Page last updated: 2024-11-06

corticosterone and Glucose Intolerance

corticosterone has been researched along with Glucose Intolerance in 38 studies

Glucose Intolerance: A pathological state in which BLOOD GLUCOSE level is less than approximately 140 mg/100 ml of PLASMA at fasting, and above approximately 200 mg/100 ml plasma at 30-, 60-, or 90-minute during a GLUCOSE TOLERANCE TEST. This condition is seen frequently in DIABETES MELLITUS, but also occurs with other diseases and MALNUTRITION.

Research Excerpts

ExcerptRelevanceReference
"Male rats of SL-water group presented on PND88: glucose intolerance, higher adiposity, plasma triglycerides, free fatty acids, total and low-density lipoprotein (LDL) cholesterol and corticosterone."8.12Lactation overnutrition-induced obesity impairs effects of exogenous corticosterone on energy homeostasis and hypothalamic-pituitary-adrenal axis in male rats. ( de Andrade, FG; de Souza, CF; Leite, CM; Lopes, GM; Martins, AB; Stopa, LRS; Uchoa, ET; Wunderlich, ALM; Zaia, CTBV; Zaia, DAM, 2022)
"Treatment with COC led to elevated fasting blood glucose, insulin, corticosterone, triglycerides (TG), TG/HDL-cholesterol ratio, insulin resistance (IR) and impaired glucose tolerance."7.83Improvement of oral contraceptive-induced glucose dysregulation and dyslipidemia by valproic acid is independent of circulating corticosterone. ( Kim, I; Olatunji, LA; Omolekulo, TE; Usman, TO, 2016)
"Footshock-stress promotes glucose intolerance associated to corticosterone serum level and epididymal white adipose tissue IL-6 concentration increase."7.77Fish oil consumption prevents glucose intolerance and hypercorticosteronemy in footshock-stressed rats. ( Cunha, CA; Eguchi, R; Oller do Nascimento, CM; Oyama, LM; Pisani, LP; Ribeiro, EB; Scarmagnani, FR; Souza, GI; Spadari-Bratfisch, RC, 2011)
" We found that high doses of CORT (100 microg/ml) result in rapid and dramatic increases in weight gain, increased adiposity, elevated plasma leptin, insulin and triglyceride levels, hyperphagia, and decreased home-cage locomotion."7.76Endocrine and physiological changes in response to chronic corticosterone: a potential model of the metabolic syndrome in mouse. ( Bhagat, SM; Bowles, NP; Karatsoreos, IN; McEwen, BS; Pfaff, DW; Weil, ZM, 2010)
"Male rats of SL-water group presented on PND88: glucose intolerance, higher adiposity, plasma triglycerides, free fatty acids, total and low-density lipoprotein (LDL) cholesterol and corticosterone."4.12Lactation overnutrition-induced obesity impairs effects of exogenous corticosterone on energy homeostasis and hypothalamic-pituitary-adrenal axis in male rats. ( de Andrade, FG; de Souza, CF; Leite, CM; Lopes, GM; Martins, AB; Stopa, LRS; Uchoa, ET; Wunderlich, ALM; Zaia, CTBV; Zaia, DAM, 2022)
"Carbonyl reductase 1 (Cbr1), a recently discovered contributor to tissue glucocorticoid metabolism converting corticosterone to 20β-dihydrocorticosterone (20β-DHB), is upregulated in adipose tissue of obese humans and mice and may contribute to cardiometabolic complications of obesity."4.02Carbonyl reductase 1 amplifies glucocorticoid action in adipose tissue and impairs glucose tolerance in lean mice. ( Allan, E; Beck, KR; Bell, RMB; Coutts, A; Denham, SG; Fawkes, A; Homer, NZM; Houtman, R; Koerner, MV; Lee, P; Meijer, OC; Miguelez-Crespo, A; Morgan, RA; Murphy, L; Nixon, M; Odermatt, A; Sharp, MGF; Villalobos, E; Walker, BR, 2021)
