cupric chloride has been researched along with Acute Confusional Senile Dementia in 8 studies
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 0 (0.00) | 18.2507 |
2000's | 5 (62.50) | 29.6817 |
2010's | 3 (37.50) | 24.3611 |
2020's | 0 (0.00) | 2.80 |
Authors | Studies |
---|---|
Hsu, HW; Kilian, JG; Kitazawa, M; Lim, SL; Rodriguez-Ortiz, CJ; Vidal, J; Zumkehr, J | 1 |
Kang, J; Kerr, RA; Kim, KS; Korshavn, KJ; Lee, HJ; Lim, MH; Nam, Y; Ozbil, M; Paul, TJ; Prabhakar, R; Ramamoorthy, A; Ruotolo, BT; Youn, IS | 1 |
Braymer, JJ; Choi, JS; DeToma, AS; Lim, MH | 1 |
Katayama, T; Manabe, T; Matsuzaki, S; Meshitsuka, S; Miyata, S; Nishikawa, A; Okuda, H; Tohyama, M; Yanagita, T; Yasuda, Y | 1 |
Andrisano, V; Bartolini, M; Bolognesi, ML; Cavalli, A; Melchiorre, C; Recanatini, M; Rosini, M; Valgimigli, L | 1 |
Lee, JH; Paik, SR; Shin, HJ | 1 |
Barrow, CJ; Beyreuther, K; Bush, AI; Cappai, R; Huang, X; Jobling, MF; Masters, CL; White, AR | 1 |
Jung, MW; Mook-Jung, I; Mori, H; Park, IH | 1 |
8 other study(ies) available for cupric chloride and Acute Confusional Senile Dementia
Article | Year |
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Copper-Induced Upregulation of MicroRNAs Directs the Suppression of Endothelial LRP1 in Alzheimer's Disease Model.
Topics: Alzheimer Disease; Animals; Brain; Cell Survival; Copper; Disease Models, Animal; Endothelial Cells; Female; Humans; Low Density Lipoprotein Receptor-Related Protein-1; Male; Maze Learning; Mice; Mice, Inbred C57BL; Mice, Transgenic; MicroRNAs; Microvessels; Spatial Memory; Transfection; Up-Regulation | 2019 |
Structural and Mechanistic Insights into Development of Chemical Tools to Control Individual and Inter-Related Pathological Features in Alzheimer's Disease.
Topics: Alzheimer Disease; Amino Acid Sequence; Amyloid beta-Peptides; Cell Line; Cell Survival; Chlorides; Copper; Humans; Metals; Oxidative Stress; Protein Binding; Protein Structure, Tertiary; Small Molecule Libraries; Spectrometry, Mass, Electrospray Ionization; Spectrophotometry; Zinc Compounds | 2017 |
Myricetin: a naturally occurring regulator of metal-induced amyloid-β aggregation and neurotoxicity.
Topics: Alzheimer Disease; Amino Acid Motifs; Amyloid beta-Peptides; Cell Line, Tumor; Cell Survival; Chlorides; Copper; Flavonoids; Humans; Molecular Structure; Zinc Compounds | 2011 |
Metals accelerate production of the aberrant splicing isoform of the presenilin-2.
Topics: Alternative Splicing; Aluminum Compounds; Alzheimer Disease; Antiviral Agents; Cell Death; Cell Hypoxia; Cell Line, Tumor; Copper; Copper Sulfate; Dose-Response Relationship, Drug; Endoplasmic Reticulum; Gene Expression Regulation; HMGA1a Protein; Humans; Iron Compounds; Membrane Proteins; Metals; Neuroblastoma; Organometallic Compounds; Oxidative Stress; Presenilin-2; Pyrones; Time Factors; Tunicamycin | 2004 |
Multi-target-directed drug design strategy: from a dual binding site acetylcholinesterase inhibitor to a trifunctional compound against Alzheimer's disease.
Topics: Acetylcholinesterase; Alzheimer Disease; Amyloid beta-Peptides; Binding Sites; Butyrylcholinesterase; Chelating Agents; Cholinesterase Inhibitors; Copper; Drug Design; Ferric Compounds; Humans; Ligands; Models, Molecular; Peptide Fragments; Structure-Activity Relationship; Tacrine; Thermodynamics | 2007 |
Metal-catalyzed oxidation of alpha-synuclein in the presence of Copper(II) and hydrogen peroxide.
Topics: Alkylating Agents; alpha-Synuclein; Alzheimer Disease; Catalysis; Chlorides; Copper; Dicyclohexylcarbodiimide; Dose-Response Relationship, Drug; Electrophoresis, Polyacrylamide Gel; Ferric Compounds; Humans; Hydrogen Peroxide; Metals; Nerve Tissue Proteins; Oxidation-Reduction; Quinolines; Recombinant Proteins; Spectrometry, Fluorescence; Synucleins; Time Factors | 2000 |
Homocysteine potentiates copper- and amyloid beta peptide-mediated toxicity in primary neuronal cultures: possible risk factors in the Alzheimer's-type neurodegenerative pathways.
Topics: Alzheimer Disease; Amyloid beta-Protein Precursor; Animals; Cell Survival; Cells, Cultured; Cerebral Cortex; Copper; Dose-Response Relationship, Drug; Drug Synergism; Homocysteine; Humans; Kinetics; Mice; Mice, Inbred Strains; Neurons; Neurotoxins; Risk Factors | 2001 |
Zinc enhances synthesis of presenilin 1 in mouse primary cortical culture.
Topics: Alzheimer Disease; Animals; Calcium Chloride; Cell Death; Cells, Cultured; Cerebral Cortex; Chelating Agents; Copper; Dose-Response Relationship, Drug; Etoposide; Ferrous Compounds; Hydrogen Peroxide; Membrane Proteins; Mice; Mice, Inbred ICR; Nucleic Acid Synthesis Inhibitors; Oxidants; Peptide Fragments; Presenilin-1; Zinc | 2001 |