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clonazepam and Brain Ischemia

clonazepam has been researched along with Brain Ischemia in 5 studies

Clonazepam: An anticonvulsant used for several types of seizures, including myotonic or atonic seizures, photosensitive epilepsy, and absence seizures, although tolerance may develop. It is seldom effective in generalized tonic-clonic or partial seizures. The mechanism of action appears to involve the enhancement of GAMMA-AMINOBUTYRIC ACID receptor responses.
clonazepam : 1,3-Dihydro-2H-1,4-benzodiazepin-2-one in which the hydrogens at positions 5 and 7 are substituted by 2-chlorophenyl and nitro groups, respectively. It is used in the treatment of all types of epilepsy and seizures, as well as myoclonus and associated abnormal movements, and panic disorders. However, its use can be limited by the development of tolerance and by sedation.

Brain Ischemia: Localized reduction of blood flow to brain tissue due to arterial obstruction or systemic hypoperfusion. This frequently occurs in conjunction with brain hypoxia (HYPOXIA, BRAIN). Prolonged ischemia is associated with BRAIN INFARCTION.

Research Excerpts

ExcerptRelevanceReference
"Hemiballismus is characterized by the abrupt onset of violent proximal flinging movements, affecting the limbs, neck and trunk on one side of the body."1.32Hemiballismus following general anesthesia. A case report. ( Buraczyńska, K; Pilarczyk, M; Sagan, M; Stelmasiak, Z; Tynecka, M, 2003)
"The woman had cardiac arrest complicated by ischemic stroke involving the left parietal region."1.29[Myoclonus of focal action and localized hemispheric lesion. A polygraphic and pharmacological study]. ( Bartolomei, F; Bureau, M; Genton, P; Paglia, G; Roger, J, 1995)
"Under conditions of bilateral cerebral ischemia, in which both carotid arteries were clamped, only irreversible ischemia (60 min) followed by reflow, was associated with highly significant damage to cerebral Na+, K+-ATPase."1.27Classification of ischemic-induced damage to Na+, K+-ATPase in gerbil forebrain. Modification by therapeutic agents. ( Callahan, AS; Christie-Pope, BC; Eddy, LJ; Palmer, GC; Palmer, SJ; Taylor, MD, 1985)

Research

Studies (5)

TimeframeStudies, this research(%)All Research%
pre-19902 (40.00)18.7374
1990's2 (40.00)18.2507
2000's1 (20.00)29.6817
2010's0 (0.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Pilarczyk, M1
Buraczyńska, K1
Sagan, M1
Tynecka, M1
Stelmasiak, Z1
Bartolomei, F1
Bureau, M1
Paglia, G1
Genton, P1
Roger, J1
Mehsen, M1
Liguoro, D1
Besse, JL1
Marguinaud, E1
Crozat, P1
Ravussin, P1
Boulard, G1
Ahlemeyer, B1
Krieglstein, J1
Palmer, GC1
Palmer, SJ1
Christie-Pope, BC1
Callahan, AS1
Taylor, MD1
Eddy, LJ1

Other Studies

5 other studies available for clonazepam and Brain Ischemia

ArticleYear
Hemiballismus following general anesthesia. A case report.
    Neurological sciences : official journal of the Italian Neurological Society and of the Italian Society of Clinical Neurophysiology, 2003, Volume: 24, Issue:4

    Topics: Anesthesia, General; Anticonvulsants; Antipsychotic Agents; Brain Ischemia; Clonazepam; Dyskinesias;

2003
[Myoclonus of focal action and localized hemispheric lesion. A polygraphic and pharmacological study].
    Revue neurologique, 1995, Volume: 151, Issue:5

    Topics: Anticonvulsants; Brain Diseases; Brain Ischemia; Cerebral Cortex; Clonazepam; Electroencephalography

1995
[Pre- and postoperative management of early aneurysm surgery].
    Minerva anestesiologica, 1993, Volume: 59, Issue:11

    Topics: Acetaminophen; Adult; Aged; Brain Ischemia; Calcium Channel Blockers; Cerebrospinal Fluid Shunts; Cl

1993
Testing drug effects against hypoxic damage of cultured neurons during long-term recovery.
    Life sciences, 1989, Volume: 45, Issue:9

    Topics: Adenosine Triphosphate; Animals; Anticonvulsants; Brain Ischemia; Cell Count; Cell Survival; Cells,

1989
Classification of ischemic-induced damage to Na+, K+-ATPase in gerbil forebrain. Modification by therapeutic agents.
    Neuropharmacology, 1985, Volume: 24, Issue:6

    Topics: Adenosine Triphosphatases; Allopurinol; Animals; Brain; Brain Ischemia; Ca(2+) Mg(2+)-ATPase; Calciu

1985