cupric chloride has been researched along with Neuroblastoma in 4 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 1 (25.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 2 (50.00) | 29.6817 |
| 2010's | 1 (25.00) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Anandhan, A; Bohovych, I; Franco, R; Griggs, AM; Khalimonchuk, O; Lee, J; Reyes-Reyes, EM; Rochet, JC; Rodriguez-Rocha, H; Seravalli, J; Stanciu, LA; Zavala-Flores, L | 1 |
| Fukuhara, K; Hiraku, Y; Kobayashi, H; Murata, M; Oikawa, S; Tada-Oikawa, S; Yada, Y | 1 |
| Katayama, T; Manabe, T; Matsuzaki, S; Meshitsuka, S; Miyata, S; Nishikawa, A; Okuda, H; Tohyama, M; Yanagita, T; Yasuda, Y | 1 |
| Eguchi, G; Masuda, A | 1 |
4 other study(ies) available for cupric chloride and Neuroblastoma
| Article | Year |
|---|---|
Overexpression of alpha-synuclein at non-toxic levels increases dopaminergic cell death induced by copper exposure via modulation of protein degradation pathways.
Topics: alpha-Synuclein; Animals; Apoptosis; Caspases; Cells, Cultured; Copper; Cysteine Proteinase Inhibitors; Dopaminergic Neurons; Embryo, Mammalian; Gene Expression Regulation; Humans; Leupeptins; Mesencephalon; Mutation; Neuroblastoma; Proteolysis; Rats; Rats, Sprague-Dawley; Signal Transduction; Time Factors; Tyrosine 3-Monooxygenase | 2015 |
The mechanisms of oxidative DNA damage and apoptosis induced by norsalsolinol, an endogenous tetrahydroisoquinoline derivative associated with Parkinson's disease.
Topics: 8-Hydroxy-2'-Deoxyguanosine; Apoptosis; Autoradiography; Benzoquinones; Caspase 3; Cell Line, Tumor; Copper; Cytochromes c; Deoxyguanosine; DNA Damage; Dose-Response Relationship, Drug; Free Radical Scavengers; Humans; NAD; Neuroblastoma; Phenanthrolines; Phosphorus Isotopes; Salsoline Alkaloids; Tetrahydroisoquinolines; Tetrazolium Salts; Thiazoles; Time Factors; Tyrosine 3-Monooxygenase | 2009 |
Metals accelerate production of the aberrant splicing isoform of the presenilin-2.
Topics: Alternative Splicing; Aluminum Compounds; Alzheimer Disease; Antiviral Agents; Cell Death; Cell Hypoxia; Cell Line, Tumor; Copper; Copper Sulfate; Dose-Response Relationship, Drug; Endoplasmic Reticulum; Gene Expression Regulation; HMGA1a Protein; Humans; Iron Compounds; Membrane Proteins; Metals; Neuroblastoma; Organometallic Compounds; Oxidative Stress; Presenilin-2; Pyrones; Time Factors; Tunicamycin | 2004 |
Phenylthiourea enhances Cu cytotoxicity in cell cultures: its mode of action.
Topics: Animals; Carcinoma, Squamous Cell; Cations, Divalent; Cell Division; Cell Line; Cell Survival; Chick Embryo; Copper; Dose-Response Relationship, Drug; Drug Interactions; Epithelium; Humans; Kinetics; Melanoma; Mice; Neuroblastoma; Phenylthiourea; Pigmentation; Thiourea | 1984 |