Page last updated: 2024-08-23

colforsin and Substance Withdrawal Syndrome

colforsin has been researched along with Substance Withdrawal Syndrome in 16 studies

Research

Studies (16)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's8 (50.00)18.2507
2000's7 (43.75)29.6817
2010's1 (6.25)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Adan, RA; Meye, FJ; Ramakers, GM; Smidt, MP; van Zessen, R1
Cousins, MM; Seidler, FJ; Slikker, W; Slotkin, TA; Xu, Z1
Christie, MJ; Hack, SP; Vaughan, CW1
Carlezon, WA; Chartoff, EH; Konradi, C; Papadopoulou, M1
Cousins, MM; Seidler, FJ; Slotkin, TA; Tate, CA1
Bonci, A; Williams, JT1
Kaplan, GB; Leite-Morris, KA; McClelland, EG; Sethi, RK1
Ho, IK; Oh, S; Wellman, SE1
Ammer, H; Schulz, R1
Ho, IK; Jang, CG; Rockhold, RW1
Chen, JC; Hsieh, MM; Huang, LI; Su, HJ1
Aghajanian, GK; Jolas, T; Nestler, EJ1
Aghajanian, GK; Akbarian, S; Bates, B; Bothwell, M; Coppola, V; Fan, G; Jaenisch, R; Kosofsky, BE; Kucera, J; Liu, RJ; Nestler, EJ; Pejchal, T; Skirboll, SL; Sun, LD; Taylor, JR; Tessarollo, L; Wilson, MA1
Aghajanian, GK; Akbarian, S; Coppola, V; Fong, HF; Gold, SJ; Jaenisch, R; Jones, KR; Liu, RJ; Nestler, EJ; Rios, M; Tessarollo, L; Zeiler, S1
Bass, PP; Olson, KG; Pugh, G; Welch, SP1
De Vries, TJ; Mulder, AH; Schoffelmeer, AN; Van Vliet, BJ; Wardeh, G1

Other Studies

16 other study(ies) available for colforsin and Substance Withdrawal Syndrome

ArticleYear
Morphine withdrawal enhances constitutive μ-opioid receptor activity in the ventral tegmental area.
    The Journal of neuroscience : the official journal of the Society for Neuroscience, 2012, Nov-14, Volume: 32, Issue:46

    Topics: Adenylyl Cyclases; Analgesics, Opioid; Analysis of Variance; Animals; Colforsin; Cyclic AMP; Dopaminergic Neurons; Enkephalin, Ala(2)-MePhe(4)-Gly(5)-; Enzyme Activators; Female; gamma-Aminobutyric Acid; Male; Mice; Mice, Inbred C57BL; Morphine; Pregnancy; Receptors, Opioid, mu; Substance Withdrawal Syndrome; Ventral Tegmental Area

2012
Adolescent nicotine administration alters serotonin receptors and cell signaling mediated through adenylyl cyclase.
    Brain research, 2002, Oct-04, Volume: 951, Issue:2

    Topics: Adenylyl Cyclases; Adrenergic beta-Agonists; Aging; Animals; Brain Chemistry; Colforsin; Female; GTP-Binding Proteins; Isoproterenol; Male; Nicotine; Nicotinic Agonists; Radioligand Assay; Rats; Rats, Sprague-Dawley; Receptors, Serotonin; Receptors, Serotonin, 5-HT1; Serotonin; Signal Transduction; Substance Withdrawal Syndrome; Up-Regulation

2002
Modulation of GABA release during morphine withdrawal in midbrain neurons in vitro.
    Neuropharmacology, 2003, Volume: 45, Issue:5

    Topics: Action Potentials; Adenosine; Affinity Labels; Animals; Colforsin; Cyclic AMP; Dipyridamole; Dose-Response Relationship, Drug; Drug Interactions; Enkephalins; Enzyme Inhibitors; gamma-Aminobutyric Acid; In Vitro Techniques; Isoquinolines; Male; Mesencephalon; Mice; Mice, Inbred C57BL; Morphine; Morphine Dependence; Naloxone; Narcotic Antagonists; Narcotics; Neural Inhibition; Neurons; Patch-Clamp Techniques; Periaqueductal Gray; Probenecid; Purinergic P1 Receptor Agonists; Purinergic P1 Receptor Antagonists; Substance Withdrawal Syndrome; Sulfonamides; Synaptic Transmission; Thioinosine; Time Factors; Uricosuric Agents; Vasodilator Agents; Xanthines

2003
Dopamine-dependent increases in phosphorylation of cAMP response element binding protein (CREB) during precipitated morphine withdrawal in primary cultures of rat striatum.
    Journal of neurochemistry, 2003, Volume: 87, Issue:1

