corticosterone has been researched along with Blood Poisoning in 53 studies
| Excerpt | Relevance | Reference |
|---|---|---|
| "The aim of the present study was to assess the effect of seven days daidzein pretreatment in cecal ligation and puncture (CLP) model of sepsis." | 7.81 | Daidzein pretreatment improves survival in mouse model of sepsis. ( Kandasamy, K; Mishra, SK; Narasimha Reddy, ChE; Panigrahi, M; Parida, S; Singh, TU; Singh, V; Thangamalai, R, 2015) |
| "We investigated the effects of sepsis, through the lipopolysaccharide (LPS)-induced inflammatory response, on plasma corticosterone and prolactin (PRL) levels during acute immobilization stress in normal and thyroidectomized rats." | 7.72 | Hypothyroidism attenuates stress-induced prolactin and corticosterone release in septic rats. ( Albuquerque-Araújo, WI; Antunes-Rodrigues, J; Ramalho, MJ; Reis, LC; Rodriguez, T, 2003) |
| "Pretreatment with mifepristone reduces the demand for adenosine triphosphate production from phosphocreatine breakdown and downregulates Na(+)-K+ adenosine triphosphatase activity during sepsis." | 7.69 | Glucocorticoid receptor antagonism by mifepristone alters phosphocreatine breakdown during sepsis. ( Jacobs, DO; Mitsuo, T; Prechek, D; Rounds, J; Wilmore, DW, 1996) |
| "The plasma concentration of various catabolic hormones, including glucagon and catecholamines, is elevated in sepsis." | 7.67 | Importance of hyperglucagonemia in eliciting the sepsis-induced increase in glucose production. ( Bagby, GJ; Blakesley, HL; Lang, CH; Spitzer, JJ, 1989) |
| "Sepsis was induced by cecal ligation-puncture (CLP) surgery." | 5.72 | Corticosterone and Adrenocorticotrophic Hormone Secretion Is Recovered after Immune Challenge or Acute Restraint Stress in Sepsis Survivor Animals. ( Alves Rocha, MJ; Catalão, CHR; da Costa, LHA; Dos Santos-Junior, NN, 2022) |
| " Compared to healthy subjects, patients with sepsis had elevated levels of 11-desoxycorticosterone and 11-desoxycortisol, consistent with activation of both glucocorticoid and mineralocorticoid pathways." | 5.51 | Corticotropin-stimulated steroid profiles to predict shock development and mortality in sepsis: From the HYPRESS study. ( Annane, D; Bogatsch, H; Briegel, J; Frank, S; Hinske, LC; Keh, D; Lange, D; Lindner, JM; Möhnle, P; Vetter, AC; Vogeser, M, 2022) |
| "Sepsis is hallmarked by high plasma cortisol/corticosterone (CORT), low adrenocorticotropic hormone (ACTH), and high pro-opiomelanocortin (POMC)." | 4.12 | Impact of Hydrocortisone and of CRH Infusion on the Hypothalamus-Pituitary-Adrenocortical Axis of Septic Male Mice. ( De Bruyn, L; Derde, S; Langouche, L; Pauwels, L; Téblick, A; Van den Berghe, G; Van Oudenhove, T; Vander Perre, S, 2022) |
| " The purpose of this study was to elucidate whether administration of a β2 adrenergic agonist, formoterol, was able to prevent the acute effects of sepsis induced by liposaccharide (LPS) injection on rat gastrocnemius muscle and to evaluate the possible roles of corticosterone, IGF-I, miR-23a, and miR-29b." | 3.88 | Formoterol treatment prevents the effects of endotoxin on muscle TNF/NF-kB, Akt/mTOR, and proteolytic pathways in a rat model. Role of IGF-I and miRNA 29b. ( Gómez-SanMiguel, AB; López-Calderón, A; Martín, AI; Priego, T, 2018) |
| "ET-26 HCl showed virtually no suppression of corticosterone synthesis, lower concentrations of pro-inflammatory cytokines, higher survival rate, and less organ injury in rats suffering from sepsis compared with the etomidate group." | 3.85 | Pharmacologic studies on ET-26 hydrochloride in a rat model of lipopolysaccharide-induced sepsis. ( Chen, J; Jiang, J; Liu, J; Wang, B; Yang, J; Zhang, W; Zhu, Z, 2017) |