" As such, we have studied the impact of HII exposure on glucose tolerance, insulin sensitivity, pancreatic islet morphology, muscle GLUT4 and plasma insulin and corticosterone levels."4.02High-intensity infrasound effects on glucose metabolism in rats. ( Águas, A; Borrecho, G; Brito, J; de Carvalho, AO; Freitas, D; Oliveira, MJ; Oliveira, P; Pereira, GM; Pereira, SS; Santos, M; Tortosa, F, 2021)
" LPF, but not DPF rats, exhibited increased hypothalamic AMPK phosphorylation, glucose intolerance, reduced urinary 6-sulfatoxymelatonin (6-S-Mel) (a metabolite of melatonin) and increased corticosterone levels."3.85Metabolic Impact of Light Phase-Restricted Fructose Consumption Is Linked to Changes in Hypothalamic AMPK Phosphorylation and Melatonin Production in Rats. ( Anhê, GF; Bordin, S; de Araújo, TM; Faria, JA; Ignácio-Souza, LM; Razolli, DS; Souza, DN, 2017)
"These results demonstrates that hypertension and insulin resistance induced by COC is associated with increased cardiac RAS and PAI-1 gene expression, which is likely to be through corticosterone-dependent but not aldosterone-dependent mechanism."3.85Activation of cardiac renin-angiotensin system and plasminogen activator inhibitor-1 gene expressions in oral contraceptive-induced cardiometabolic disorder. ( Kim, IK; Olatunji, LA; Seok, YM; Usman, TO, 2017)
"Treatment with COC led to elevated fasting blood glucose, insulin, corticosterone, triglycerides (TG), TG/HDL-cholesterol ratio, insulin resistance (IR) and impaired glucose tolerance."3.83Improvement of oral contraceptive-induced glucose dysregulation and dyslipidemia by valproic acid is independent of circulating corticosterone. ( Kim, I; Olatunji, LA; Omolekulo, TE; Usman, TO, 2016)
" Nine-month-old HF male offspring was normoglycemic but showed mild glucose intolerance, hyperinsulinemia, and hypercorticosteronemia."3.83Depot- and sex-specific effects of maternal obesity in offspring's adipose tissue. ( Breton, C; Deracinois, B; Eberlé, D; Gabory, A; Guinez, C; Junien, C; Laborie, C; Lecoutre, S; Lesage, J; Panchenko, PE; Vieau, D, 2016)
" Body weight remained elevated for more than 8 wk and was associated with elevated corticosterone and impaired glucose tolerance."3.78Hypothalamic Fkbp51 is induced by fasting, and elevated hypothalamic expression promotes obese phenotypes. ( Dunn-Meynell, A; Fan, X; Isoda, F; Janssen, W; Kleopoulos, SP; Levin, B; Mastaitis, J; McCrimmon, R; Mobbs, CV; Musatov, S; Sherwin, R; Yang, L; Yen, K, 2012)
" Osteoblast-targeted disruption of glucocorticoid signaling significantly attenuated the suppression of osteocalcin synthesis and prevented the development of insulin resistance, glucose intolerance, and abnormal weight gain in corticosterone-treated mice."3.78Osteoblasts mediate the adverse effects of glucocorticoids on fuel metabolism. ( Blankenstein, KI; Brennan-Speranza, TC; Buttgereit, F; Cogger, VC; Cooney, GJ; Dunstan, CR; Gasparini, SJ; Gundberg, C; Heinevetter, U; Henneicke, H; Seibel, MJ; Svistounov, D; Zhang, Y; Zhou, H, 2012)
" Insulin resistance and glucose intolerance were determined along with tissue glycogen content."3.78Carbenoxolone treatment ameliorated metabolic syndrome in WNIN/Ob obese rats, but induced severe fat loss and glucose intolerance in lean rats. ( Acharya, V; Ayyalasomayajula, V; Koppala, SR; Nemani, H; Ponday, LR; Pothana, S; Prasad Sakamuri, SS; Prathipati, VK; Putcha, UK; Sukapaka, M; Veetill, GN, 2012)