    Topics: Animals; Cells, Cultured; Colforsin; Corpus Striatum; Cyclic AMP Response Element-Binding Protein; Cyclic AMP-Dependent Protein Kinases; Dopamine; Dopamine Agonists; Enzyme Inhibitors; Morphine; Naloxone; Narcotic Antagonists; Narcotics; Phosphorylation; Proto-Oncogene Proteins c-fos; Rats; Rats, Sprague-Dawley; RNA, Messenger; Substance Withdrawal Syndrome

2003
Prenatal nicotine exposure alters the responses to subsequent nicotine administration and withdrawal in adolescence: Serotonin receptors and cell signaling.
    Neuropsychopharmacology : official publication of the American College of Neuropsychopharmacology, 2006, Volume: 31, Issue:11

    Topics: Age Factors; Analysis of Variance; Animals; Brain; Colforsin; Female; Male; Nicotine; Nicotinic Agonists; Pregnancy; Prenatal Exposure Delayed Effects; Protein Binding; Rats; Receptors, Serotonin; Sex Factors; Signal Transduction; Substance Withdrawal Syndrome

2006
Increased probability of GABA release during withdrawal from morphine.
    The Journal of neuroscience : the official journal of the Society for Neuroscience, 1997, Jan-15, Volume: 17, Issue:2

    Topics: 2-Amino-5-phosphonovalerate; 6-Cyano-7-nitroquinoxaline-2,3-dione; Action Potentials; Adenylyl Cyclase Inhibitors; Adenylyl Cyclases; Animals; Colforsin; Cyclic AMP; Cyclic AMP-Dependent Protein Kinases; Dopamine; Dopamine Antagonists; Enzyme Activation; Enzyme Inhibitors; Excitatory Amino Acid Antagonists; GABA Antagonists; gamma-Aminobutyric Acid; Guinea Pigs; Interneurons; Morphine; Nerve Tissue Proteins; Organophosphorus Compounds; Patch-Clamp Techniques; Phorbol 12,13-Dibutyrate; Picrotoxin; Receptors, GABA-A; Salicylamides; Serotonin; Signal Transduction; Staurosporine; Strychnine; Substance Withdrawal Syndrome; Tegmentum Mesencephali; Thionucleotides

1997
Regulation of G protein-mediated adenylyl cyclase in striatum and cortex of opiate-dependent and opiate withdrawing mice.
    Brain research, 1998, Mar-30, Volume: 788, Issue:1-2

    Topics: Adenylyl Cyclases; Analysis of Variance; Animals; Cerebral Cortex; Colforsin; Corpus Striatum; GTP-Binding Proteins; Logistic Models; Male; Mice; Mice, Inbred Strains; Morphine; Morphine Dependence; Substance Withdrawal Syndrome; Telencephalon

1998
Changes in [3H]forskolin binding to adenylate cyclase and [3H]phorbol dibutyrate binding to protein kinase C in pentobarbital tolerant/dependent rats.
    Neurochemical research, 1998, Volume: 23, Issue:4

    Topics: Adenylyl Cyclases; Animals; Brain; Cerebral Ventricles; Colforsin; Drug Tolerance; Infusions, Parenteral; Male; Organ Specificity; Pentobarbital; Phorbol 12,13-Dibutyrate; Protein Binding; Protein Kinase C; Rats; Rats, Sprague-Dawley; Substance Withdrawal Syndrome; Substance-Related Disorders

1998
Adenylyl cyclase supersensitivity in opioid-withdrawn NG108-15 hybrid cells requires Gs but is not mediated by the Gsalpha subunit.
    The Journal of pharmacology and experimental therapeutics, 1998, Volume: 286, Issue:2

    Topics: Adenylyl Cyclases; Alprostadil; Cell Line; Colforsin; GTP-Binding Protein alpha Subunits, Gs; Humans; Hybrid Cells; Morphine; Naloxone; Narcotic Antagonists; Narcotics; Receptors, Opioid, delta; Receptors, Prostaglandin; Signal Transduction; Substance Withdrawal Syndrome

1998
Withdrawal from butorphanol dependence alters binding of [3H]phorbol dibutyrate to protein kinase C, but not of [3H]forskolin to adenylate cyclase.
    Brain research, 1999, Mar-13, Volume: 821, Issue:2

    Topics: Adenylyl Cyclases; Animals; Autoradiography; Brain; Brain Chemistry; Butorphanol; Carcinogens; Colforsin; Male; Narcotic Antagonists; Opioid-Related Disorders; Phorbol 12,13-Dibutyrate; Protein Kinase C; Rats; Rats, Sprague-Dawley; Substance Withdrawal Syndrome; Tritium