| "Both hyperinflammation during sepsis and etomidate can suppress adrenal function." | 3.83 | The pituitary adenylate cyclase-activating polypeptide (PACAP) protects adrenal function in septic rats administered etomidate. ( Liu, N; Liu, S; Lv, S; Xiong, JY; Zhang, Y; Zhu, J, 2016) |
| "The aim of the present study was to assess the effect of seven days daidzein pretreatment in cecal ligation and puncture (CLP) model of sepsis." | 3.81 | Daidzein pretreatment improves survival in mouse model of sepsis. ( Kandasamy, K; Mishra, SK; Narasimha Reddy, ChE; Panigrahi, M; Parida, S; Singh, TU; Singh, V; Thangamalai, R, 2015) |
| "An overproduction of corticosterone during severe sepsis results in increased apoptosis of immune cells, which may result in relative immunosuppression and an impaired ability to fight infections." | 3.80 | Selective histone deacetylase-6 inhibition attenuates stress responses and prevents immune organ atrophy in a lethal septic model. ( Alam, HB; Bronson, RT; Li, Y; Liu, B; Velmahos, GC; Zhao, T, 2014) |
| " In the present study, we compared endogenous corticosterone production of wild-type (WT) and TLR2-deficient (TLR2) mice and analyzed survival after hydrocortisone therapy during sepsis induced by cecal ligation and puncture (CLP)." | 3.79 | Hydrocortisone reduces the beneficial effects of toll-like receptor 2 deficiency on survival in a mouse model of polymicrobial sepsis. ( Bergt, S; Butschkau, A; Heidrich, M; Nöldge-Schomburg, GE; Roesner, JP; Vollmar, B; Wagner, NM, 2013) |
| " The aim of this study was to evaluate changes in plasma adrenocorticotrophic hormone (ACTH), corticosterone and cortisol levels in a rabbit model in which sepsis was induced by the intravenous administration of LPS." | 3.75 | Changes in plasma hormone levels following lipopolysaccharide injection in rabbits. ( Gómez-Quintero, A; Marca, MC; Rodríguez-Yoldi, MJ; Viñuales, C, 2009) |
| "We investigated the effects of sepsis, through the lipopolysaccharide (LPS)-induced inflammatory response, on plasma corticosterone and prolactin (PRL) levels during acute immobilization stress in normal and thyroidectomized rats." | 3.72 | Hypothyroidism attenuates stress-induced prolactin and corticosterone release in septic rats. ( Albuquerque-Araújo, WI; Antunes-Rodrigues, J; Ramalho, MJ; Reis, LC; Rodriguez, T, 2003) |
| "The effects of dantrolene on serum TNFalpha and corticosterone levels and on muscle calcium, calpain gene expression, and protein breakdown were studied in rats with abdominal sepsis induced by cecal ligation and puncture." | 3.71 | Dantrolene reduces serum TNFalpha and corticosterone levels and muscle calcium, calpain gene expression, and protein breakdown in septic rats. ( Fischer, DR; Fischer, JE; Gang, G; Hasselgren, PO; James, JH; Molloy, M; Paul, RJ; Pritts, TA; Sun, X; Williams, AB, 2001) |
| ", a late stage of sepsis) or sham operation to measure plasma levels of corticosterone and corticotropin as well as adrenal contents of corticosterone." | 3.71 | Adrenal insufficiency during the late stage of polymicrobial sepsis. ( Chaudry, IH; Jackman, D; Koo, DJ; Wang, P, 2001) |
| "Treatment of rats with dantrolene prevented the sepsis-induced increase in mRNA levels for ubiquitin, the ubiquitin ligase E3alpha, and the 20S proteasome subunit C3." | 3.71 | Dantrolene downregulates the gene expression and activity of the ubiquitin-proteasome proteolytic pathway in septic skeletal muscle. ( Gang, GI; Hasselgren, PO; Sun, X; Wray, CJ, 2002) |
| "Pretreatment with mifepristone reduces the demand for adenosine triphosphate production from phosphocreatine breakdown and downregulates Na(+)-K+ adenosine triphosphatase activity during sepsis." | 3.69 | Glucocorticoid receptor antagonism by mifepristone alters phosphocreatine breakdown during sepsis. ( Jacobs, DO; Mitsuo, T; Prechek, D; Rounds, J; Wilmore, DW, 1996) |
