corticosterone has been researched along with Allergic Encephalomyelitis in 49 studies
Excerpt | Relevance | Reference |
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"In an effort to understand the role of endogenous corticosterone production on the induction of experimental allergic encephalomyelitis (EAE) in rats, experiments in our study were performed using inbred rat strains that differ in basal corticosterone levels." | 7.68 | Experimental allergic encephalomyelitis and corticosterone studies in resistant and susceptible rat strains. ( Blankenhorn, EP; Dronsfield, MJ; Villas, PA, 1991) |
"In experimental allergic encephalomyelitis (EAE), another chronic stress model, the role of substance P action is not clear." | 5.32 | Hypothalamic response to experimental allergic encephalomyelitis: role of substance P. ( Depaulis, A; Fernandes, GA; Levy, S; Namer, IJ; Ruocco, HH, 2004) |
"However, disease progression is inhibited if the circulating corticosterone level is maintained at levels seen during the initial phase of disease." | 5.31 | Disease progression in chronic relapsing experimental allergic encephalomyelitis is associated with reduced inflammation-driven production of corticosterone. ( Holsboer, F; Lassmann, H; Linington, C; Pohl, T; Reul, JM; Stadelmann, C; Stefferl, A; Storch, MK; Tilders, FJ, 2001) |
"Memantine was administered orally either semiprophylactically, from day 7 postinoculation (PI), or therapeutically, 10 to 11 days PI." | 5.31 | Modulation of blood-brain barrier dysfunction and neurological deficits during acute experimental allergic encephalomyelitis by the N-methyl-D-aspartate receptor antagonist memantine. ( Bolton, C; Paul, C, 2002) |
"The recovery of animals from experimental autoimmune encephalomyelitis (EAE) is clearly dependent on an increase in endogenous glucocorticoid levels." | 2.41 | The cytokine-HPA axis circuit contributes to prevent or moderate autoimmune processes. ( Besedovsky, HO; del Rey, A, 2000) |
"Experimental allergic encephalomyelitis (EAE) is considered to be a useful animal model of human multiple sclerosis (MS)." | 1.56 | Expression of hypothalamic feeding-related peptide genes and neuroendocrine responses in an experimental allergic encephalomyelitis rat model. ( Kusuhara, K; Maruyama, T; Matsuura, T; Motojima, Y; Nishimura, H; Nishimura, K; Onaka, T; Saito, R; Sanada, K; Sonoda, S; Tanaka, K; Ueno, H; Ueta, Y; Yamamoto, Y; Yoshimura, M, 2020) |
"Treatment with alprazolam reversed the adverse effects of stress." | 1.36 | Inhibitory effects of alprazolam on the development of acute experimental autoimmune encephalomyelitis in stressed rats. ( Almeida-Dias, A; Freire-Garabal, M; Novío, S; Núñez-Iglesias, MJ, 2010) |
"Acute experimental autoimmune encephalomyelitis (EAE) is an inflammatory disease of the central nervous system, mediated by T lymphocytes." | 1.33 | Neurointermediate pituitary lobectomy decreases the incidence and severity of experimental autoimmune encephalomyelitis in Lewis rats. ( Berczi, I; Chavira-Ramírez, R; Kovacs, K; Quintanar-Stephano, A, 2005) |
"In experimental allergic encephalomyelitis (EAE), another chronic stress model, the role of substance P action is not clear." | 1.32 | Hypothalamic response to experimental allergic encephalomyelitis: role of substance P. ( Depaulis, A; Fernandes, GA; Levy, S; Namer, IJ; Ruocco, HH, 2004) |
