Page last updated: 2024-08-26

cp 96345 and Inhalation Injury, Smoke

cp 96345 has been researched along with Inhalation Injury, Smoke in 2 studies

Research

Studies (2)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's2 (100.00)29.6817
2010's0 (0.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Chang, LC; Chen, WC; Li, PC; Lin, SC1
Kou, YR; Lin, YS1

Other Studies

2 other study(ies) available for cp 96345 and Inhalation Injury, Smoke

ArticleYear
Substance P acts via the neurokinin receptor 1 to elicit bronchoconstriction, oxidative stress, and upregulated ICAM-1 expression after oil smoke exposure.
    American journal of physiology. Lung cellular and molecular physiology, 2008, Volume: 294, Issue:5

    Topics: Animals; Anti-Inflammatory Agents, Non-Steroidal; Antioxidants; Ascorbic Acid; Benzamides; Biphenyl Compounds; Blood Pressure; Bronchoalveolar Lavage Fluid; Bronchoconstriction; Catechin; Intercellular Adhesion Molecule-1; Male; Muscle, Smooth; Neurokinin-1 Receptor Antagonists; Oxidative Stress; Petroleum; Piperidines; Rats; Rats, Sprague-Dawley; Receptors, Neurokinin-1; Receptors, Neurokinin-2; Respiratory Distress Syndrome; Smoke; Smoke Inhalation Injury; Substance P; Up-Regulation

2008
Acute neurogenic airway plasma exudation and edema induced by inhaled wood smoke in guinea pigs: role of tachykinins and hydroxyl radical.
    European journal of pharmacology, 2000, Apr-07, Volume: 394, Issue:1

    Topics: Animals; Biphenyl Compounds; Capillary Permeability; Edema; Guinea Pigs; Hydroxyl Radical; Male; Neprilysin; Nitric Oxide; Receptors, Neurokinin-1; Receptors, Neurokinin-2; Respiratory Tract Diseases; Smoke Inhalation Injury; Tachykinins; Wood

2000