| "Burn wound sepsis in rats results in sustained corticosterone elevations and the prolonged presence of translocated bacteria in the mesenteric lymph nodes (MLNs)." | 3.68 | Pathophysiologic glucocorticoid levels and survival of translocating bacteria. ( Barber, AE; Calvano, SE; Fahey, TJ; Hawes, AJ; Jones, WG; Kapur, S; Minei, JP; Shires, GT, 1991) |
| "The plasma concentration of various catabolic hormones, including glucagon and catecholamines, is elevated in sepsis." | 3.67 | Importance of hyperglucagonemia in eliciting the sepsis-induced increase in glucose production. ( Bagby, GJ; Blakesley, HL; Lang, CH; Spitzer, JJ, 1989) |
| "Sepsis was induced by cecal ligation-puncture (CLP) surgery." | 1.72 | Corticosterone and Adrenocorticotrophic Hormone Secretion Is Recovered after Immune Challenge or Acute Restraint Stress in Sepsis Survivor Animals. ( Alves Rocha, MJ; Catalão, CHR; da Costa, LHA; Dos Santos-Junior, NN, 2022) |
| "Sepsis is hallmarked by hypercortisolemia, a stress response essential for survival." | 1.48 | The Hepatic Glucocorticoid Receptor Is Crucial for Cortisol Homeostasis and Sepsis Survival in Humans and Male Mice. ( Derde, S; Dufour, T; Güiza, F; Jenniskens, M; Langouche, L; Pauwels, L; Téblick, A; Van den Berghe, G; Vander Perre, S; Weckx, R, 2018) |
| "Sepsis was induced using cecal ligation and puncture (CLP) in wild-type (WT) mice, IL-18 knockout (KO) mice, and IL-18 KO mice pretreated with recombinant IL-18." | 1.46 | Interleukin-18 Reduces Blood Glucose and Modulates Plasma Corticosterone in a Septic Mouse Model. ( Inoue, T; Ishikawa, M; Kotani, J; Usami, M; Usami, Y; Yamashita, H, 2017) |
| "Sepsis was induced by cecal ligation and puncture (CLP) in wild type (WT) and OPN gene knockout (OPN(-/-) ) mice." | 1.42 | Osteopontin is associated with inflammation and mortality in a mouse model of polymicrobial sepsis. ( Fortis, S; Haitsma, JJ; Khadaroo, RG; Zhang, H, 2015) |
| "The treatment with imipramine reversed all the parameters described above." | 1.36 | Depressive-like parameters in sepsis survivor rats. ( Barichello, T; Cassol, OJ; Comim, CM; Constantino, LC; Constantino, LS; Dal-Pizzol, F; Kapczinski, F; Petronilho, F; Quevedo, J; Stertz, L, 2010) |
| "Sepsis is characterized by a systemic inflammatory response of the immune system against an infection, presenting with hypothalamic-pituitary-adrenal (HPA) axis dysfunction, behavior alterations, and high mortality." | 1.36 | Low dose dexamethasone reverses depressive-like parameters and memory impairment in rats submitted to sepsis. ( Cassol, OJ; Comim, CM; Constantino, LS; Dal-Pizzol, F; Petronilho, F; Quevedo, J; Streck, EL, 2010) |
| "Sepsis is a leading cause of death that is characterized by uncontrolled inflammatory response." | 1.35 | Scavenger Receptor BI Protects against Septic Death through Its Role in Modulating Inflammatory Response. ( Bernard, P; Daugherty, A; Feng, H; Guo, L; Huang, B; Li, M; Li, XA; Song, Z; Wang, D; Wu, Q, 2009) |
| "Sepsis is a leading cause of death in the Western world and can be associated with failure of the hypothalamic-pituitary-adrenal axis." | 1.34 | Toll-like receptor 9 expression in murine and human adrenal glands and possible implications during inflammation. ( Baumgarten, G; Berkels, R; Boehm, O; Bornstein, SR; Kanczkowski, W; Knuefermann, P; Koch, A; Lightman, SL; Schott, M; Tran, N; Zacharowski, K; Zacharowski, PA, 2007) |