"Oral vancomycin treatment after disease induction significantly decreased clinical symptoms of AIA." | 1.31 | Oral antibiotics as a novel therapy for arthritis: evidence for a beneficial effect of intestinal Escherichia coli. ( Akkermans, LM; Cobelens, PM; Fleer, A; Harmsen, W; Heijnen, CJ; Kavelaars, A; Nieuwenhuis, EE; Verhoef, J; Visser, MR, 2000) |
"experimental allergic encephalomyelitis (EAE)." | 1.31 | Priming with interleukin-1beta suppresses experimental allergic encephalomyelitis in the Lewis rat. ( Binnekade, R; Huitinga, I; Schmidt, ED; Tilders, FJ; van der Cammen, MJ, 2000) |
"However, disease progression is inhibited if the circulating corticosterone level is maintained at levels seen during the initial phase of disease." | 1.31 | Disease progression in chronic relapsing experimental allergic encephalomyelitis is associated with reduced inflammation-driven production of corticosterone. ( Holsboer, F; Lassmann, H; Linington, C; Pohl, T; Reul, JM; Stadelmann, C; Stefferl, A; Storch, MK; Tilders, FJ, 2001) |
"Memantine was administered orally either semiprophylactically, from day 7 postinoculation (PI), or therapeutically, 10 to 11 days PI." | 1.31 | Modulation of blood-brain barrier dysfunction and neurological deficits during acute experimental allergic encephalomyelitis by the N-methyl-D-aspartate receptor antagonist memantine. ( Bolton, C; Paul, C, 2002) |
"Susceptibility to experimental allergic encephalomyelitis (EAE) may be influenced by variations in the production of endogenous glucocorticoids." | 1.30 | Susceptibility and resistance to experimental allergic encephalomyelitis: relationship with hypothalamic-pituitary-adrenocortical axis responsiveness in the rat. ( Holsboer, F; Linington, C; Reul, JM; Stefferl, A, 1999) |
"Dehydroepiandrosterone (DHEA) is a native neurosteroid with immunomodulating activity." | 1.30 | Dehydroepiandrosterone selectively inhibits production of tumor necrosis factor alpha and interleukin-6 [correction of interlukin-6] in astrocytes. ( Brenner, T; Danenberg, HD; Galilly, R; Kipper-Galperin, M, 1999) |
"High corticosterone levels were detected during stages of acute paralysis, whereas a decrease to normal levels was noted during each recovery phase." | 1.29 | Immunoregulation and drug treatment in chronic relapsing experimental allergic encephalomyelitis in the Lewis rat. ( Borel, JF; Chabannes, D; Kovarik, J, 1995) |
"Spontaneous recovery from acute experimental allergic encephalomyelitis (EAE) by the Lewis rat is probably mediated by endogenous corticosteroids." | 1.29 | Lipocortin 1 (annexin 1) immunoreactivity in the cervical spinal cord of Lewis rats with acute experimental allergic encephalomyelitis. ( Bolton, C; Elderfield, AJ; Flower, RJ, 1993) |
"Induction of experimental allergic encephalomyelitis (EAE) in female Lewis rats led to the well-known clinical symptoms and histological signs." | 1.29 | Suppression of acute experimental allergic encephalomyelitis by the synthetic sex hormone 17-alpha-ethinylestradiol: an immunological study in the Lewis rat. ( Kampinga, J; Loof, JG; Minderhoud, JM; Nieuwenhuis, P; Teelken, AW; Trooster, WJ, 1993) |