"Footshock-stress promotes glucose intolerance associated to corticosterone serum level and epididymal white adipose tissue IL-6 concentration increase."3.77Fish oil consumption prevents glucose intolerance and hypercorticosteronemy in footshock-stressed rats. ( Cunha, CA; Eguchi, R; Oller do Nascimento, CM; Oyama, LM; Pisani, LP; Ribeiro, EB; Scarmagnani, FR; Souza, GI; Spadari-Bratfisch, RC, 2011)
" In mice, genetic loss of cryptochrome 1 and/or 2 results in glucose intolerance and constitutively high levels of circulating corticosterone, suggesting reduced suppression of the hypothalamic-pituitary-adrenal axis coupled with increased glucocorticoid transactivation in the liver."3.77Cryptochromes mediate rhythmic repression of the glucocorticoid receptor. ( Barish, GD; Downes, M; Evans, RM; Jonker, JW; Lamia, KA; Papp, SJ; Uhlenhaut, NH; Yu, RT, 2011)
"Body weight and food intake in pregnancy, lactation, and after weaning, plasma leptin, insulin, corticosterone and blood glucose concentrations on days 7, 13 and 18 of pregnancy, days 1, 10, 21 and 80 postpartum, glucose and insulin tolerance on pregnancy days 7 and 18 were measured in C57Bl/6J mice of a/a (normal metabolism) and A(y)/a genotypes."3.76Pregnancy and lactation have anti-obesity and anti-diabetic effects in A(y)/a mice. ( Bazhan, NM; Makarova, EN; Shevchenko, AY; Yakovleva, TV, 2010)
" We found that high doses of CORT (100 microg/ml) result in rapid and dramatic increases in weight gain, increased adiposity, elevated plasma leptin, insulin and triglyceride levels, hyperphagia, and decreased home-cage locomotion."3.76Endocrine and physiological changes in response to chronic corticosterone: a potential model of the metabolic syndrome in mouse. ( Bhagat, SM; Bowles, NP; Karatsoreos, IN; McEwen, BS; Pfaff, DW; Weil, ZM, 2010)
" In aged male rats (24 months of age), prenatal stress induced hyperglycaemia and glucose intolerance and decreased basal leptin levels."3.72Prenatal stress induces intrauterine growth restriction and programmes glucose intolerance and feeding behaviour disturbances in the aged rat. ( Darnaudery, M; Del-Favero, F; Leonhardt, M; Lesage, J; Louvart, H; Maccari, S; Vieau, D, 2004)
"A group of rats was challenged by 4-h sleep deprivation in the early rest period, leading to prolonged (16 h) wakefulness."1.43Sleep restriction acutely impairs glucose tolerance in rats. ( Challet, E; Foppen, E; Jha, PK; Kalsbeek, A, 2016)
"Schizophrenia is associated with increased risk for multiple metabolic abnormalities, including altered glucose homeostasis, type-2 diabetes, obesity, and cardiovascular disease."1.39Priming of metabolic dysfunctions by prenatal immune activation in mice: relevance to schizophrenia. ( Giovanoli, S; Langhans, W; Meyer, U; Pacheco-López, G, 2013)
"Metabolic syndrome is a condition that typically includes central obesity, insulin resistance, glucose intolerance, dyslipidemia, and hypertension."1.38Anatomic, hematologic, and biochemical features of C57BL/6NCrl mice maintained on chronic oral corticosterone. ( Cassano, AE; Karatsoreos, IN; Penraat, KA; Rasmussen, S; White, JR; Wilson, CD, 2012)