| "Septicemia is one of the major health concerns worldwide, and rapid activation of adrenal steroid release is a key event in the organism's first line of defense during this form of severe illness." | 1.32 | Impaired adrenal stress response in Toll-like receptor 2-deficient mice. ( Barthel, A; Bornstein, SR; McCann, SM; Papewalis, C; Rettori, V; Scherbaum, WA; Schulze-Osthoff, K; Schumann, RR; Tarnow, J; Tran, N; Zacharowski, K; Zacharowski, P, 2004) |
| "Sepsis was induced with an intraperitoneal (i." | 1.30 | Effect of NG-nitro-L-arginine methyl ester on testicular blood flow and serum steroid hormones during sepsis. ( Bosmann, HB; Ferguson, JL; Hales, DB; Law, WR; Lee, LY; Sam, AD; Sharma, AC, 1998) |
| "Sepsis was induced with cecal slurry (200 mg/kg/5 mL 5% dextrose in water (D5W); i." | 1.29 | Steroid hormone alterations following induction of chronic intraperitoneal sepsis in male rats. ( Bosmann, HB; Ferguson, JL; Hales, DB; Hales, KH; Motew, SJ; Sharma, AC, 1996) |
| "Corticosterone plasma levels were significantly elevated in both trauma-hemorrhage groups compared with those in sham-operated mice." | 1.29 | Mechanism of immunosuppression in males following trauma-hemorrhage. Critical role of testosterone. ( Ayala, A; Chaudry, IH; DeMaso, CM; Wichmann, MW; Zellweger, R, 1996) |
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 3 (5.66) | 18.7374 |
| 1990's | 8 (15.09) | 18.2507 |
| 2000's | 15 (28.30) | 29.6817 |
| 2010's | 20 (37.74) | 24.3611 |
| 2020's | 7 (13.21) | 2.80 |
| Authors | Studies |
|---|---|
| Eryılmaz-Eren, E | 1 |
| Dinç, G | 1 |
| Kontaş, O | 1 |
| Alp, E | 1 |
| Doğanay, M | 1 |
| Téblick, A | 2 |
| De Bruyn, L | 1 |
| Van Oudenhove, T | 1 |
| Vander Perre, S | 2 |
| Pauwels, L | 2 |
| Derde, S | 2 |
| Langouche, L | 2 |
| Van den Berghe, G | 2 |
| Dos Santos-Junior, NN | 1 |
| da Costa, LHA | 2 |
| Catalão, CHR | 2 |
| Alves Rocha, MJ | 1 |
| Briegel, J | 1 |
| Möhnle, P | 1 |
| Keh, D | 1 |
| Lindner, JM | 1 |
| Vetter, AC | 1 |
| Bogatsch, H | 1 |
| Lange, D | 1 |
| Frank, S | 1 |
| Hinske, LC | 1 |
| Annane, D | 1 |
| Vogeser, M | 1 |
| Kono, H | 1 |
| Furuya, S | 1 |
| Sun, C | 1 |
| Akazawa, Y | 1 |
| Nakata, Y | 1 |
| Fukushima, H | 1 |
| Wakana, H | 1 |
| Fujii, H | 1 |
| Ichikawa, D | 1 |
| Spencer-Segal, JL | 1 |
| Singer, BH | 1 |
| Laborc, K | 1 |
| Somayaji, K | 1 |
| Watson, SJ | 1 |
| Standiford, TJ | 1 |
| Akil, H | 1 |
| Santos-Junior, NN | 1 |
| Rocha, MJA | 1 |
| Wang, B | 1 |
| Jiang, J | 1 |
| Yang, J | 1 |
| Chen, J | 1 |
| Zhu, Z | 1 |
| Liu, J | 1 |
| Zhang, W | 1 |
| Jenniskens, M | 1 |
| Weckx, R | 1 |
| Dufour, T | 1 |
| Güiza, F | 1 |
| Martín, AI | 1 |
| Gómez-SanMiguel, AB | 1 |
| Priego, T | 1 |
| López-Calderón, A | 1 |
| Diedrich, S | 2 |
| van der Linde, J | 2 |
| Nielson, M | 2 |
| Menges, P | 2 |
| Kühn, JP | 1 |
| Käding, A | 1 |
| Ngyuen Trung, D | 1 |
| Heidecke, CD | 2 |
| Partecke, LI | 2 |
| Kessler, W | 2 |
| Packiriswamy, N | 1 |
| Lee, T | 1 |
| Raghavendra, PB | 1 |
| Durairaj, H | 1 |
| Wang, H | 1 |
| Parameswaran, N | 1 |
| Guo, L | 3 |
| Ai, J | 2 |
| Zheng, Z | 2 |
| Howatt, DA | 2 |
| Daugherty, A | 3 |
| Huang, B | 2 |
| Li, XA | 3 |
| Lee, JK | 1 |
| Jung, JS | 1 |
| Park, SH | 1 |
| Sim, YB | 1 |
| Suh, HW | 1 |
| Bergt, S | 1 |
| Wagner, NM | 1 |
| Heidrich, M | 1 |
| Butschkau, A | 1 |
| Nöldge-Schomburg, GE | 1 |
| Vollmar, B | 1 |
| Roesner, JP | 1 |
| Mittelstadt, PR | 1 |
| Thacker, S | 1 |
| Ashwell, JD | 1 |
| Remaley, AT | 1 |
| Zhao, T | 1 |
| Li, Y | 1 |
| Bronson, RT | 1 |
| Liu, B | 1 |
| Velmahos, GC | 1 |
| Alam, HB | 1 |
| Fortis, S | 1 |
| Khadaroo, RG | 1 |
| Haitsma, JJ | 1 |
| Zhang, H | 1 |
| Parida, S | 1 |
| Singh, TU | 1 |
| Thangamalai, R | 1 |
| Narasimha Reddy, ChE | 1 |
| Panigrahi, M | 1 |
| Kandasamy, K | 1 |
| Singh, V | 1 |
| Mishra, SK | 1 |
| Liu, N | 1 |
| Zhang, Y | 1 |
| Xiong, JY | 1 |
| Liu, S | 2 |
