coproporphyrinogen iii has been researched along with Porphyria Cutanea Tarda in 1 studies
Porphyria Cutanea Tarda: An autosomal dominant or acquired porphyria due to a deficiency of UROPORPHYRINOGEN DECARBOXYLASE in the LIVER. It is characterized by photosensitivity and cutaneous lesions with little or no neurologic symptoms. Type I is the acquired form and is strongly associated with liver diseases and hepatic toxicities caused by alcohol or estrogenic steroids. Type II is the familial form.
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 1 (100.00) | 29.6817 |
| 2010's | 0 (0.00) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Phillips, JD | 1 |
| Jackson, LK | 1 |
| Bunting, M | 1 |
| Franklin, MR | 1 |
| Thomas, KR | 1 |
| Levy, JE | 1 |
| Andrews, NC | 1 |
| Kushner, JP | 1 |
1 other study available for coproporphyrinogen iii and Porphyria Cutanea Tarda
| Article | Year |
|---|---|
A mouse model of familial porphyria cutanea tarda.
Topics: Aminolevulinic Acid; Animals; Cloning, Molecular; Coproporphyrinogens; Disease Models, Animal; Enzym | 2001 |