colforsin has been researched along with Shock, Septic in 8 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 5 (62.50) | 18.2507 |
| 2000's | 1 (12.50) | 29.6817 |
| 2010's | 2 (25.00) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Böhm, M; Lenz, M; Link, A; Maack, C; Selejan, S | 1 |
| Liu, J; Qian, X; Zhang, J | 1 |
| Hattori, Y; Oishi, H; Takano, K; Takebe, M; Tomita, K; Yamazaki, M; Yokoo, H | 1 |
| Metakis, L; Notterman, DA; Singh, M | 1 |
| Fischer, W; Schudt, C; Wendel, A | 1 |
| De Souza, EB; Hashimoto, K; Takao, T | 1 |
| Asahi, M; Fujii, J; Fujiwara, N; Kaneto, H; Seo, HG; Taniguchi, N | 1 |
| Mustafa, SB; Olson, MS | 1 |
1 review(s) available for colforsin and Shock, Septic
| Article | Year |
|---|---|
Interleukin-1 receptors in the brain-endocrine-immune axis. Modulation by stress and infection.
Topics: Animals; Colforsin; Corticotropin-Releasing Hormone; Cyclic AMP; Ether; Isoproterenol; Laparotomy; Mice; Pituitary Gland; Receptors, Interleukin-1; Shock, Septic; Stress, Physiological; Tumor Cells, Cultured | 1995 |
7 other study(ies) available for colforsin and Shock, Septic
| Article | Year |
|---|---|
Phosphodiesterase 4 inhibition but not beta-adrenergic stimulation suppresses tumor necrosis factor-alpha release in peripheral blood mononuclear cells in septic shock.
Topics: Adrenergic beta-2 Receptor Agonists; Case-Control Studies; Colforsin; Flow Cytometry; GTP-Binding Proteins; Humans; Isoproterenol; Leukocytes, Mononuclear; Phosphodiesterase 4 Inhibitors; Phosphodiesterase Inhibitors; Rolipram; Shock, Septic; Toll-Like Receptor 2; Toll-Like Receptor 4; Tumor Necrosis Factor-alpha | 2008 |
Tumor-secreted PGE2 inhibits CCL5 production in activated macrophages through cAMP/PKA signaling pathway.
Topics: Animals; Anti-Inflammatory Agents, Non-Steroidal; Bacterial Infections; Cell Line; Chemokine CCL5; Colforsin; Cyclic AMP; Cyclic AMP-Dependent Protein Kinases; Dinoprostone; Interferon-gamma; Interleukin-2; Isoquinolines; Lipopolysaccharides; Macrophage Activation; Macrophages; Male; Mice; Mice, Inbred BALB C; Neoplasms, Experimental; Nitrobenzenes; Prostaglandin Antagonists; Protein Kinase Inhibitors; Shock, Septic; Signal Transduction; Sulfonamides; Tumor Escape; Xanthones | 2011 |
Olprinone and colforsin daropate alleviate septic lung inflammation and apoptosis through CREB-independent activation of the Akt pathway.
Topics: Acute Lung Injury; Androstadienes; Animals; Apoptosis; Cecum; Colforsin; Cyclic AMP Response Element-Binding Protein; Cytokines; Enzyme Activation; Enzyme Activators; Hypotension; Imidazoles; Ligation; Lung; Male; Mice; Mice, Inbred BALB C; NF-kappa B; Phosphatidylinositol 3-Kinases; Phosphodiesterase Inhibitors; Phosphoinositide-3 Kinase Inhibitors; Pneumonia; Proto-Oncogene Proteins c-akt; Pyridones; Shock, Septic; Signal Transduction; Wortmannin | 2012 |
Tumor necrosis factor produces homologous desensitization of lymphocyte beta 2-adrenergic responses.
Topics: Alprostadil; Colforsin; Cyclic AMP; Humans; In Vitro Techniques; Isoproterenol; Lymphocytes; Receptors, Adrenergic, beta; Shock, Septic; Tumor Necrosis Factor-alpha | 1993 |
Protection by phosphodiesterase inhibitors against endotoxin-induced liver injury in galactosamine-sensitized mice.
Topics: Animals; Cells, Cultured; Colforsin; Cyclic AMP; Dinoprostone; Galactosamine; Hepatitis; Lipopolysaccharides; Liver; Macrophages; Male; Mice; Phosphodiesterase Inhibitors; Pyridazines; Pyrrolidinones; Rolipram; Shock, Septic; Tumor Necrosis Factor-alpha | 1993 |
Effect of a nitric oxide synthase inhibitor, S-ethylisothiourea, on cultured cells and cardiovascular functions of normal and lipopolysaccharide-treated rabbits.
Topics: Animals; Blood Pressure; Brain; Cells, Cultured; Citrulline; Colforsin; DNA; Dose-Response Relationship, Drug; Enzyme Inhibitors; Heart Rate; Interleukin-1; Isothiuronium; L-Lactate Dehydrogenase; Lipopolysaccharides; Nitric Oxide Synthase; Nitrites; Nucleosomes; Rabbits; Rats; Shock, Septic | 1996 |
Expression of nitric-oxide synthase in rat Kupffer cells is regulated by cAMP.
Topics: Animals; Biological Transport; Cell Nucleus; Cells, Cultured; Colforsin; Cyclic AMP; Cytoplasm; DNA-Binding Proteins; Gene Expression Regulation, Enzymologic; I-kappa B Proteins; Kupffer Cells; Lipopolysaccharides; Liver; NF-kappa B; NF-KappaB Inhibitor alpha; Nitric Oxide Synthase; Nitric Oxide Synthase Type II; Nitrites; Rats; RNA, Messenger; Shock, Septic; Transcription, Genetic | 1998 |