| Zhu, J | 1 |
| Lv, S | 1 |
| Yamashita, H | 1 |
| Ishikawa, M | 1 |
| Inoue, T | 1 |
| Usami, M | 1 |
| Usami, Y | 1 |
| Kotani, J | 1 |
| Song, Z | 1 |
| Li, M | 1 |
| Wu, Q | 1 |
| Wang, D | 1 |
| Feng, H | 1 |
| Bernard, P | 1 |
| Comim, CM | 2 |
| Cassol, OJ | 2 |
| Constantino, LC | 1 |
| Petronilho, F | 2 |
| Constantino, LS | 2 |
| Stertz, L | 1 |
| Kapczinski, F | 1 |
| Barichello, T | 1 |
| Quevedo, J | 2 |
| Dal-Pizzol, F | 2 |
| Spencer, SJ | 1 |
| Field, E | 1 |
| Pittman, QJ | 1 |
| Streck, EL | 1 |
| Zhu, X | 1 |
| Liu, Y | 1 |
| Wang, C | 1 |
| Wang, S | 1 |
| Tang, X | 1 |
| Ni, X | 1 |
| Ebker, T | 1 |
| Traeger, T | 1 |
| Cziupka, K | 1 |
| Puls, R | 1 |
| Busemann, A | 1 |
| Maier, S | 1 |
| Goodwin, JE | 1 |
| Feng, Y | 1 |
| Velazquez, H | 1 |
| Sessa, WC | 1 |
| Sun, X | 3 |
| Mammen, JM | 1 |
| Tian, X | 1 |
| ANDO, S | 1 |
| GUZE, LB | 1 |
| GOLD, EM | 1 |
| Rodriguez, T | 1 |
| Albuquerque-Araújo, WI | 1 |
| Reis, LC | 1 |
| Antunes-Rodrigues, J | 1 |
| Ramalho, MJ | 1 |
| Ocasio, FM | 1 |
| Jiang, Y | 1 |
| House, SD | 1 |
| Chang, SL | 1 |
| Bornstein, SR | 2 |
| Zacharowski, P | 1 |
| Schumann, RR | 1 |
| Barthel, A | 1 |
| Tran, N | 2 |
| Papewalis, C | 1 |
| Rettori, V | 1 |
| McCann, SM | 1 |
| Schulze-Osthoff, K | 1 |
| Scherbaum, WA | 1 |
| Tarnow, J | 1 |
| Zacharowski, K | 2 |
| Frost, RA | 2 |
| Nystrom, GJ | 1 |
| Jefferson, LS | 1 |
| Lang, CH | 4 |
| Carlson, DE | 2 |
| Chiu, WC | 2 |
| Fiedler, SM | 1 |
| Hoffman, GE | 1 |
| Koch, A | 1 |
| Berkels, R | 1 |
| Boehm, O | 1 |
| Zacharowski, PA | 1 |
| Baumgarten, G | 1 |
| Knuefermann, P | 1 |
| Schott, M | 1 |
| Kanczkowski, W | 1 |
| Lightman, SL | 1 |
| Marca, MC | 1 |
| Gómez-Quintero, A | 1 |
| Viñuales, C | 1 |
| Rodríguez-Yoldi, MJ | 1 |
| Molina, PE | 1 |
| Abumrad, NN | 1 |
| Mekaouche, M | 1 |
| Siaud, P | 1 |
| Givalois, L | 1 |
| Barbanel, G | 1 |
| Malaval, F | 1 |
| Maurel, D | 1 |
| Assenmacher, I | 1 |
| Ixart, G | 1 |
| Sharma, AC | 2 |
| Bosmann, HB | 2 |
| Motew, SJ | 1 |
| Hales, KH | 1 |
| Hales, DB | 2 |
| Ferguson, JL | 2 |
| Mitsuo, T | 1 |
| Rounds, J | 1 |
| Prechek, D | 1 |
| Wilmore, DW | 1 |
| Jacobs, DO | 1 |
| Wichmann, MW | 1 |
| Zellweger, R | 1 |
| DeMaso, CM | 1 |
| Ayala, A | 1 |
| Chaudry, IH | 2 |
| Lanza-Jacoby, S | 1 |
| Phetteplace, H | 1 |
| Sedkova, N | 1 |
| Knee, G | 1 |
| Sam, AD | 1 |
| Lee, LY | 1 |
| Law, WR | 1 |
| Fischer, DR | 1 |
| Williams, AB | 1 |
| Gang, G | 1 |
| Pritts, TA | 1 |
| James, JH | 1 |
| Molloy, M | 1 |
| Fischer, JE | 1 |
| Paul, RJ | 1 |
| Hasselgren, PO | 2 |
| Beno, DW | 1 |
| Uhing, MR | 1 |
| Goto, M | 1 |
| Chen, Y | 1 |
| Jiyamapa-Serna, VA | 1 |
| Kimura, RE | 1 |
| Koo, DJ | 1 |
| Jackman, D | 1 |
| Wang, P | 1 |
| Silvis, C | 1 |
| Nystrom, G | 1 |
| Wray, CJ | 1 |
| Gang, GI | 1 |
| Jones, WG | 1 |
| Barber, AE | 1 |
| Kapur, S | 1 |
| Hawes, AJ | 1 |
| Fahey, TJ | 1 |
| Minei, JP | 1 |
| Shires, GT | 2 |
| Calvano, SE | 1 |
| Bagby, GJ | 1 |
| Blakesley, HL | 1 |
| Spitzer, JJ | 1 |
| Goel, KM | 1 |
| Shanks, RA | 1 |
| Trial | Phase | Enrollment | Study Type | Start Date | Status | ||
|---|---|---|---|---|---|---|---|
| Placebo-controlled, Randomised, Double-blind Study to Investigate the Efficacy and Safety of Low Dose Hydrocortisone to Prevent the Development of Septic Shock in Patients With Severe Sepsis[NCT00670254] | Phase 3 | 380 participants (Actual) | Interventional | 2009-01-31 | Completed | ||
| [information is prepared from clinicaltrials.gov, extracted Sep-2024] | |||||||
1 trial available for corticosterone and Blood Poisoning
| Article | Year |
|---|---|
Corticotropin-stimulated steroid profiles to predict shock development and mortality in sepsis: From the HYPRESS study.