"When experimental autoimmune encephalomyelitis (EAE) is induced by adoptive transfer of myelin basic protein (MBP)-specific lymphocytes the splenic noradrenergic and adrenocortical responses mirror in most respects those that occur following sensitization with spinal cord and Freund's adjuvant (CFA), despite the absence of the primary immune challenge." | 1.28 | Splenic noradrenergic and adrenocortical responses during the preclinical and clinical stages of adoptively transferred experimental autoimmune encephalomyelitis (EAE). ( Cuzner, ML; Leonard, JP; MacKenzie, FJ; Patel, HA, 1990) |
"Experimental allergic encephalomyelitis (EAE) can be induced in some strains of rat but not others, by the injection of guinea-pig myelin basic protein in Freund's complete adjuvant." | 1.28 | The role of the neuroendocrine system in determining genetic susceptibility to experimental allergic encephalomyelitis in the rat. ( Antoni, F; MacPhee, I; Mason, D, 1990) |
"Lewis rats with experimental allergic encephalomyelitis (EAE), induced either by the subcutaneous injection of guinea pig myelin basic protein (MBP) or by the adoptive transfer of MBP-primed spleen cells, suffer from a single episode of paralysis from which they recover spontaneously." | 1.28 | Spontaneous recovery of rats from experimental allergic encephalomyelitis is dependent on regulation of the immune system by endogenous adrenal corticosteroids. ( Antoni, FA; MacPhee, IA; Mason, DW, 1989) |
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 4 (8.16) | 18.7374 |
1990's | 19 (38.78) | 18.2507 |
2000's | 14 (28.57) | 29.6817 |
2010's | 10 (20.41) | 24.3611 |
2020's | 2 (4.08) | 2.80 |
Authors | Studies |
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Tanaka, K | 1 |
Saito, R | 1 |
Sanada, K | 1 |
Nishimura, H | 1 |
Nishimura, K | 1 |
Sonoda, S | 1 |
Ueno, H | 1 |
Motojima, Y | 1 |
Matsuura, T | 1 |
Yoshimura, M | 1 |
Maruyama, T | 1 |
Onaka, T | 1 |
Yamamoto, Y | 1 |
Kusuhara, K | 1 |
Ueta, Y | 1 |
Moriguchi, K | 1 |
Miyamoto, K | 1 |
Fukumoto, Y | 1 |
Kusunoki, S | 1 |
Dos Santos, N | 1 |
Novaes, LS | 1 |
Dragunas, G | 1 |
Rodrigues, JR | 1 |
Brandão, W | 1 |
Camarini, R | 1 |
Peron, JPS | 1 |
Munhoz, CD | 1 |
Acharjee, S | 1 |
Nayani, N | 1 |
Tsutsui, M | 1 |
Hill, MN | 1 |
Ousman, SS | 1 |
Pittman, QJ | 1 |
Benkhoucha, M | 1 |
Molnarfi, N | 1 |
Dunand-Sauthier, I | 1 |
Merkler, D | 1 |
Schneiter, G | 1 |
Bruscoli, S | 1 |
Riccardi, C | 1 |
Tabata, Y | 1 |
Funakoshi, H | 1 |
Nakamura, T | 1 |
Reith, W | 1 |
Santiago-Raber, ML | 1 |
Lalive, PH | 1 |
Majidi-Zolbanin, J | 1 |
Doosti, MH | 1 |
Kosari-Nasab, M | 1 |
Salari, AA | 1 |
Núñez-Iglesias, MJ | 1 |
Novío, S | 1 |
Almeida-Dias, A | 1 |
Freire-Garabal, M | 1 |
Pérez-Nievas, BG | 1 |
García-Bueno, B | 1 |
Madrigal, JL | 1 |
Leza, JC | 1 |
Wang, ZW | 1 |
Wang, P | 1 |
Lin, FH | 1 |
Li, XL | 1 |
Li, XF | 1 |
O'Byrne, KT | 1 |
Hou, ST | 1 |
Zheng, RY | 1 |
Buenafe, AC | 1 |
Quintanar-Stephano, A | 2 |
Organista-Esparza, A | 1 |
Chavira-Ramírez, R | 2 |
Kovacs, K | 2 |
Berczi, I | 2 |
Harpaz, I | 1 |
Abutbul, S | 1 |
Nemirovsky, A | 1 |
Gal, R | 1 |
Cohen, H | 1 |
Monsonego, A | 1 |
Ruocco, HH | 1 |
Fernandes, GA | 1 |
Namer, IJ | 1 |
Depaulis, A | 1 |
Levy, S | 1 |