Research

Studies (38)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's2 (5.26)18.2507
2000's6 (15.79)29.6817
2010's24 (63.16)24.3611
2020's6 (15.79)2.80

Authors

AuthorsStudies
de Souza, CF1
Stopa, LRS1
Martins, AB1
Wunderlich, ALM1
Lopes, GM1
Zaia, DAM1
Zaia, CTBV1
de Andrade, FG1
Leite, CM1
Uchoa, ET1
Yamazaki, H1
Uehara, M1
Yoshikawa, N1
Kuribara-Souta, A1
Yamamoto, M1
Hirakawa, Y1
Kabe, Y1
Suematsu, M1
Tanaka, H1
Masís-Vargas, A1
Hicks, D1
Kalsbeek, A2
Mendoza, J1
Appiakannan, HS1
Rasimowicz, ML1
Harrison, CB1
Weber, ET1
Sahagun, E1
Bachman, BB1
Kinzig, KP1
Bell, RMB1
Villalobos, E1
Nixon, M1
Miguelez-Crespo, A1
Murphy, L1
Fawkes, A1
Coutts, A1
Sharp, MGF1
Koerner, MV1
Allan, E1
Meijer, OC1
Houtman, R1
Odermatt, A1
Beck, KR1
Denham, SG1
Lee, P1
Homer, NZM1
Walker, BR1
Morgan, RA1
Pereira, GM1
Santos, M1
Pereira, SS1
Borrecho, G1
Tortosa, F1
Brito, J1
Freitas, D1
de Carvalho, AO1
Águas, A1
Oliveira, MJ1
Oliveira, P1
Faria, JA1
de Araújo, TM1
Razolli, DS1
Ignácio-Souza, LM1
Souza, DN1
Bordin, S1
Anhê, GF1
Snow, SJ2
McGee, MA1
Henriquez, A1
Richards, JE2
Schladweiler, MC2
Ledbetter, AD2
Kodavanti, UP2
Teich, T1
Pivovarov, JA1
Porras, DP1
Dunford, EC1
Riddell, MC1
Wang, Y1
Zhao, W1
Shi, J1
Wang, J1
Hao, J1
Pang, X1
Huang, X1
Chen, X1
Li, Y1
Jin, R1
Ge, Q1
Nikolic, I1
Vujicic, M1
Saksida, T1
Berki, T1
Stosic-Grujicic, S1
Stojanovic, I1
Miller, DB1
Ghio, AJ1
Olatunji, LA2
Omolekulo, TE1
Usman, TO2
Kim, I1
Seok, YM1
Kim, IK1
Lecoutre, S1
Deracinois, B1
Laborie, C1
Eberlé, D1
Guinez, C1
Panchenko, PE1
Lesage, J2
Vieau, D2
Junien, C1
Gabory, A1
Breton, C1
Jha, PK1
Foppen, E1
Challet, E1
Pereira, VH1
Marques, F1
Lages, V1
Pereira, FG1
Patchev, A1
Almeida, OF1
Almeida-Palha, J1
Sousa, N1
Cerqueira, JJ1
Mendez, N1
Halabi, D1
Spichiger, C1
Salazar, ER1
Vergara, K1
Alonso-Vasquez, P1
Carmona, P1
Sarmiento, JM1
Richter, HG1
Seron-Ferre, M1
Torres-Farfan, C1
Makarova, EN1
Yakovleva, TV1
Shevchenko, AY1
Bazhan, NM1
Karatsoreos, IN2
Bhagat, SM1
Bowles, NP1
Weil, ZM1
Pfaff, DW1
McEwen, BS1
Eguchi, R1
Scarmagnani, FR1
Cunha, CA1
Souza, GI1
Pisani, LP1
Ribeiro, EB1
Oller do Nascimento, CM1
Spadari-Bratfisch, RC1
Oyama, LM1
Lamia, KA1
Papp, SJ1
Yu, RT1
Barish, GD1
Uhlenhaut, NH1
Jonker, JW1
Downes, M1
Evans, RM1
Pacheco-López, G1
Giovanoli, S1
Langhans, W1
Meyer, U1
Yang, L1
Isoda, F1
Yen, K1
Kleopoulos, SP1
Janssen, W1
Fan, X1
Mastaitis, J1
Dunn-Meynell, A1
Levin, B1
McCrimmon, R1
Sherwin, R1
Musatov, S1
Mobbs, CV1
Cordoba-Chacon, J1
Gahete, MD1
Pozo-Salas, AI1
Moreno-Herrera, A1
Castaño, JP1
Kineman, RD1
Luque, RM1
Brennan-Speranza, TC1
Henneicke, H1
Gasparini, SJ1
Blankenstein, KI1
Heinevetter, U1
Cogger, VC1
Svistounov, D1
Zhang, Y1
Cooney, GJ1
Buttgereit, F1
Dunstan, CR1
Gundberg, C1
Zhou, H1
Seibel, MJ1
Müller-Fielitz, H1
Raasch, W1
Cassano, AE1
White, JR1
Penraat, KA1
Wilson, CD1
Rasmussen, S1
Prasad Sakamuri, SS1
Sukapaka, M1
Prathipati, VK1
Nemani, H1
Putcha, UK1
Pothana, S1
Koppala, SR1
Ponday, LR1
Acharya, V1
Veetill, GN1
Ayyalasomayajula, V1
Del-Favero, F1
Leonhardt, M1
Louvart, H1
Maccari, S1
Darnaudery, M1
O'Regan, D1
Kenyon, CJ2
Seckl, JR2
Holmes, MC1
Gault, VA2
McClean, PL2
Cassidy, RS2
Irwin, N2
Flatt, PR2
Holst, JJ1
Savontaus, E1
Fagerholm, V1
Rahkonen, O1
Scheinin, M1
Boullu-Ciocca, S1
Achard, V1
Tassistro, V1
Dutour, A1
Grino, M1
Trocki, O1
Baer, DJ1
Castonguay, TW1
Nyirenda, MJ1
Lindsay, RS1
Burchell, A1