Topics: Adrenocorticotropic Hormone; Adult; Chromatography, Liquid; Corticosterone; Cortodoxone; Desoxycorti | 2022 |
52 other studies available for corticosterone and Blood Poisoning
| Article | Year |
|---|---|
The investigation of adrenal involvement in carbapenem resistant Acinetobacter baumannii sepsis: experimental mouse model
Topics: Acinetobacter baumannii; Acinetobacter Infections; Adrenal Insufficiency; Animals; Anti-Bacterial Ag | 2021 |
Impact of Hydrocortisone and of CRH Infusion on the Hypothalamus-Pituitary-Adrenocortical Axis of Septic Male Mice.
Topics: Adrenocorticotropic Hormone; Animals; Arginine Vasopressin; Corticosterone; Corticotropin-Releasing | 2022 |
Corticosterone and Adrenocorticotrophic Hormone Secretion Is Recovered after Immune Challenge or Acute Restraint Stress in Sepsis Survivor Animals.
Topics: Adrenocorticotropic Hormone; Animals; Corticosterone; Hypothalamo-Hypophyseal System; Lipopolysaccha | 2022 |
Pituitary Adenylate Cyclase-Activating Polypeptide Prevents Mortality Caused by Septic Peritonitis in Mice.
Topics: Acute Lung Injury; Adrenocorticotropic Hormone; Animals; Cells, Cultured; Corticosterone; Cytokines; | 2020 |
Sepsis survivor mice exhibit a behavioral endocrine syndrome with ventral hippocampal dysfunction.
Topics: Acute Disease; Animals; Anxiety; Behavior, Animal; Corticosterone; Critical Illness; Depression; Dis | 2020 |
Microglial Activation Modulates Neuroendocrine Secretion During Experimental Sepsis.
Topics: Animals; Brain; Corticosterone; Disease Models, Animal; Male; Microglia; Minocycline; Neurons; Neuro | 2021 |
Pharmacologic studies on ET-26 hydrochloride in a rat model of lipopolysaccharide-induced sepsis.
Topics: Animals; Corticosterone; Cytokines; Etomidate; Female; Hypnotics and Sedatives; Kidney; Lipopolysacc | 2017 |
The Hepatic Glucocorticoid Receptor Is Crucial for Cortisol Homeostasis and Sepsis Survival in Humans and Male Mice.
Topics: Animals; Corticosterone; Homeostasis; Humans; Hydrocortisone; Liver; Male; Mice; Receptors, Glucocor | 2018 |
Formoterol treatment prevents the effects of endotoxin on muscle TNF/NF-kB, Akt/mTOR, and proteolytic pathways in a rat model. Role of IGF-I and miRNA 29b.
Topics: Adrenergic beta-2 Receptor Agonists; Animals; Corticosterone; Formoterol Fumarate; Insulin-Like Grow | 2018 |
The MRI Sepsis Score: An Innovative Tool for the Evaluation of Septic Peritonitis in Mice Using 7-Tesla Small Animal MRI.
Topics: Animals; Corticosterone; Female; Magnetic Resonance Imaging; Mice; Mice, Inbred C57BL; Peritonitis; | 2018 |
G-protein-coupled receptor kinase-5 mediates inflammation but does not regulate cellular infiltration or bacterial load in a polymicrobial sepsis model in mice.
Topics: Animals; Apoptosis; Bacterial Load; Cecum; Cell Movement; Cells, Cultured; Corticosterone; Disease M | 2013 |
High density lipoprotein protects against polymicrobe-induced sepsis in mice.
Topics: Animals; Apolipoprotein A-I; Bacterial Infections; Cecum; Corticosterone; Interleukin-6; Ligation; L | 2013 |
Deficiency of alpha-calcitonin gene-related peptide induces inflammatory responses and lethality in sepsis.
Topics: Alanine Transaminase; Animals; Aspartate Aminotransferases; Brain; Calcitonin Gene-Related Peptide; | 2013 |
Hydrocortisone reduces the beneficial effects of toll-like receptor 2 deficiency on survival in a mouse model of polymicrobial sepsis.
