Page last updated: 2024-08-23

colforsin and Ventricular Dysfunction, Left

colforsin has been researched along with Ventricular Dysfunction, Left in 6 studies

Research

Studies (6)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's2 (33.33)18.2507
2000's3 (50.00)29.6817
2010's1 (16.67)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Carlson, M; Jones, JA; Ma, YJ; Rajagopalan, V; Zucker, IH1
Houser, SR; Kendrick, ZV; Kolwicz, SC; Libonati, JR; MacDonnell, SM1
Dhar, SK; Gao, S; Ghaleh, B; Guellich, A; Hittinger, L; Hong, C; Ishikawa, Y; Iwatsubo, K; Vatner, DE; Vatner, SF; Wagner, TE; Yan, L1
Baker, AJ; Bergman, MR; Karliner, JS; Lovett, DH; Nguyen, AP; Rodrigo, MC; Simpson, PC; Swigart, PM; Turcato, S; Wang, GY1
Hata, K; Hayashi, Y; Mori, M; Takaoka, H; Takeuchi, M; Yokoyama, M1
Crawford, FA; Doscher, MA; Hebbar, L; Houck, WV; Joshi, JD; Mukherjee, R; Spinale, FG; Thomas, CV; Wang, YH1

Other Studies

6 other study(ies) available for colforsin and Ventricular Dysfunction, Left

ArticleYear
Cardiac ErbB-1/ErbB-2 mutant expression in young adult mice leads to cardiac dysfunction.
    American journal of physiology. Heart and circulatory physiology, 2008, Volume: 295, Issue:2

    Topics: Adenylyl Cyclases; Animals; Cardiomyopathy, Dilated; Cardiotonic Agents; Colforsin; Dobutamine; ErbB Receptors; Female; Heart Failure; Male; Mice; Mice, Inbred C57BL; Mice, Inbred DBA; Mice, Transgenic; Mutation; Myocardial Contraction; Myocytes, Cardiac; Phenotype; Phosphorylation; Receptor, ErbB-2; Signal Transduction; Time Factors; Ventricular Dysfunction, Left

2008
Voluntary wheel running and pacing-induced dysfunction in hypertension.
    Clinical and experimental hypertension (New York, N.Y. : 1993), 2008, Volume: 30, Issue:7

    Topics: Adenylyl Cyclases; Animals; Blood Pressure; Cardiac Pacing, Artificial; Colforsin; Female; Heart Rate; Hypertension; In Vitro Techniques; Physical Exertion; Rats; Rats, Inbred SHR; Rats, Inbred WKY; Receptors, Adrenergic, beta; Ventricular Dysfunction, Left

2008
Effects of cardiac overexpression of type 6 adenylyl cyclase affects on the response to chronic pressure overload.
    American journal of physiology. Heart and circulatory physiology, 2010, Volume: 299, Issue:3

    Topics: Adenylyl Cyclases; Analysis of Variance; Animals; Apoptosis; Colforsin; Echocardiography; Heart; Heart Rate; Heart Ventricles; Hemodynamics; Hypertrophy, Left Ventricular; Isoproterenol; Mice; Mice, Transgenic; Myocardial Contraction; Myocardium; Stress, Physiological; Ventricular Dysfunction, Left

2010
Cardiac transgenic matrix metalloproteinase-2 expression directly induces impaired contractility.
    Cardiovascular research, 2006, Feb-15, Volume: 69, Issue:3

    Topics: Actin Cytoskeleton; Adenylyl Cyclases; Adrenergic beta-Antagonists; Animals; Calcium; Colforsin; Electric Stimulation; Endoplasmic Reticulum; Fibroblasts; In Vitro Techniques; Isoproterenol; Matrix Metalloproteinase 2; Mice; Mice, Transgenic; Microscopy, Electron; Microscopy, Fluorescence; Myocardial Contraction; Myocardium; Sarcomeres; Stimulation, Chemical; Ventricular Dysfunction, Left; Ventricular Remodeling

2006
Effect of NKH477, a new water-soluble forskolin derivative, on arterial-ventricular coupling and mechanical energy transduction in patients with left ventricular systolic dysfunction: comparison with dobutamine.
    Journal of cardiovascular pharmacology, 1994, Volume: 24, Issue:2

    Topics: Adult; Aged; Cardiotonic Agents; Colforsin; Dobutamine; Energy Metabolism; Female; Heart; Hemodynamics; Humans; Male; Middle Aged; Systole; Ventricular Dysfunction, Left

1994
Downstream defects in beta-adrenergic signaling and relation to myocyte contractility after cardioplegic arrest.
    The Journal of thoracic and cardiovascular surgery, 1998, Volume: 115, Issue:1

    Topics: 3-Pyridinecarboxylic acid, 1,4-dihydro-2,6-dimethyl-5-nitro-4-(2-(trifluoromethyl)phenyl)-, Methyl ester; Adenylyl Cyclases; Adrenergic beta-Agonists; Animals; Calcium; Calcium Channel Agonists; Calcium Channels; Colforsin; Heart Arrest, Induced; Isoproterenol; Myocardial Contraction; Receptors, Adrenergic, beta; Signal Transduction; Swine; Time Factors; Ventricular Dysfunction, Left

1998