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colforsin and Huntington Disease

colforsin has been researched along with Huntington Disease in 4 studies

Research

Studies (4)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's1 (25.00)18.2507
2000's3 (75.00)29.6817
2010's0 (0.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Cuddon, P; Fleming, A; Floto, RA; Goldsmith, P; Jahreiss, L; O'Kane, CJ; Pask, D; Rubinsztein, DC; Saiki, S; Sarkar, S; Siddiqi, FH; Ttofi, EK; Williams, A1
Brose, N; Gibson, HE; Jones, S; Morton, AJ; Reim, K1
Aronin, N; Chase, K; Difiglia, M; Hayden, MR; Heller, A; Kalchman, MA; Kim, M; Laforet, G; Velier, J; Won, L1
Abbracchio, MP; Borea, PA; Camurri, A; Cattabeni, F; Cattaneo, E; Rigamonti, D; Sipione, S; Varani, K1

Other Studies

4 other study(ies) available for colforsin and Huntington Disease

ArticleYear
Novel targets for Huntington's disease in an mTOR-independent autophagy pathway.
    Nature chemical biology, 2008, Volume: 4, Issue:5

    Topics: Animals; Autophagy; Calcium Channels, L-Type; Clonidine; Cyclic AMP; Humans; Huntington Disease; Imidazoline Receptors; Minoxidil; Protein Kinases; Signal Transduction; TOR Serine-Threonine Kinases; Type C Phospholipases; Verapamil

2008
A similar impairment in CA3 mossy fibre LTP in the R6/2 mouse model of Huntington's disease and in the complexin II knockout mouse.
    The European journal of neuroscience, 2005, Volume: 22, Issue:7

    Topics: 2-Amino-5-phosphonovalerate; Adaptor Proteins, Vesicular Transport; Age Factors; Animals; Anticonvulsants; Colforsin; Cyclopropanes; Disease Models, Animal; Dose-Response Relationship, Drug; Dose-Response Relationship, Radiation; Drug Interactions; Electric Stimulation; Excitatory Amino Acid Antagonists; Excitatory Postsynaptic Potentials; Glycine; Hippocampus; Huntington Disease; In Vitro Techniques; Long-Term Potentiation; Mice; Mice, Knockout; Mossy Fibers, Hippocampal; Nerve Tissue Proteins; Patch-Clamp Techniques; Time Factors

2005
Forskolin and dopamine D1 receptor activation increase huntingtin's association with endosomes in immortalized neuronal cells of striatal origin.
    Neuroscience, 1999, Volume: 89, Issue:4

    Topics: Animals; Cell Line, Transformed; Clathrin; Colforsin; Corpus Striatum; Dopamine; Endocytosis; Endosomes; Huntingtin Protein; Huntington Disease; Mice; Mice, Inbred C57BL; Nerve Tissue Proteins; Neuroblastoma; Neurons; Nuclear Proteins; Receptors, Dopamine D1; Tumor Cells, Cultured

1999
Aberrant amplification of A(2A) receptor signaling in striatal cells expressing mutant huntingtin.
    FASEB journal : official publication of the Federation of American Societies for Experimental Biology, 2001, Volume: 15, Issue:7

    Topics: Adenosine-5'-(N-ethylcarboxamide); Adenylyl Cyclases; Cell Line; Colforsin; Corpus Striatum; Cyclic AMP; Humans; Huntingtin Protein; Huntington Disease; Kinetics; Models, Biological; Mutation; Nerve Tissue Proteins; Nuclear Proteins; Purinergic P1 Receptor Antagonists; Radioligand Assay; Receptor, Adenosine A2A; Receptors, Purinergic P1; Second Messenger Systems; Signal Transduction

2001