Macció, DR | 2 |
Calfa, G | 2 |
Volosín, M | 1 |
Roth, GA | 2 |
Esquifino, AI | 1 |
Cano, P | 1 |
Jiménez, V | 1 |
Cutrera, RA | 1 |
Cardinali, DP | 1 |
Dimitrijević, M | 1 |
Rauski, A | 1 |
Radojević, K | 1 |
Kosec, D | 1 |
Stanojević, S | 1 |
Pilipović, I | 1 |
Leposavić, G | 1 |
Levine, S | 1 |
Sowinski, R | 1 |
Steinetz, B | 1 |
Kovarik, J | 1 |
Chabannes, D | 1 |
Borel, JF | 1 |
Elderfield, AJ | 1 |
Bolton, C | 4 |
Flower, RJ | 1 |
Trooster, WJ | 1 |
Teelken, AW | 1 |
Kampinga, J | 1 |
Loof, JG | 1 |
Nieuwenhuis, P | 1 |
Minderhoud, JM | 1 |
Smith, T | 1 |
Schmied, M | 1 |
Hewson, AK | 1 |
Lassmann, H | 2 |
Cuzner, ML | 4 |
Ruuls, SR | 1 |
de Labie, MC | 1 |
Weber, KS | 1 |
Botman, CA | 1 |
Groenestein, RJ | 1 |
Dijkstra, CD | 3 |
Olsson, T | 1 |
van der Meide, PH | 1 |
Paul, C | 2 |
O'Neill, JK | 1 |
Allen, SJ | 1 |
Baker, D | 1 |
Weidenfeld, J | 1 |
Karussis, D | 1 |
Abramsky, O | 1 |
Lehmann, D | 1 |
Arbell, I | 1 |
Ovadia, H | 1 |
del Rey, A | 2 |
Klusman, I | 1 |
Besedovsky, HO | 2 |
Pekarski, O | 1 |
Björk, J | 1 |
Hedlund, G | 1 |
Andersson, G | 1 |
Stefferl, A | 2 |
Linington, C | 2 |
Holsboer, F | 2 |
Reul, JM | 2 |
Kipper-Galperin, M | 1 |
Galilly, R | 1 |
Danenberg, HD | 1 |
Brenner, T | 1 |
Kavelaars, A | 3 |
Heijnen, CJ | 3 |
Tennekes, R | 1 |
Bruggink, JE | 1 |
Koolhaas, JM | 1 |
Dowdell, KC | 1 |
Gienapp, IE | 1 |
Stuckman, S | 1 |
Wardrop, RM | 1 |
Whitacre, CC | 1 |
Bakker, JM | 1 |
Kamphuis, PJ | 1 |
Cobelens, PM | 2 |
van Vugt, HH | 1 |
van Bel, F | 1 |
Nieuwenhuis, EE | 1 |
Visser, MR | 1 |
Fleer, A | 1 |
Harmsen, W | 1 |
Verhoef, J | 1 |
Akkermans, LM | 1 |
Huitinga, I | 2 |
Schmidt, ED | 1 |
van der Cammen, MJ | 1 |
Binnekade, R | 1 |
Tilders, FJ | 2 |
Storch, MK | 1 |
Stadelmann, C | 1 |
Pohl, T | 1 |
Marchetti, B | 1 |
Morale, MC | 1 |
Brouwer, J | 1 |
Tirolo, C | 1 |
Testa, N | 1 |
Caniglia, S | 1 |
Barden, N | 1 |
Amor, S | 1 |
Smith, PA | 1 |
Stephan, M | 1 |
Straub, RH | 1 |
Breivik, T | 1 |
Pabst, R | 1 |
von Hörsten, S | 1 |
Leonard, JP | 3 |
MacKenzie, FJ | 3 |
Patel, HA | 2 |
Villas, PA | 1 |
Dronsfield, MJ | 1 |
Blankenhorn, EP | 1 |
van Rooijen, N | 1 |
de Groot, CJ | 1 |
Uitdehaag, BM | 1 |
Mason, D | 1 |
MacPhee, I | 1 |
Antoni, F | 1 |
MacPhee, IA | 1 |
Antoni, FA | 1 |
Mason, DW | 1 |
Khoruzhaya, TA | 1 |
1 review available for corticosterone and Allergic Encephalomyelitis
Article | Year |
---|---|
The cytokine-HPA axis circuit contributes to prevent or moderate autoimmune processes.
Topics: Adrenocorticotropic Hormone; Animals; Autoimmune Diseases; Corticosterone; Cytokines; Encephalomyeli | 2000 |
48 other studies available for corticosterone and Allergic Encephalomyelitis
Article | Year |
---|---|
Expression of hypothalamic feeding-related peptide genes and neuroendocrine responses in an experimental allergic encephalomyelitis rat model.
Topics: Animals; Arginine Vasopressin; Body Weight; Corticosterone; Eating; Encephalomyelitis, Autoimmune, E | 2020 |
Change in light-dark cycle affects experimental autoimmune encephalomyelitis.
Topics: Animals; Circadian Rhythm; Corticosterone; Encephalomyelitis, Autoimmune, Experimental; Female; Mice | 2021 |
High dose of dexamethasone protects against EAE-induced motor deficits but impairs learning/memory in C57BL/6 mice.
