cosyntropin and Infertility--Female

cosyntropin has been researched along with Infertility--Female* in 2 studies

Reviews

1 review(s) available for cosyntropin and Infertility--Female

ArticleYear
Detection and management of late-onset 21-hydroxylase deficiency in women with hyperandrogenism.
    Annales d'endocrinologie, 2010, Volume: 71, Issue:1

    Moderate forms of 21-hydroxylase deficiency (D21OH-NC), the so-called non-classical or late-onset forms are a frequently reported cause of hyperandrogenism in women [1-5]. The purpose of this collective and synthetic work was to provide the endocrinologist, gynecologist and dermatologist with consensual information so as to detect the maximum cases with acceptable cost-benefit ratio and to define the main lines of optimal patient management, given the data currently available in medical literature.

    Topics: Adrenal Hyperplasia, Congenital; Adrenal Insufficiency; Cosyntropin; Female; Genetic Counseling; Glucocorticoids; Hirsutism; Hormone Replacement Therapy; Humans; Hyperandrogenism; Infertility, Female; Steroid 21-Hydroxylase

2010

Other Studies

1 other study(ies) available for cosyntropin and Infertility--Female

ArticleYear
Preliminary investigations into the endocrine systems of subfertile cattle: location of a common lesion (rate-limiting step).
    The Journal of endocrinology, 1987, Volume: 113, Issue:2

    Dairy cows with a variety of clinical conditions were investigated in an attempt to identify the cause(s) of subfertility. Sequential or simultaneous injections of 20 micrograms gonadotrophin-releasing hormone (GnRH), 1 mg oestradiol benzoate and 0.06 mg ACTH(1-24) into five clinical cases of ovarian follicular cysts, two cases of poor body condition and one case of lameness and into control cows revealed a failure in the LH positive-feedback response to oestradiol in all eight clinical cases, but in only two out of twelve control cows. Two of the clinical cases and the two non-responding control cows had high or rising initial progesterone concentrations which would explain the absence of response. All cows studied responded similarly to GnRH and ACTH(1-24). It is suggested that hypothalamus-pituitary control of LH release may involve a rate-limiting step (in the oestradiol positive-feedback system) and that lesions at this point result in subfertility in a variety of clinical situations.

    Topics: Animals; Cattle; Cattle Diseases; Cosyntropin; Estradiol; Feedback; Female; Follicular Cyst; Infertility, Female; Luteinizing Hormone; Pituitary Hormone-Releasing Hormones

1987