cuprizone has been researched along with Acute Relapsing Multiple Sclerosis in 4 studies
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 0 (0.00) | 18.2507 |
2000's | 1 (25.00) | 29.6817 |
2010's | 1 (25.00) | 24.3611 |
2020's | 2 (50.00) | 2.80 |
Authors | Studies |
---|---|
Chen, L; Feng, Z; Gao, Z; Liu, Z; Yang, K; Yang, Y; Yao, R; Zhang, C | 1 |
Bø, L; Lereim, RR; Myhr, KM; Nystad, AE; Oveland, E; Torkildsen, Ø; Wergeland, S | 1 |
Cai, QY; Chen, XS; Ma, ZY; Yao, ZX; Zhan, XL; Zhang, M | 1 |
Balasubramaniyan, V; Boddeke, E; Copray, S; de Bruijn, J; Levenga, J; Liem, R | 1 |
4 other study(ies) available for cuprizone and Acute Relapsing Multiple Sclerosis
Article | Year |
---|---|
C1q inhibits differentiation of oligodendrocyte progenitor cells via Wnt/β-catenin signaling activation in a cuprizone-induced mouse model of multiple sclerosis.
Topics: Adult; Animals; Cell Differentiation; Cells, Cultured; Chelating Agents; Complement C1q; Cuprizone; Disease Models, Animal; Female; Gene Knockdown Techniques; Humans; Male; Mice; Mice, Inbred C57BL; Middle Aged; Multiple Sclerosis, Relapsing-Remitting; Oligodendrocyte Precursor Cells; Rats; Rats, Sprague-Dawley; Wnt Signaling Pathway | 2022 |
Fingolimod downregulates brain sphingosine-1-phosphate receptor 1 levels but does not promote remyelination or neuroprotection in the cuprizone model.
Topics: Animals; Brain; Cuprizone; Female; Fingolimod Hydrochloride; Mice; Mice, Inbred C57BL; Multiple Sclerosis, Relapsing-Remitting; Neuroprotection; Random Allocation; Remyelination; Sphingosine 1 Phosphate Receptor Modulators; Sphingosine-1-Phosphate Receptors | 2020 |
Thyroid hormone alleviates demyelination induced by cuprizone through its role in remyelination during the remission period.
Topics: Animals; Astrocytes; Axons; Cell Differentiation; Cell Lineage; Cell Proliferation; Cuprizone; Demyelinating Diseases; Disease Models, Animal; Humans; Male; Mice; Mice, Inbred C57BL; Microglia; Multiple Sclerosis, Relapsing-Remitting; Myelin Sheath; Oligodendroglia; Thyroid Hormones | 2015 |
Olig2 overexpression induces the in vitro differentiation of neural stem cells into mature oligodendrocytes.
Topics: Animals; Basic Helix-Loop-Helix Transcription Factors; Cell Differentiation; Chelating Agents; Cuprizone; Disease Models, Animal; Gene Expression; Homeobox Protein Nkx-2.2; Homeodomain Proteins; In Vitro Techniques; Mice; Mice, Inbred C57BL; Microscopy, Electron; Multiple Sclerosis, Relapsing-Remitting; Myelin Sheath; Nerve Tissue Proteins; Neurons; Oligodendrocyte Transcription Factor 2; Oligodendroglia; Stem Cells; Transcription Factors; Transfection; Zebrafish Proteins | 2006 |