Topics: Animals; Anti-Inflammatory Agents; Corticosterone; Dexamethasone; Disease Models, Animal; Encephalom | 2019 |
Altered cognitive-emotional behavior in early experimental autoimmune encephalitis--cytokine and hormonal correlates.
Topics: Animals; Behavior, Animal; Cognition Disorders; Corticosterone; Cytokines; Early Diagnosis; Emotions | 2013 |
Hepatocyte growth factor limits autoimmune neuroinflammation via glucocorticoid-induced leucine zipper expression in dendritic cells.
Topics: Adoptive Transfer; Animals; Autoimmunity; Cell Proliferation; Cells, Cultured; Central Nervous Syste | 2014 |
Prenatal maternal immune activation increases anxiety- and depressive-like behaviors in offspring with experimental autoimmune encephalomyelitis.
Topics: Animals; Anxiety; Anxiety Disorders; Behavior, Animal; Corticosterone; Depression; Encephalomyelitis | 2015 |
Inhibitory effects of alprazolam on the development of acute experimental autoimmune encephalomyelitis in stressed rats.
Topics: Alprazolam; Animals; Anti-Anxiety Agents; Body Weight; Corticosterone; Encephalomyelitis, Autoimmune | 2010 |
Chronic immobilisation stress ameliorates clinical score and neuroinflammation in a MOG-induced EAE in Dark Agouti rats: mechanisms implicated.
Topics: Analysis of Variance; Animals; Blotting, Western; Corticosterone; Dinoprostone; Encephalomyelitis, A | 2010 |
Early-life exposure to lipopolysaccharide reduces the severity of experimental autoimmune encephalomyelitis in adulthood and correlated with increased urine corticosterone and apoptotic CD4+ T cells.
Topics: Analysis of Variance; Animals; Animals, Newborn; Annexin A5; Antigens, CD; Apoptosis; CD4-Positive T | 2011 |
Diurnal rhythms are altered in a mouse model of multiple sclerosis.
Topics: Animals; Chronobiology Disorders; Circadian Rhythm; CLOCK Proteins; Corticosterone; Disease Models, | 2012 |
Effects of neurointermediate pituitary lobectomy and desmopressin on acute experimental autoimmune encephalomyelitis in Lewis rats.
Topics: Adrenocorticotropic Hormone; Animals; Arginine Vasopressin; Corticosterone; Deamino Arginine Vasopre | 2012 |
Chronic exposure to stress predisposes to higher autoimmune susceptibility in C57BL/6 mice: glucocorticoids as a double-edged sword.
Topics: Animals; Anxiety; Autoimmunity; Corticosterone; Disease Susceptibility; Encephalomyelitis, Autoimmun | 2013 |
Hypothalamic response to experimental allergic encephalomyelitis: role of substance P.
Topics: Adrenocorticotropic Hormone; Animals; Arginine Vasopressin; Body Weight; Chronic Disease; Corticoste | 2004 |
Serum testosterone and corticosterone levels in acute experimental autoimmune encephalomyelitis (EAE) in male Wistar rats.
Topics: Acute Disease; Animals; Corticosterone; Encephalomyelitis, Autoimmune, Experimental; Male; Rats; Rat | 2004 |
Neurointermediate pituitary lobectomy decreases the incidence and severity of experimental autoimmune encephalomyelitis in Lewis rats.
Topics: Adrenocorticotropic Hormone; Animals; Brain; Corticosterone; Encephalomyelitis, Autoimmune, Experime | 2005 |
Experimental allergic encephalomyelitis in male Lewis rats subjected to calorie restriction.
Topics: Adrenocorticotropic Hormone; Animals; Caloric Restriction; Cell Division; Concanavalin A; Corticoste | 2004 |
Oral testosterone in male rats and the development of experimental autoimmune encephalomyelitis.
Topics: Administration, Oral; Animals; Body Weight; Cell Proliferation; Cholesterol; Corticosterone; Disease | 2005 |
Beta-adrenoceptor blockade ameliorates the clinical course of experimental allergic encephalomyelitis and diminishes its aggravation in adrenalectomized rats.
Topics: Adrenalectomy; Adrenergic beta-Antagonists; Animals; Brain; Corticosterone; Encephalomyelitis, Autoi | 2007 |
Effects of experimental allergic encephalomyelitis on thymus and adrenal: relation to remission and relapse.
