cupric chloride has been researched along with Carcinoma, Hepatocellular in 4 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 3 (75.00) | 29.6817 |
| 2010's | 1 (25.00) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Aggarwal, A; Bhatt, M; Chandhok, G; Horvath, J; Schmidt, HH; Zibert, A | 1 |
| Kennedy, DC; Lyn, RK; Pezacki, JP | 1 |
| Babushkin, T; Hait-Darshan, R; Malik, Z | 1 |
| Barth, S; Camenisch, G; Eckhardt, K; Flamme, I; Katschinski, DM; Linden, T; Martin, F; Mukhopadhyay, CK; Oehme, F; Tröger, J; Wenger, RH | 1 |
4 other study(ies) available for cupric chloride and Carcinoma, Hepatocellular
| Article | Year |
|---|---|
Functional analysis and drug response to zinc and D-penicillamine in stable ATP7B mutant hepatic cell lines.
Topics: Adenosine Triphosphatases; Apoptosis; Carcinoma, Hepatocellular; Cation Transport Proteins; Cell Survival; Chelating Agents; Chlorides; Copper; Copper-Transporting ATPases; Dose-Response Relationship, Drug; Genotype; Hep G2 Cells; Hepatocytes; Hepatolenticular Degeneration; Humans; Liver Neoplasms; Mutation; Penicillamine; Phenotype; Protein Transport; Transfection; Zinc Compounds | 2016 |
Cellular lipid metabolism is influenced by the coordination environment of copper.
Topics: Carcinoma, Hepatocellular; Cell Line, Tumor; Copper; Edetic Acid; Histidine; Humans; Lipid Metabolism; Liver; Organometallic Compounds | 2009 |
Regulation of heme synthesis and proteasomal activity by copper: possible implications for Wilson's disease.
Topics: Adenosine Triphosphatases; Carcinoma, Hepatocellular; Cation Transport Proteins; Cell Line, Tumor; Cell Survival; Copper; Copper-Transporting ATPases; Disulfiram; Down-Regulation; Enzyme Inhibitors; Heme; Hepatocytes; Hepatolenticular Degeneration; Humans; Hydroxymethylbilane Synthase; Liver Neoplasms; Porphobilinogen Synthase; Proteasome Endopeptidase Complex; Protoporphyrins; Reactive Oxygen Species; Ubiquitin | 2009 |
Copper-dependent activation of hypoxia-inducible factor (HIF)-1: implications for ceruloplasmin regulation.
Topics: Animals; Carcinoma, Hepatocellular; Cell Line; Cell Nucleus; Cell Proliferation; Ceruloplasmin; CHO Cells; Coloring Agents; Copper; Cricetinae; DNA-Binding Proteins; Dose-Response Relationship, Drug; Gene Expression Regulation; Genes, Reporter; Glucose Transporter Type 1; HeLa Cells; Humans; Hypoxia; Hypoxia-Inducible Factor 1; Hypoxia-Inducible Factor 1, alpha Subunit; Immunoblotting; Iron; Liver; Luciferases; Mice; Microscopy, Fluorescence; Monosaccharide Transport Proteins; Nuclear Proteins; Oxygen; Procollagen-Proline Dioxygenase; Promoter Regions, Genetic; RNA, Messenger; Tetrazolium Salts; Thiazoles; Transcription Factors; Transcription, Genetic; Transfection; Vascular Endothelial Growth Factor A | 2005 |