colforsin has been researched along with Ventricular Dysfunction, Left in 6 studies
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 2 (33.33) | 18.2507 |
2000's | 3 (50.00) | 29.6817 |
2010's | 1 (16.67) | 24.3611 |
2020's | 0 (0.00) | 2.80 |
Authors | Studies |
---|---|
Carlson, M; Jones, JA; Ma, YJ; Rajagopalan, V; Zucker, IH | 1 |
Houser, SR; Kendrick, ZV; Kolwicz, SC; Libonati, JR; MacDonnell, SM | 1 |
Dhar, SK; Gao, S; Ghaleh, B; Guellich, A; Hittinger, L; Hong, C; Ishikawa, Y; Iwatsubo, K; Vatner, DE; Vatner, SF; Wagner, TE; Yan, L | 1 |
Baker, AJ; Bergman, MR; Karliner, JS; Lovett, DH; Nguyen, AP; Rodrigo, MC; Simpson, PC; Swigart, PM; Turcato, S; Wang, GY | 1 |
Hata, K; Hayashi, Y; Mori, M; Takaoka, H; Takeuchi, M; Yokoyama, M | 1 |
Crawford, FA; Doscher, MA; Hebbar, L; Houck, WV; Joshi, JD; Mukherjee, R; Spinale, FG; Thomas, CV; Wang, YH | 1 |
6 other study(ies) available for colforsin and Ventricular Dysfunction, Left
Article | Year |
---|---|
Cardiac ErbB-1/ErbB-2 mutant expression in young adult mice leads to cardiac dysfunction.
Topics: Adenylyl Cyclases; Animals; Cardiomyopathy, Dilated; Cardiotonic Agents; Colforsin; Dobutamine; ErbB Receptors; Female; Heart Failure; Male; Mice; Mice, Inbred C57BL; Mice, Inbred DBA; Mice, Transgenic; Mutation; Myocardial Contraction; Myocytes, Cardiac; Phenotype; Phosphorylation; Receptor, ErbB-2; Signal Transduction; Time Factors; Ventricular Dysfunction, Left | 2008 |
Voluntary wheel running and pacing-induced dysfunction in hypertension.
Topics: Adenylyl Cyclases; Animals; Blood Pressure; Cardiac Pacing, Artificial; Colforsin; Female; Heart Rate; Hypertension; In Vitro Techniques; Physical Exertion; Rats; Rats, Inbred SHR; Rats, Inbred WKY; Receptors, Adrenergic, beta; Ventricular Dysfunction, Left | 2008 |
Effects of cardiac overexpression of type 6 adenylyl cyclase affects on the response to chronic pressure overload.
Topics: Adenylyl Cyclases; Analysis of Variance; Animals; Apoptosis; Colforsin; Echocardiography; Heart; Heart Rate; Heart Ventricles; Hemodynamics; Hypertrophy, Left Ventricular; Isoproterenol; Mice; Mice, Transgenic; Myocardial Contraction; Myocardium; Stress, Physiological; Ventricular Dysfunction, Left | 2010 |
Cardiac transgenic matrix metalloproteinase-2 expression directly induces impaired contractility.
Topics: Actin Cytoskeleton; Adenylyl Cyclases; Adrenergic beta-Antagonists; Animals; Calcium; Colforsin; Electric Stimulation; Endoplasmic Reticulum; Fibroblasts; In Vitro Techniques; Isoproterenol; Matrix Metalloproteinase 2; Mice; Mice, Transgenic; Microscopy, Electron; Microscopy, Fluorescence; Myocardial Contraction; Myocardium; Sarcomeres; Stimulation, Chemical; Ventricular Dysfunction, Left; Ventricular Remodeling | 2006 |
Effect of NKH477, a new water-soluble forskolin derivative, on arterial-ventricular coupling and mechanical energy transduction in patients with left ventricular systolic dysfunction: comparison with dobutamine.
Topics: Adult; Aged; Cardiotonic Agents; Colforsin; Dobutamine; Energy Metabolism; Female; Heart; Hemodynamics; Humans; Male; Middle Aged; Systole; Ventricular Dysfunction, Left | 1994 |
Downstream defects in beta-adrenergic signaling and relation to myocyte contractility after cardioplegic arrest.
Topics: 3-Pyridinecarboxylic acid, 1,4-dihydro-2,6-dimethyl-5-nitro-4-(2-(trifluoromethyl)phenyl)-, Methyl ester; Adenylyl Cyclases; Adrenergic beta-Agonists; Animals; Calcium; Calcium Channel Agonists; Calcium Channels; Colforsin; Heart Arrest, Induced; Isoproterenol; Myocardial Contraction; Receptors, Adrenergic, beta; Signal Transduction; Swine; Time Factors; Ventricular Dysfunction, Left | 1998 |