cobaltous chloride has been researched along with Bone Cancer in 3 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 1 (33.33) | 18.2507 |
| 2000's | 1 (33.33) | 29.6817 |
| 2010's | 1 (33.33) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Guo, CH; Hao, CP; Ling, M; Ma, W; Wu, XY | 1 |
| Berra, E; Delaunay, F; Dulong, S; Filipski, E; Grechez-Cassiau, A; Guillaumond, F; Lacoche, S; Lévi, F; Li, XM; Teboul, M | 1 |
| An, WG; Blagosklonny, MV; Costa, M; Melillo, G; Salnikow, K | 1 |
3 other study(ies) available for cobaltous chloride and Bone Cancer
| Article | Year |
|---|---|
Hypoxia-induced apoptosis is blocked by adrenomedullin via upregulation of Bcl-2 in human osteosarcoma cells.
Topics: Adrenomedullin; Apoptosis; Bone Neoplasms; Cell Hypoxia; Cell Line, Tumor; Cell Proliferation; Cobalt; Gene Expression Regulation, Neoplastic; Humans; Hypoxia-Inducible Factor 1, alpha Subunit; MAP Kinase Signaling System; Osteosarcoma; Proto-Oncogene Proteins c-bcl-2 | 2015 |
Altered Stra13 and Dec2 circadian gene expression in hypoxic cells.
Topics: Animals; ARNTL Transcription Factors; Basic Helix-Loop-Helix Transcription Factors; Bone Neoplasms; Cell Hypoxia; Circadian Rhythm; CLOCK Proteins; Cobalt; DNA-Binding Proteins; Fibroblasts; Gene Expression; Gene Expression Regulation, Neoplastic; Homeodomain Proteins; Hypoxia-Inducible Factor 1; Male; Mice; Mice, Inbred Strains; Nuclear Receptor Subfamily 1, Group D, Member 1; Osteosarcoma; Promoter Regions, Genetic; Receptors, Cytoplasmic and Nuclear; Trans-Activators; Transcription Factors; Transcriptional Activation | 2008 |
Nickel-induced transformation shifts the balance between HIF-1 and p53 transcription factors.
Topics: 3T3 Cells; Adenocarcinoma; Animals; Bone Neoplasms; Breast Neoplasms; Bronchi; Cadmium Chloride; Carcinogens, Environmental; Cell Hypoxia; Cell Transformation, Neoplastic; Cobalt; Cricetinae; Cricetulus; DNA-Binding Proteins; Epithelial Cells; Fibroblasts; Gene Expression Regulation; Genes, p53; Humans; Hypoxia-Inducible Factor 1; Hypoxia-Inducible Factor 1, alpha Subunit; Lung Neoplasms; Mice; Neoplasm Proteins; Nickel; Nuclear Proteins; Osteosarcoma; Promoter Regions, Genetic; Recombinant Fusion Proteins; Transcription Factors; Transcription, Genetic; Tumor Cells, Cultured; Tumor Suppressor Protein p53 | 1999 |