1999
Reductions in binding and functions of D2 dopamine receptors in the rat ventral striatum during amphetamine sensitization.
    Life sciences, 1999, Volume: 64, Issue:5

    Topics: Adenylyl Cyclases; Amphetamine; Analysis of Variance; Animals; Bromocriptine; Colforsin; Corpus Striatum; Cyclic AMP; Drug Administration Schedule; GTP-Binding Proteins; Guanylyl Imidodiphosphate; Injections, Intraperitoneal; Male; Motor Activity; Quinpirole; Rats; Rats, Sprague-Dawley; Receptors, Dopamine D2; Stereotyped Behavior; Substance Withdrawal Syndrome; Time Factors

1999
Chronic morphine increases GABA tone on serotonergic neurons of the dorsal raphe nucleus: association with an up-regulation of the cyclic AMP pathway.
    Neuroscience, 2000, Volume: 95, Issue:2

    Topics: Adenylyl Cyclases; Analgesics, Opioid; Animals; Cardiovascular Agents; Colforsin; Cyclic AMP; Cyclic AMP-Dependent Protein Kinases; Electrophysiology; Enkephalin, Ala(2)-MePhe(4)-Gly(5)-; Enzyme Inhibitors; Excitatory Amino Acid Agonists; gamma-Aminobutyric Acid; In Vitro Techniques; Interneurons; Isoquinolines; Male; Membrane Potentials; Morphine; N-Methylaspartate; Neural Inhibition; Periaqueductal Gray; Phenylephrine; Pyrimidines; Raphe Nuclei; Rats; Rats, Sprague-Dawley; Serotonin; Substance Withdrawal Syndrome; Sulfonamides; Sympathomimetics; Tetrazoles; Tetrodotoxin; Thionucleotides

2000
Neurotrophin-3 modulates noradrenergic neuron function and opiate withdrawal.
    Molecular psychiatry, 2001, Volume: 6, Issue:5

    Topics: Aging; Animals; Avoidance Learning; Brain; Colforsin; Cyclic AMP; Electric Stimulation; Enkephalin, Ala(2)-MePhe(4)-Gly(5)-; Gene Expression Regulation, Enzymologic; In Vitro Techniques; Intermediate Filament Proteins; Locus Coeruleus; Mice; Mice, Knockout; Mice, Transgenic; Morphine; Morphine Dependence; Nerve Tissue Proteins; Nestin; Neurons; Neurotrophin 3; Signal Transduction; Substance Withdrawal Syndrome; Tyrosine 3-Monooxygenase

2001
Brain-derived neurotrophic factor is essential for opiate-induced plasticity of noradrenergic neurons.
    The Journal of neuroscience : the official journal of the Society for Neuroscience, 2002, May-15, Volume: 22, Issue:10

    Topics: Animals; Behavior, Animal; Brain-Derived Neurotrophic Factor; Calcium-Calmodulin-Dependent Protein Kinase Type 2; Calcium-Calmodulin-Dependent Protein Kinases; Cell Count; Colforsin; Cyclic AMP; Down-Regulation; Gene Deletion; Genes, Reporter; Genotype; In Situ Hybridization; In Vitro Techniques; Integrases; Locus Coeruleus; Mice; Mice, Mutant Strains; Mice, Transgenic; Narcotics; Neuronal Plasticity; Neurons; Norepinephrine; Receptors, Opioid, mu; Recombination, Genetic; Severity of Illness Index; Signal Transduction; Substance Withdrawal Syndrome; Transgenes; Tyrosine 3-Monooxygenase; Viral Proteins

2002
Morphine-induced modulation of calcitonin gene-related peptide levels.
    Pharmacology, biochemistry, and behavior, 1992, Volume: 43, Issue:4

    Topics: Adenylyl Cyclases; Animals; Behavior, Animal; Calcitonin Gene-Related Peptide; Colforsin; Cyclic AMP; Drug Implants; Drug Tolerance; Enzyme Activation; In Vitro Techniques; Mice; Morphine; Naloxone; Rats; Spinal Cord; Substance Withdrawal Syndrome; Synaptosomes

1992
mu-Opioid receptor-regulated adenylate cyclase activity in primary cultures of rat striatal neurons upon chronic morphine exposure.
    European journal of pharmacology, 1991, Oct-14, Volume: 208, Issue:2

    Topics: 2,3,4,5-Tetrahydro-7,8-dihydroxy-1-phenyl-1H-3-benzazepine; Adenylyl Cyclases; Animals; Cells, Cultured; Colforsin; Corpus Striatum; Morphine; Neurons; Rats; Rats, Inbred Strains; Receptors, Adrenergic, beta; Receptors, Dopamine; Receptors, Opioid; Receptors, Opioid, mu; Stimulation, Chemical; Substance Withdrawal Syndrome

1991