jürgens, T; Kreutzfeldt, M; Kuhlmann, T; Manrique-Hoyos, N; Merkler, D; Schedensack, M; Schrick, C; Simons, M; Urlaub, H | 1 |
| Blakemore, WF; Irvine, KA | 1 |
| Akita, M; Asou, H; Fukutake, M; Ishige, A; Sakai, R; Seiwa, C; Takeda, S; Tanaka, K; Ueki, T; Watanabe, K; Yagi, T; Yamamoto, M | 1 |
| Carey, EM; Freeman, NM | 1 |
| Ludwin, SK | 1 |
| Hoppel, CL; Tandler, B | 1 |
Reviews
1 review(s) available for cuprizone and Aging
| Article | Year |
|---|---|
Studies on giant mitochondria.
Topics: Aging; Animals; Cuprizone; Ethanol; Hormones; Humans; Microscopy, Electron; Mitochondria; Mitochondria, Liver; Nutrition Disorders; Riboflavin Deficiency | 1986 |
Other Studies
12 other study(ies) available for cuprizone and Aging
| Article | Year |
|---|---|
Oxidative tissue injury in multiple sclerosis is only partly reflected in experimental disease models.
Topics: Aging; Animals; CD4 Antigens; CD8 Antigens; Coronavirus Infections; Cuprizone; Disease Models, Animal; Encephalomyelitis, Autoimmune, Experimental; Gene Expression; Iron; Lipopolysaccharides; Macrophages; Mice, Inbred C57BL; Microglia; Multiple Sclerosis; Myelin-Oligodendrocyte Glycoprotein; Oxidative Stress; Peptide Fragments; Rats; Rats, Inbred Lew; Respiratory Burst; T-Lymphocytes | 2014 |
Specific ablation of Nampt in adult neural stem cells recapitulates their functional defects during aging.
Topics: Adult Stem Cells; Aging; Animals; Bromodeoxyuridine; Cell Culture Techniques; Cell Differentiation; Cell Proliferation; Crosses, Genetic; Cuprizone; Cytokines; Fluorescent Antibody Technique; Mice; Mice, Transgenic; Neural Stem Cells; Nicotinamide Mononucleotide; Nicotinamide Phosphoribosyltransferase | 2014 |
TREM2 sustains microglial expansion during aging and response to demyelination.
Topics: Aging; Animals; Axons; Brain; Cell Count; Cuprizone; Demyelinating Diseases; Disease Models, Animal; Gene Expression Profiling; Genes, Reporter; Humans; Lipodystrophy; Membrane Glycoproteins; Mice; Mice, Knockout; Microglia; Myelin Sheath; Nerve Tissue Proteins; Osteochondrodysplasias; Phagocytosis; Receptors, Immunologic; Regeneration; Subacute Sclerosing Panencephalitis; Transcription, Genetic | 2015 |
Remyelination After Cuprizone-Induced Demyelination Is Accelerated in Juvenile Mice.
Topics: Aging; Animals; Brain; Cuprizone; Demyelinating Diseases; Disease Models, Animal; Immunohistochemistry; Mice; Mice, Inbred C57BL; Microscopy, Electron, Transmission; Monoamine Oxidase Inhibitors; Nerve Regeneration; Oligodendroglia | 2015 |
Cuprizone treatment induces demyelination and astrocytosis in the mouse hippocampus.
Topics: Aging; Animals; Astrocytes; Cuprizone; Demyelinating Diseases; Disease Models, Animal; Gliosis; Hippocampus; Immunohistochemistry; Indoles; Male; Mice; Mice, Inbred C57BL; Microglia; Microscopy, Electron; Multiple Sclerosis; Myelin Sheath; Oligodendroglia; Reverse Transcriptase Polymerase Chain Reaction | 2009 |
Age-dependent epigenetic control of differentiation inhibitors is critical for remyelination efficiency.
Topics: Aging; Animals; Animals, Newborn; Antigens, CD; Antigens, Differentiation, Myelomonocytic; Cell Differentiation; Cells, Cultured; Cerebral Cortex; Cuprizone; Demyelinating Diseases; Disease Models, Animal; Enzyme Inhibitors; Epigenesis, Genetic; Glial Fibrillary Acidic Protein; Histone Deacetylases; Mice; Mice, Inbred C57BL; Microglia; Microscopy, Electron, Transmission; Myelin Proteins; Neurosecretory Systems; Rats; Regeneration; Stem Cells; Time Factors; Transcription Factors; Transcription, Genetic; Valproic Acid | 2008 |
Age-related and cuprizone-induced changes in myelin and transcription factor gene expression and in oligodendrocyte cell densities in the rostral corpus callosum of mice.
Topics: Aging; Animals; Behavior, Animal; Biomarkers; Cell Lineage; Chelating Agents; Corpus Callosum; Cuprizone; Diet; Gene Expression; Humans; Mice; Mice, Inbred C57BL; Myelin Sheath; Oligodendroglia; Stem Cells; Transcription Factors | 2010 |
Late motor decline after accomplished remyelination: impact for progressive multiple sclerosis.
Topics: Aging; Animals; Axons; Cuprizone; Demyelinating Diseases; Disease Models, Animal; Humans; Mice; Mice, Inbred C57BL; Monoamine Oxidase Inhibitors; Motor Skills; Multiple Sclerosis, Chronic Progressive; Myelin Sheath; Time Factors | 2012 |
Age increases axon loss associated with primary demyelination in cuprizone-induced demyelination in C57BL/6 mice.
Topics: Aging; Animals; Axons; Corpus Callosum; Cuprizone; Demyelinating Diseases; Female; Mice; Mice, Inbred C57BL | 2006 |
Restoration of FcRgamma/Fyn signaling repairs central nervous system demyelination.
Topics: Aging; Animals; Anti-Inflammatory Agents; Central Nervous System; Cuprizone; Demyelinating Diseases; Disease Models, Animal; Drugs, Chinese Herbal; Female; Male; Mice; Mice, Inbred C57BL; Mice, Knockout; Monoamine Oxidase Inhibitors; Multiple Sclerosis; Myelin Basic Protein; Myelin Sheath; Nerve Regeneration; Neuroprotective Agents; Neurotoxins; Proto-Oncogene Proteins c-fyn; Receptors, IgG; Recovery of Function; Signal Transduction | 2007 |
Biochemical changes in Cuprizone-induced spongiform encephalopathy. I. Changes in the activities of 2',3'-cyclic nucleotide 3'-phosphohydrolase, oligodendroglial ceramide galactosyl transferase, and the hydrolysis of the alkenyl group of alkenyl, acyl-gly
Topics: 2',3'-Cyclic Nucleotide 3'-Phosphodiesterase; 2',3'-Cyclic-Nucleotide Phosphodiesterases; Aging; Animals; Brain; Cuprizone; Cyclohexanes; Demyelinating Diseases; Galactolipids; Galactosyltransferases; Glycolipids; Hydrolases; Male; Mice; N-Acylsphingosine Galactosyltransferase; Oligodendroglia; Phosphoric Diester Hydrolases; Plasmalogens | 1983 |
Chronic demyelination inhibits remyelination in the central nervous system. An analysis of contributing factors.
Topics: Aging; Animals; Astrocytes; Axons; Central Nervous System; Cerebellum; Chronic Disease; Cuprizone; Demyelinating Diseases; Male; Mice; Microscopy, Electron; Myelin Sheath; Oligodendroglia | 1980 |