creatine has been researched along with Cerebellar Diseases in 8 studies
Cerebellar Diseases: Diseases that affect the structure or function of the cerebellum. Cardinal manifestations of cerebellar dysfunction include dysmetria, GAIT ATAXIA, and MUSCLE HYPOTONIA.
| Excerpt | Relevance | Reference |
|---|---|---|
| "Cerebral creatine deficiency syndromes (CCDSs) are a group of inborn errors of creatine metabolism comprising two autosomal recessive disorders that affect the biosynthesis of creatine--i." | 8.84 | Cerebral creatine deficiency syndromes: clinical aspects, treatment and pathophysiology. ( Salomons, GS; Schutz, PW; Stockler, S, 2007) |
| "Cerebral creatine deficiency syndromes (CCDSs) are a group of inborn errors of creatine metabolism comprising two autosomal recessive disorders that affect the biosynthesis of creatine--i." | 4.84 | Cerebral creatine deficiency syndromes: clinical aspects, treatment and pathophysiology. ( Salomons, GS; Schutz, PW; Stockler, S, 2007) |
| "(a) Hamartomas showed higher N-acetyl aspartate/creatine, creatine/choline, and N-acetyl aspartate/choline ratios than gliomas." | 3.69 | Proton MR spectroscopy in patients with neurofibromatosis type 1: evaluation of hamartomas and clinical correlation. ( Castillo, M; Green, C; Greenwood, R; Kwock, L; Schiro, S; Smith, K; Wilson, D, 1995) |
| "Creatine (Cr) is a nitrogenous organic acid and plays roles such as fast phosphate energy buffer to replenish ATP, osmolyte, antioxidant, neuromodulator, and as a compound with anabolic and ergogenic properties in muscle." | 1.72 | Creatine transporter-deficient rat model shows motor dysfunction, cerebellar alterations, and muscle creatine deficiency without muscle atrophy. ( Binz, PA; Braissant, O; Cudalbu, C; De Bock, K; Duran-Trio, L; Fernandes-Pires, G; Grosse, J; Roux-Petronelli, C; Sandi, C; Soro-Arnaiz, I, 2022) |
| "MR studies of these infants showed obstructive hydrocephalus caused by mass effect produced by an enlarged cerebellum." | 1.30 | Imaging studies in a unique familial dysmyelinating disorder. ( Duhaime, AC; Gripp, KW; Molloy, PT; Muenke, M; Rorke, LB; Schut, L; Tucker, SH; Wang, ZJ; Zackai, EH; Zimmerman, RA, 1998) |
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 4 (50.00) | 18.2507 |
| 2000's | 3 (37.50) | 29.6817 |
| 2010's | 0 (0.00) | 24.3611 |
| 2020's | 1 (12.50) | 2.80 |
| Authors | Studies |
|---|---|
| Duran-Trio, L | 1 |
| Fernandes-Pires, G | 1 |
| Grosse, J | 1 |
| Soro-Arnaiz, I | 1 |
| Roux-Petronelli, C | 1 |
| Binz, PA | 1 |
| De Bock, K | 1 |
| Cudalbu, C | 1 |
| Sandi, C | 1 |
| Braissant, O | 1 |
| Stockler, S | 1 |
| Schutz, PW | 1 |
| Salomons, GS | 1 |
| Guerrini, L | 1 |
| Belli, G | 1 |
| Cellerini, M | 1 |
| Nencini, P | 1 |
| Mascalchi, M | 1 |
| Melek, E | 1 |
| Ozyer, U | 1 |
| Erol, I | 1 |
| Alehan, F | 1 |
| Muhteşem Ağildere, A | 1 |
| Castillo, M | 1 |
| Green, C | 1 |
| Kwock, L | 1 |
| Smith, K | 1 |
| Wilson, D | 1 |
| Schiro, S | 1 |
| Greenwood, R | 1 |
| Tedeschi, G | 1 |
| Bertolino, A | 1 |
| Massaquoi, SG | 1 |
| Campbell, G | 1 |
| Patronas, NJ | 1 |
| Bonavita, S | 1 |
| Barnett, AS | 1 |
| Alger, JR | 1 |
| Hallett, M | 1 |
| Gripp, KW | 1 |
| Zimmerman, RA | 1 |
| Wang, ZJ | 1 |
| Rorke, LB | 1 |
| Duhaime, AC | 1 |
| Schut, L | 1 |
| Molloy, PT | 1 |
| Tucker, SH | 1 |
| Zackai, EH | 1 |
| Muenke, M | 1 |
| Terakawa, H | 1 |
| Abe, K | 1 |
| Watanabe, Y | 1 |
| Nakamura, M | 1 |
| Fujita, N | 1 |
| Hirabuki, N | 1 |
| Yanagihara, T | 1 |
1 review available for creatine and Cerebellar Diseases
| Article | Year |
|---|---|
Cerebral creatine deficiency syndromes: clinical aspects, treatment and pathophysiology.
Topics: Adult; Amidinotransferases; Animals; Brain Diseases, Metabolic, Inborn; Cerebellar Diseases; Child; | 2007 |
7 other studies available for creatine and Cerebellar Diseases
| Article | Year |
|---|---|
Creatine transporter-deficient rat model shows motor dysfunction, cerebellar alterations, and muscle creatine deficiency without muscle atrophy.
Topics: Animals; Cerebellar Diseases; Cerebellum; Creatine; Guanidinoacetate N-Methyltransferase; Humans; Me | 2022 |
Proton MR spectroscopy of cerebellitis.
Topics: Acute-Phase Reaction; Adult; Aspartic Acid; Cerebellar Diseases; Choline; Creatine; Humans; Magnetic | 2002 |
+H-proton-magnetic resonance spectroscopic findings in a patient with acute hemicerebellitis presenting without localized signs: a case report.
Topics: Acute Disease; Adolescent; Aspartic Acid; Cerebellar Diseases; Choline; Cranial Fossa, Posterior; Cr | 2006 |
Proton MR spectroscopy in patients with neurofibromatosis type 1: evaluation of hamartomas and clinical correlation.
Topics: Adolescent; Adult; Aspartic Acid; Astrocytoma; Brain; Brain Diseases; Brain Neoplasms; Cerebellar Di | 1995 |
Proton magnetic resonance spectroscopic imaging in patients with cerebellar degeneration.
Topics: Adult; Aged; Aspartic Acid; Atrophy; Cerebellar Diseases; Cerebellum; Choline; Creatine; Humans; Lac | 1996 |
Imaging studies in a unique familial dysmyelinating disorder.
Topics: Adolescent; Adult; Aspartic Acid; Brain; Canavan Disease; Cerebellar Diseases; Cerebral Cortex; Chol | 1998 |
Proton magnetic resonance spectroscopy (1H MRS) in patients with sporadic cerebellar degeneration.
Topics: Adult; Aged; Aspartic Acid; Atrophy; Cerebellar Diseases; Cerebellum; Choline; Creatine; Disease Pro | 1999 |