Topics: Adrenal Glands; Adrenalectomy; Animals; Corticosterone; Encephalomyelitis, Autoimmune, Experimental; | 1980 |
Immunoregulation and drug treatment in chronic relapsing experimental allergic encephalomyelitis in the Lewis rat.
Topics: Animals; Autoimmune Diseases; Concanavalin A; Corticosterone; Cyclosporine; Dexamethasone; Disease M | 1995 |
Lipocortin 1 (annexin 1) immunoreactivity in the cervical spinal cord of Lewis rats with acute experimental allergic encephalomyelitis.
Topics: Acute Disease; Animals; Annexin A1; Corticosterone; Encephalomyelitis, Autoimmune, Experimental; Imm | 1993 |
Suppression of acute experimental allergic encephalomyelitis by the synthetic sex hormone 17-alpha-ethinylestradiol: an immunological study in the Lewis rat.
Topics: Acute Disease; Animals; Corticosterone; Encephalomyelitis, Autoimmune, Experimental; Ethinyl Estradi | 1993 |
Apoptosis of T cells and macrophages in the central nervous system of intact and adrenalectomized Lewis rats during experimental allergic encephalomyelitis.
Topics: Adrenalectomy; Animals; Apoptosis; Cell Count; Central Nervous System; Corticosterone; Disease Model | 1996 |
The length of treatment determines whether IFN-beta prevents or aggravates experimental autoimmune encephalomyelitis in Lewis rats.
Topics: Animals; Autoantibodies; Body Weight; Corticosterone; Dose-Response Relationship, Drug; Drug Adminis | 1996 |
MK-801 limits neurovascular dysfunction during experimental allergic encephalomyelitis.
Topics: Animals; Blood-Brain Barrier; Capillary Permeability; Corticosterone; Dizocilpine Maleate; Encephalo | 1997 |
Regulation of chronic relapsing experimental allergic encephalomyelitis by endogenous and exogenous glucocorticoids.
Topics: Animals; Chronic Disease; Corticosterone; Dexamethasone; Disease Models, Animal; Encephalomyelitis, | 1997 |
Linomide activates the adrenocortical axis in the rat: inhibition of experimental autoimmune encephalomyelitis by linomide is not related to the increase of corticosterone.
Topics: Adjuvants, Immunologic; Adrenal Cortex; Adrenalectomy; Adrenocorticotropic Hormone; Animals; Cortico | 1997 |
Cytokines mediate protective stimulation of glucocorticoid output during autoimmunity: involvement of IL-1.
Topics: Analysis of Variance; Animals; Corticosterone; Encephalomyelitis, Autoimmune, Experimental; Freund's | 1998 |
The inhibitory effect in experimental autoimmune encephalomyelitis by the immunomodulatory drug Linomide (PNU-212616) is not mediated via release of endogenous glucocorticoids.
Topics: Adjuvants, Immunologic; Adrenalectomy; Animals; Corticosterone; Encephalomyelitis, Autoimmune, Exper | 1998 |
Susceptibility and resistance to experimental allergic encephalomyelitis: relationship with hypothalamic-pituitary-adrenocortical axis responsiveness in the rat.
Topics: Adrenal Cortex; Adrenocorticotropic Hormone; Animals; Antigens, Surface; Corticosterone; Disease Sus | 1999 |
Dehydroepiandrosterone selectively inhibits production of tumor necrosis factor alpha and interleukin-6 [correction of interlukin-6] in astrocytes.
Topics: Adjuvants, Immunologic; Animals; Anti-Inflammatory Agents; Astrocytes; Cells, Cultured; Corticostero | 1999 |
Individual behavioral characteristics of wild-type rats predict susceptibility to experimental autoimmune encephalomyelitis.
Topics: Aggression; Animals; Animals, Wild; Behavior, Animal; Corticosterone; Encephalomyelitis, Autoimmune, | 1999 |
Neuroendocrine modulation of chronic relapsing experimental autoimmune encephalomyelitis: a critical role for the hypothalamic-pituitary-adrenal axis.
Topics: Adrenergic beta-Antagonists; Aminoglutethimide; Animals; Anti-Inflammatory Agents; Corticosterone; E | 1999 |
Neonatal dexamethasone treatment increases susceptibility to experimental autoimmune disease in adult rats.