Topics: Animals; Corticosterone; Disease Models, Animal; Feedback, Physiological; Hydrocortisone; Hypothalam | 2013 |
Scavenger receptor BI and high-density lipoprotein regulate thymocyte apoptosis in sepsis.
Topics: Animals; Apoptosis; Cecum; Cells, Cultured; Cholesterol, HDL; Corticosterone; Disease Models, Animal | 2014 |
Selective histone deacetylase-6 inhibition attenuates stress responses and prevents immune organ atrophy in a lethal septic model.
Topics: Adrenocorticotropic Hormone; Animals; Apoptosis; Atrophy; Bone Marrow; Corticosterone; Disease Model | 2014 |
Osteopontin is associated with inflammation and mortality in a mouse model of polymicrobial sepsis.
Topics: Animals; Corticosterone; Cytokines; Disease Models, Animal; Inflammation; Interleukin-6; Male; Mice; | 2015 |
Daidzein pretreatment improves survival in mouse model of sepsis.
Topics: Acute Lung Injury; Animals; Bacterial Load; Biomarkers; Cecum; Corticosterone; Drug Administration S | 2015 |
The pituitary adenylate cyclase-activating polypeptide (PACAP) protects adrenal function in septic rats administered etomidate.
Topics: Adrenal Glands; Adrenocorticotropic Hormone; Anesthetics, Intravenous; Animals; Apoptosis; Corticost | 2016 |
Interleukin-18 Reduces Blood Glucose and Modulates Plasma Corticosterone in a Septic Mouse Model.
Topics: Animals; Blood Glucose; Corticosterone; Disease Models, Animal; Glucose-6-Phosphatase; Insulin; Inte | 2017 |
Scavenger Receptor BI Protects against Septic Death through Its Role in Modulating Inflammatory Response.
Topics: Animals; Cells, Cultured; Corticosterone; Cytokines; Gene Expression; Gram-Negative Bacteria; Lipopo | 2009 |
Depressive-like parameters in sepsis survivor rats.
Topics: Adrenal Glands; Adrenocorticotropic Hormone; Analysis of Variance; Animals; Antidepressive Agents, T | 2010 |
Neonatal programming by neuroimmune challenge: effects on responses and tolerance to septic doses of lipopolysaccharide in adult male and female rats.
Topics: Aging; Animals; Animals, Newborn; Body Temperature; Corticosterone; Dose-Response Relationship, Drug | 2010 |
Low dose dexamethasone reverses depressive-like parameters and memory impairment in rats submitted to sepsis.
Topics: Adrenal Glands; Adrenocorticotropic Hormone; Animals; Anti-Inflammatory Agents; Avoidance Learning; | 2010 |
Endotoxin tolerance of adrenal gland: attenuation of corticosterone production in response to lipopolysaccharide and adrenocorticotropic hormone.
Topics: Adrenal Glands; Adrenal Insufficiency; Adrenocorticotropic Hormone; Animals; Blotting, Western; Cell | 2011 |
The role of the vagus nerve: modulation of the inflammatory reaction in murine polymicrobial sepsis.
Topics: Animals; Corticosterone; Female; Inflammation; Kupffer Cells; Lipopolysaccharides; Magnetic Resonanc | 2012 |
Endothelial glucocorticoid receptor is required for protection against sepsis.
Topics: Animals; Apoptosis; Corticosterone; Endothelium; Gene Deletion; Hemodynamics; Human Umbilical Vein E | 2013 |
Sepsis induces the transcription of the glucocorticoid receptor in skeletal muscle cells.
Topics: Animals; Cell Line; Corticosterone; Dexamethasone; Glucocorticoids; Hormone Antagonists; Hydrocortis | 2003 |
ACTH RELEASE IN VIVO AND IN VITRO: EXTRAPITUITARY MEDIATION DURING ESCH. COLI BACTEREMIA.
Topics: Adrenocorticotropic Hormone; Arginine Vasopressin; Bacteremia; Corticosterone; Escherichia coli Infe | 1964 |
Hypothyroidism attenuates stress-induced prolactin and corticosterone release in septic rats.
Topics: Animals; Corticosterone; Hypothyroidism; Lipopolysaccharides; Male; Prolactin; Rats; Rats, Wistar; S | 2003 |
Chronic morphine accelerates the progression of lipopolysaccharide-induced sepsis to septic shock.
Topics: Animals; Antithrombin III; Blood Pressure; Body Temperature; Cell Communication; Corticosterone; Dis | 2004 |
Impaired adrenal stress response in Toll-like receptor 2-deficient mice.
Topics: Adrenal Cortex; Adrenocorticotropic Hormone; Animals; Corticosterone; Cytokines; Endotoxemia; Humans | 2004 |
Hormone, cytokine, and nutritional regulation of sepsis-induced increases in atrogin-1 and MuRF1 in skeletal muscle.
Topics: Animals; Corticosterone; Cytokines; Diet; Glucocorticoids; Insulin; Male; Muscle Proteins; Muscle, S | 2007 |
Central neural distribution of immunoreactive Fos and CRH in relation to plasma ACTH and corticosterone during sepsis in the rat.