Other Studies

38 other studies available for corticosterone and Glucose Intolerance

ArticleYear
Lactation overnutrition-induced obesity impairs effects of exogenous corticosterone on energy homeostasis and hypothalamic-pituitary-adrenal axis in male rats.
    Life sciences, 2022, Sep-01, Volume: 304

    Topics: Animals; Corticosterone; Corticotropin-Releasing Hormone; Female; Glucocorticoids; Glucose Intoleran

2022
The crucial role of muscle glucocorticoid signaling in accelerating obesity and glucose intolerance via hyperinsulinemia.
    JCI insight, 2023, 04-24, Volume: 8, Issue:8

    Topics: Animals; Corticosterone; Diabetes Mellitus; Glucocorticoids; Glucose Intolerance; Hyperinsulinism; M

2023
Blue light at night acutely impairs glucose tolerance and increases sugar intake in the diurnal rodent Arvicanthis ansorgei in a sex-dependent manner.
    Physiological reports, 2019, Volume: 7, Issue:20

    Topics: Animals; Biological Clocks; Blood Glucose; Circadian Rhythm; Corticosterone; Dietary Sugars; Eating;

2019
Differential effects of high-fat diet on glucose tolerance, food intake, and glucocorticoid regulation in male C57BL/6J and BALB/cJ mice.
    Physiology & behavior, 2020, 03-01, Volume: 215

    Topics: Adipose Tissue; Animals; Blood Glucose; Corticosterone; Diet, High-Fat; Eating; Glucocorticoids; Glu

2020
Sex-specific effects of ketogenic diet after pre-exposure to a high-fat, high-sugar diet in rats.
    Nutrition, metabolism, and cardiovascular diseases : NMCD, 2021, 03-10, Volume: 31, Issue:3

    Topics: 3-Hydroxybutyric Acid; Adiposity; Animals; Biomarkers; Blood Glucose; Corticosterone; Diet, High-Fat

