Page last updated: 2024-10-25

ciglitazone and Pneumococcal Infections

ciglitazone has been researched along with Pneumococcal Infections in 1 studies

ciglitazone: structure given in second source; PPAR agonist used for type II diabetes
ciglitazone : An aromatic ether that consists of 1,3-thiazolidine-2,4-dione with position 5 substituted by a 4-[(1-methylcyclohexyl)methoxy]benzyl group. A selective PPARgamma agonist.

Pneumococcal Infections: Infections with bacteria of the species STREPTOCOCCUS PNEUMONIAE.

Research Excerpts

ExcerptRelevanceReference
" Because patients with type 2 diabetes have an increased risk for pneumonia, we evaluated the influence of ciglitazone, a TZD, on markers of inflammation and outcome during pneumonia caused by Streptococcus pneumoniae."7.75The thiazolidinedione ciglitazone reduces bacterial outgrowth and early inflammation during Streptococcus pneumoniae pneumonia in mice. ( de Vos, AF; Florquin, S; Stegenga, ME; van der Poll, T, 2009)
" Because patients with type 2 diabetes have an increased risk for pneumonia, we evaluated the influence of ciglitazone, a TZD, on markers of inflammation and outcome during pneumonia caused by Streptococcus pneumoniae."3.75The thiazolidinedione ciglitazone reduces bacterial outgrowth and early inflammation during Streptococcus pneumoniae pneumonia in mice. ( de Vos, AF; Florquin, S; Stegenga, ME; van der Poll, T, 2009)

Research

Studies (1)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's1 (100.00)29.6817
2010's0 (0.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Stegenga, ME1
Florquin, S1
de Vos, AF1
van der Poll, T1

Other Studies

1 other study available for ciglitazone and Pneumococcal Infections

ArticleYear
The thiazolidinedione ciglitazone reduces bacterial outgrowth and early inflammation during Streptococcus pneumoniae pneumonia in mice.
    Critical care medicine, 2009, Volume: 37, Issue:2

    Topics: Animals; Colony Count, Microbial; Cytokines; Female; Hypoglycemic Agents; Inflammation; Lung; Mice;

2009