cordycepin has been researched along with Breast Neoplasms in 7 studies
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 0 (0.00) | 18.2507 |
2000's | 1 (14.29) | 29.6817 |
2010's | 5 (71.43) | 24.3611 |
2020's | 1 (14.29) | 2.80 |
Authors | Studies |
---|---|
Fu, J; Li, L; Liu, C; Qi, M; Wu, X; Yuan, Y | 1 |
Cui, M; Dong, J; Fan, S; Jiang, M; Li, Y; Lu, L; Luo, D; Xiao, H; Zhang, S; Zhu, C | 1 |
Choi, YJ; Jeong, MH; Jeong, YK; Jo, WS; Kang, BW; Lee, CM; Lee, SW; Seo, MJ; Seo, SY; Yang, KM | 1 |
Chung, EY; Han, JH; Jeong, YJ; Jung, JY; Jung, SH; Kim, BS; Kim, JS; Lee, SH; Lee, YR; Noh, EM; Youn, HJ | 1 |
Choi, S; Jeon, BH; Kim, KM; Kim, TW; Lim, MH; Song, WO | 1 |
BrĂ¼ning, A; Burger, P; Friese, K; Lee, HJ; Vogel, M | 1 |
Scorilas, A; Thomadaki, H; Tsiapalis, CM | 1 |
7 other study(ies) available for cordycepin and Breast Neoplasms
Article | Year |
---|---|
Natural cordycepin induces apoptosis and suppresses metastasis in breast cancer cells by inhibiting the Hedgehog pathway.
Topics: Antineoplastic Agents; Apoptosis; Breast Neoplasms; Cell Line, Tumor; Deoxyadenosines; Down-Regulation; Female; Hedgehog Proteins; Humans; Neoplasm Metastasis | 2020 |
Cordycepin sensitizes breast cancer cells toward irradiation through elevating ROS production involving Nrf2.
Topics: Animals; Apoptosis; Breast Neoplasms; Cell Proliferation; Deoxyadenosines; DNA Damage; Female; G2 Phase Cell Cycle Checkpoints; Heme Oxygenase-1; Histones; Humans; MCF-7 Cells; Mice, Inbred BALB C; Mice, Nude; NF-E2-Related Factor 2; Oxidative Stress; Radiation Tolerance; Radiation-Sensitizing Agents; Reactive Oxygen Species; Signal Transduction; Up-Regulation; Xenograft Model Antitumor Assays | 2019 |
Cordycepin-enriched Cordyceps militaris induces immunomodulation and tumor growth delay in mouse-derived breast cancer.
Topics: Animals; Antineoplastic Agents; Breast Neoplasms; CD4-Positive T-Lymphocytes; CD8-Positive T-Lymphocytes; Cell Proliferation; Cordyceps; Deoxyadenosines; Female; Forkhead Transcription Factors; Immunomodulation; Immunotherapy; Interferon-gamma; Interleukin-2; Interleukin-2 Receptor alpha Subunit; Interleukin-4; Mice; Mice, Inbred C3H; Survival Rate; Transforming Growth Factor beta | 2013 |
Cordycepin inhibits TPA-induced matrix metalloproteinase-9 expression by suppressing the MAPK/AP-1 pathway in MCF-7 human breast cancer cells.
Topics: Analysis of Variance; Antineoplastic Agents; Breast Neoplasms; Cell Line, Tumor; Cell Survival; Deoxyadenosines; Female; Gene Expression; Humans; MAP Kinase Signaling System; Matrix Metalloproteinase 9; Matrix Metalloproteinase Inhibitors; Neoplasm Invasiveness; NF-kappa B; Polymerase Chain Reaction; Tetradecanoylphorbol Acetate; Transcription Factor AP-1 | 2010 |
Cordycepin-induced apoptosis and autophagy in breast cancer cells are independent of the estrogen receptor.
Topics: Apoptosis; Autophagy; Breast Neoplasms; Cell Line, Tumor; Deoxyadenosines; Dose-Response Relationship, Drug; Female; Humans; Receptors, Estrogen | 2011 |
The nucleoside antagonist cordycepin causes DNA double strand breaks in breast cancer cells.
Topics: Apoptosis; Ataxia Telangiectasia Mutated Proteins; Breast Neoplasms; Cell Cycle Checkpoints; Cell Cycle Proteins; Cell Death; Cell Line, Tumor; Cell Survival; Deoxyadenosines; DNA Breaks, Double-Stranded; DNA-Binding Proteins; Female; Histones; Humans; MCF-7 Cells; Nucleic Acid Synthesis Inhibitors; Nucleosides; Phosphorylation; Poly Adenosine Diphosphate Ribose; Protein Serine-Threonine Kinases; Proteins; Ribonucleosides; Tumor Suppressor Proteins | 2012 |
Polyadenylate polymerase modulations in human epithelioid cervix and breast cancer cell lines, treated with etoposide or cordycepin, follow cell cycle rather than apoptosis induction.
Topics: Antineoplastic Agents; Apoptosis; Breast Neoplasms; Caspase 6; Caspases; Cell Cycle; Cell Line, Tumor; Deoxyadenosines; DNA Fragmentation; Etoposide; Female; Humans; Isoenzymes; Neoplasms, Glandular and Epithelial; Polynucleotide Adenylyltransferase; Time Factors; Uterine Cervical Neoplasms | 2005 |