Topics: Adrenocorticotropic Hormone; Animals; Cecum; Corticosterone; Corticotropin-Releasing Hormone; Dience | 2007 |
Toll-like receptor 9 expression in murine and human adrenal glands and possible implications during inflammation.
Topics: Adjuvants, Immunologic; Adrenal Cortex; Adrenal Cortex Neoplasms; Adrenocorticotropic Hormone; Anima | 2007 |
The absence of circadian cues during recovery from sepsis modifies pituitary-adrenocortical function and impairs survival.
Topics: Adrenal Cortex; Adrenocorticotropic Hormone; Animals; Circadian Rhythm; Corticosterone; Disease Mode | 2008 |
Changes in plasma hormone levels following lipopolysaccharide injection in rabbits.
Topics: Adrenocorticotropic Hormone; Animals; Corticosterone; Hydrocortisone; Lipopolysaccharides; Male; Rab | 2009 |
Granulocyte colony-stimulating factor prevents ethanol-induced impairment in host defense in septic rats.
Topics: Alcoholism; Animals; Blood Bactericidal Activity; Blood Glucose; Corticosterone; Escherichia coli In | 1993 |
Different responses of plasma ACTH and corticosterone and of plasma interleukin-1 beta to single and recurrent endotoxin challenges.
Topics: Adrenocorticotropic Hormone; Animals; Bacterial Toxins; Corticosterone; Escherichia coli; Interleuki | 1996 |
Steroid hormone alterations following induction of chronic intraperitoneal sepsis in male rats.
Topics: Animals; Blood Pressure; Chronic Disease; Corticosterone; Heart Rate; Male; Peritonitis; Progesteron | 1996 |
Glucocorticoid receptor antagonism by mifepristone alters phosphocreatine breakdown during sepsis.
Topics: Adenosine Triphosphate; Animals; Corticosterone; Creatine; Creatine Kinase; Down-Regulation; Energy | 1996 |
Mechanism of immunosuppression in males following trauma-hemorrhage. Critical role of testosterone.
Topics: Androgens; Animals; Blood Pressure; Cell Division; Corticosterone; Disease Susceptibility; Immune To | 1996 |
Sequential alterations in tissue lipoprotein lipase, triglyceride secretion rates, and serum tumor necrosis factor alpha during Escherichia coli bacteremic sepsis in relation to the development of hypertriglyceridemia.
Topics: Adipose Tissue; Animals; Body Temperature Regulation; Catecholamines; Corticosterone; Escherichia co | 1998 |
Effect of NG-nitro-L-arginine methyl ester on testicular blood flow and serum steroid hormones during sepsis.
Topics: Animals; Cardiac Output; Corticosterone; Enzyme Inhibitors; Male; NG-Nitroarginine Methyl Ester; Nit | 1998 |
Dantrolene reduces serum TNFalpha and corticosterone levels and muscle calcium, calpain gene expression, and protein breakdown in septic rats.
Topics: Animals; Calcium; Calpain; Corticosterone; Dantrolene; Male; Muscle Proteins; Muscle Relaxants, Cent | 2001 |
Endotoxin-induced reduction in biliary indocyanine green excretion rate in a chronically catheterized rat model.
Topics: Animals; Bile; Catheters, Indwelling; Common Bile Duct; Corticosterone; Endotoxins; Escherichia coli | 2001 |
Adrenal insufficiency during the late stage of polymicrobial sepsis.
Topics: Adrenal Glands; Adrenal Insufficiency; Adrenocorticotropic Hormone; Animals; Bacterial Infections; C | 2001 |
Regulation of myostatin by glucocorticoids after thermal injury.
Topics: Animals; Burns; Corticosterone; Dexamethasone; Endotoxins; Escherichia coli; Gene Expression Regulat | 2001 |
Dantrolene downregulates the gene expression and activity of the ubiquitin-proteasome proteolytic pathway in septic skeletal muscle.
Topics: Animals; Calcium; Cecum; Chymotrypsin; Corticosterone; Cysteine Endopeptidases; Dantrolene; Gene Exp | 2002 |
Pathophysiologic glucocorticoid levels and survival of translocating bacteria.
Topics: Administration, Cutaneous; Animals; Burns; Cecum; Corticosterone; Cyclosporins; Liver; Lymph Nodes; | 1991 |
Importance of hyperglucagonemia in eliciting the sepsis-induced increase in glucose production.
Topics: Animals; Corticosterone; Glucagon; Glucose; Insulin; Male; Rats; Rats, Inbred Strains; Sepsis; Somat | 1989 |
Follow-up study of 100 cases of juvenile rheumatoid arthritis.
Topics: Adolescent; Amyloidosis; Arthritis, Juvenile; Child; Child, Preschool; Corticosterone; Female; Follo | 1974 |