2021
Carbonyl reductase 1 amplifies glucocorticoid action in adipose tissue and impairs glucose tolerance in lean mice.
    Molecular metabolism, 2021, Volume: 48

    Topics: Adipose Tissue; Alcohol Oxidoreductases; Animals; Corticosterone; Diet, High-Fat; Disease Models, An

2021
High-intensity infrasound effects on glucose metabolism in rats.
    Scientific reports, 2021, 08-26, Volume: 11, Issue:1

    Topics: Animals; Blood Glucose; Corticosterone; Glucose; Glucose Intolerance; Glucose Transporter Type 4; Im

2021
Metabolic Impact of Light Phase-Restricted Fructose Consumption Is Linked to Changes in Hypothalamic AMPK Phosphorylation and Melatonin Production in Rats.
    Nutrients, 2017, Mar-27, Volume: 9, Issue:4

    Topics: AMP-Activated Protein Kinases; Animals; Circadian Rhythm; Corticosterone; Dose-Response Relationship

2017
Respiratory Effects and Systemic Stress Response Following Acute Acrolein Inhalation in Rats.
    Toxicological sciences : an official journal of the Society of Toxicology, 2017, 08-01, Volume: 158, Issue:2

    Topics: Acrolein; Animals; Cholesterol; Corticosterone; Diabetes Mellitus, Experimental; Epinephrine; Fatty

2017
Curcumin limits weight gain, adipose tissue growth, and glucose intolerance following the cessation of exercise and caloric restriction in rats.
    Journal of applied physiology (Bethesda, Md. : 1985), 2017, 12-01, Volume: 123, Issue:6

    Topics: 11-beta-Hydroxysteroid Dehydrogenase Type 1; Adipose Tissue; Animals; Caloric Restriction; Corticost

2017
Intestinal microbiota contributes to altered glucose metabolism in simulated microgravity mouse model.
    FASEB journal : official publication of the Federation of American Societies for Experimental Biology, 2019, Volume: 33, Issue:9

    Topics: Acute-Phase Proteins; Akkermansia; Animals; Bifidobacterium; Carrier Proteins; Corticosterone; Dysbi

2019
The role of endogenous glucocorticoids in glucose metabolism and immune status of MIF-deficient mice.
    European journal of pharmacology, 2013, Aug-15, Volume: 714, Issue:1-3

    Topics: Animals; Corticosterone; Gene Knockout Techniques; Glucocorticoids; Glucose; Glucose Intolerance; Im

2013
Acute Ozone-Induced Pulmonary and Systemic Metabolic Effects Are Diminished in Adrenalectomized Rats.
    Toxicological sciences : an official journal of the Society of Toxicology, 2016, Volume: 150, Issue:2

    Topics: Acute Lung Injury; Adrenal Glands; Adrenalectomy; Animals; Corticosterone; Epinephrine; Glucose Into

2016
Improvement of oral contraceptive-induced glucose dysregulation and dyslipidemia by valproic acid is independent of circulating corticosterone.
    Archives of physiology and biochemistry, 2016, Volume: 122, Issue:3

    Topics: Animals; Anticonvulsants; Contraceptives, Oral, Combined; Corticosterone; Dyslipidemias; Female; Glu

2016
Activation of cardiac renin-angiotensin system and plasminogen activator inhibitor-1 gene expressions in oral contraceptive-induced cardiometabolic disorder.
    Archives of physiology and biochemistry, 2017, Volume: 123, Issue:1

    Topics: Aldosterone; Animals; Ataxia Telangiectasia Mutated Proteins; Cardiovascular Diseases; Contraceptive

2017
Depot- and sex-specific effects of maternal obesity in offspring's adipose tissue.
    The Journal of endocrinology, 2016, Volume: 230, Issue:1

    Topics: Adipose Tissue; Animals; Body Weight; Corticosterone; Female; Glucose Intolerance; Hyperinsulinism;