Topics: Aging; Animals; Animals, Newborn; Body Weight; Cells, Cultured; Corticosterone; Cytokines; Dexametha | 2000 |
Oral antibiotics as a novel therapy for arthritis: evidence for a beneficial effect of intestinal Escherichia coli.
Topics: Administration, Oral; Animals; Anti-Bacterial Agents; Arthritis, Experimental; Arthritis, Rheumatoid | 2000 |
Priming with interleukin-1beta suppresses experimental allergic encephalomyelitis in the Lewis rat.
Topics: Adrenocorticotropic Hormone; Animals; Cell Division; Concanavalin A; Corticosterone; Corticotropin-R | 2000 |
Disease progression in chronic relapsing experimental allergic encephalomyelitis is associated with reduced inflammation-driven production of corticosterone.
Topics: Animals; Autoantibodies; Chronic Disease; Corticosterone; Cytokines; Disease Progression; Encephalom | 2001 |
Exposure to a dysfunctional glucocorticoid receptor from early embryonic life programs the resistance to experimental autoimmune encephalomyelitis via nitric oxide-induced immunosuppression.
Topics: Animals; Corticosterone; Embryo, Mammalian; Encephalomyelitis, Autoimmune, Experimental; Female; Imm | 2002 |
Postnatal maternal deprivation aggravates experimental autoimmune encephalomyelitis in adult Lewis rats: reversal by chronic imipramine treatment.
Topics: Animals; Animals, Newborn; Antidepressive Agents, Tricyclic; Corticosterone; Encephalomyelitis, Auto | 2002 |
Modulation of blood-brain barrier dysfunction and neurological deficits during acute experimental allergic encephalomyelitis by the N-methyl-D-aspartate receptor antagonist memantine.
Topics: Animals; Blood-Brain Barrier; Body Weight; Corticosterone; Disease Progression; Encephalomyelitis, A | 2002 |
Splenic noradrenergic and adrenocortical responses during the preclinical and clinical stages of adoptively transferred experimental autoimmune encephalomyelitis (EAE).
Topics: Adrenal Cortex; Animals; Corticosterone; Encephalomyelitis, Autoimmune, Experimental; Female; Immuni | 1990 |
Hypothalamic noradrenergic pathways exert an influence on neuroendocrine and clinical status in experimental autoimmune encephalomyelitis.
Topics: Adrenal Glands; Adrenalectomy; Animals; Corticosterone; Encephalomyelitis, Autoimmune, Experimental; | 1991 |
Experimental allergic encephalomyelitis and corticosterone studies in resistant and susceptible rat strains.
Topics: Animals; Corticosterone; Crosses, Genetic; Encephalomyelitis, Autoimmune, Experimental; Female; Male | 1991 |
Suppression of experimental allergic encephalomyelitis in Lewis rats after elimination of macrophages.
Topics: Animals; Brain; CD4 Antigens; Clodronic Acid; Corticosterone; Encephalomyelitis, Autoimmune, Experim | 1990 |
The role of the neuroendocrine system in determining genetic susceptibility to experimental allergic encephalomyelitis in the rat.
Topics: Adrenalectomy; Animals; Corticosterone; Encephalomyelitis, Autoimmune, Experimental; Genetic Predisp | 1990 |
Changes in lymphocyte beta-adrenergic receptor density and noradrenaline content of the spleen are early indicators of immune reactivity in acute experimental allergic encephalomyelitis in the Lewis rat.
Topics: Animals; Corticosterone; Encephalomyelitis, Autoimmune, Experimental; Female; Lymphocytes; Norepinep | 1989 |
Spontaneous recovery of rats from experimental allergic encephalomyelitis is dependent on regulation of the immune system by endogenous adrenal corticosteroids.
Topics: Adrenal Glands; Adrenalectomy; Animals; Corticosterone; Encephalomyelitis, Autoimmune, Experimental; | 1989 |
Secretion and utilization of adrenal corticosteroids in experimental allergic encephalomyelitis.
Topics: Adrenal Glands; Animals; Brain; Chromatography, Thin Layer; Corticosterone; Cortisone; Dogs; Encepha | 1974 |