2016
Sleep restriction acutely impairs glucose tolerance in rats.
    Physiological reports, 2016, Volume: 4, Issue:12

    Topics: Animals; Blood Glucose; Corticosterone; Glucose Intolerance; Insulin; Male; Rats; Rats, Wistar; Slee

2016
Glucose intolerance after chronic stress is related with downregulated PPAR-γ in adipose tissue.
    Cardiovascular diabetology, 2016, 08-19, Volume: 15, Issue:1

    Topics: Animals; Biomarkers; Blood Glucose; Chronic Disease; Corticosterone; Disease Models, Animal; Down-Re

2016
Gestational Chronodisruption Impairs Circadian Physiology in Rat Male Offspring, Increasing the Risk of Chronic Disease.
    Endocrinology, 2016, Volume: 157, Issue:12

    Topics: Aldosterone; Animals; Blood Pressure; Body Temperature; Chronic Disease; Circadian Rhythm; CLOCK Pro

2016
Pregnancy and lactation have anti-obesity and anti-diabetic effects in A(y)/a mice.
    Acta physiologica (Oxford, England), 2010, Volume: 198, Issue:2

    Topics: Age Factors; Animals; Blood Glucose; Body Weight; Breast Feeding; Corticosterone; Diabetes Mellitus;

2010
Endocrine and physiological changes in response to chronic corticosterone: a potential model of the metabolic syndrome in mouse.
    Endocrinology, 2010, Volume: 151, Issue:5

    Topics: Adipose Tissue, White; Adiposity; Adrenal Glands; Animals; Atrophy; Chemical Phenomena; Corticostero

2010
Fish oil consumption prevents glucose intolerance and hypercorticosteronemy in footshock-stressed rats.
    Lipids in health and disease, 2011, May-11, Volume: 10

    Topics: Adiponectin; Animals; Area Under Curve; Blood Glucose; Body Composition; Corticosterone; Cytokines;

2011
Cryptochromes mediate rhythmic repression of the glucocorticoid receptor.
    Nature, 2011, Dec-14, Volume: 480, Issue:7378

    Topics: Animals; Circadian Rhythm; Corticosterone; Cryptochromes; Dexamethasone; Female; Gene Expression Reg

2011
Priming of metabolic dysfunctions by prenatal immune activation in mice: relevance to schizophrenia.
    Schizophrenia bulletin, 2013, Volume: 39, Issue:2

    Topics: Acute-Phase Reaction; Animals; Body Composition; Corticosterone; Cytokines; Disease Models, Animal;

2013
Hypothalamic Fkbp51 is induced by fasting, and elevated hypothalamic expression promotes obese phenotypes.
    American journal of physiology. Endocrinology and metabolism, 2012, Apr-15, Volume: 302, Issue:8

    Topics: Animals; Arcuate Nucleus of Hypothalamus; Corticosterone; Energy Intake; Fasting; Gene Expression Pr

2012
Peripubertal-onset but not adult-onset obesity increases IGF-I and drives development of lean mass, which may lessen the metabolic impairment in adult obesity.
    American journal of physiology. Endocrinology and metabolism, 2012, Nov-01, Volume: 303, Issue:9

    Topics: Adrenocorticotropic Hormone; Aging; Animals; Body Composition; Corticosterone; Diet, High-Fat; Disea

2012
Osteoblasts mediate the adverse effects of glucocorticoids on fuel metabolism.
    The Journal of clinical investigation, 2012, Volume: 122, Issue:11

    Topics: Animals; Anti-Inflammatory Agents; Corticosterone; Energy Metabolism; Glucocorticoids; Glucose Intol

2012
Angiotensin II impairs glucose utilization in obese Zucker rats by increasing HPA activity via an adrenal-dependent mechanism.
    Hormone and metabolic research = Hormon- und Stoffwechselforschung = Hormones et metabolisme, 2013, Volume: 45, Issue:2

    Topics: Adrenalectomy; Adrenocorticotropic Hormone; Angiotensin II; Animals; Blood Glucose; Corticosterone;

2013
Anatomic, hematologic, and biochemical features of C57BL/6NCrl mice maintained on chronic oral corticosterone.
    Comparative medicine, 2012, Volume: 62, Issue:5

    Topics: Adrenal Glands; Analysis of Variance; Animals; Case-Control Studies; Corticosterone; Dose-Response R

2012
Carbenoxolone treatment ameliorated metabolic syndrome in WNIN/Ob obese rats, but induced severe fat loss and glucose intolerance in lean rats.
    PloS one, 2012, Volume: 7, Issue:12

    Topics: 11-beta-Hydroxysteroid Dehydrogenase Type 1; Adipocytes; Adipose Tissue; Adrenal Glands; Animals; Bo

2012
Prenatal stress induces intrauterine growth restriction and programmes glucose intolerance and feeding behaviour disturbances in the aged rat.
    The Journal of endocrinology, 2004, Volume: 181, Issue:2

    Topics: Adrenal Glands; Aging; Animals; Birth Weight; Blood Glucose; Corticosterone; Feeding Behavior; Femal

2004
Glucocorticoid exposure in late gestation in the rat permanently programs gender-specific differences in adult cardiovascular and metabolic physiology.
    American journal of physiology. Endocrinology and metabolism, 2004, Volume: 287, Issue:5

    Topics: Adrenocorticotropic Hormone; Animals; Birth Weight; Blood Pressure; Corticosterone; Dexamethasone; F

2004
Chemical gastric inhibitory polypeptide receptor antagonism protects against obesity, insulin resistance, glucose intolerance and associated disturbances in mice fed high-fat and cafeteria diets.
    Diabetologia, 2007, Volume: 50, Issue:8

    Topics: Adiponectin; Animals; Blood Glucose; Body Weight; Corticosterone; Dietary Fats; Eating; Gastric Inhi

2007
GIP receptor antagonism reverses obesity, insulin resistance, and associated metabolic disturbances induced in mice by prolonged consumption of high-fat diet.
    American journal of physiology. Endocrinology and metabolism, 2007, Volume: 293, Issue:6

    Topics: Adipokines; Adipose Tissue; Animals; Anti-Obesity Agents; Blood Glucose; Body Weight; Corticosterone

2007
Reduced blood glucose levels, increased insulin levels and improved glucose tolerance in alpha2A-adrenoceptor knockout mice.
    European journal of pharmacology, 2008, Jan-14, Volume: 578, Issue:2-3

    Topics: Adrenergic beta-Antagonists; Animals; Atropine; Blood Glucose; Corticosterone; Fasting; Female; Gluc

2008
Postnatal programming of glucocorticoid metabolism in rats modulates high-fat diet-induced regulation of visceral adipose tissue glucocorticoid exposure and sensitivity and adiponectin and proinflammatory adipokines gene expression in adulthood.
    Diabetes, 2008, Volume: 57, Issue:3

    Topics: 11-beta-Hydroxysteroid Dehydrogenase Type 1; Adipokines; Adiponectin; Adipose Tissue; Aging; Animals

2008
Comparison of effects of adrenalectomy and RU-486 in rats given a choice of maintenance diet and fat supplement.
    The American journal of physiology, 1995, Volume: 269, Issue:3 Pt 2

    Topics: Adrenalectomy; Animals; Body Composition; Body Weight; Corticosterone; Diet; Dietary Fats; Eating; E

1995
Glucocorticoid exposure in late gestation permanently programs rat hepatic phosphoenolpyruvate carboxykinase and glucocorticoid receptor expression and causes glucose intolerance in adult offspring.
    The Journal of clinical investigation, 1998, May-15, Volume: 101, Issue:10

    Topics: 11-beta-Hydroxysteroid Dehydrogenases; Animals; Birth Weight; Blood Glucose; Body Weight; Corticoste

1998