chrysophanic acid has been researched along with Cirrhosis in 4 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 0 (0.00) | 29.6817 |
| 2010's | 1 (25.00) | 24.3611 |
| 2020's | 3 (75.00) | 2.80 |
| Authors | Studies |
|---|---|
| Bai, Z; Bao, N; Cheng, X; Gao, L; Gu, M; Liao, N; Wei, Q; Zhang, H; Zhou, Y; Zhu, Y | 1 |
| Ding, Y; Dou, F; Duan, J; Wang, C; Wang, J; Wang, W; Wen, A; Xu, H; Zhao, X | 1 |
| Chen, ML; Chen, YR; Chiu, V; Hsieh, PC; Huang, CY; Huang, SJ; Kuo, CY; Liu, CT; Tsai, FM; Tzeng, IS | 1 |
| Lian, Y; Xia, X; Zhao, H; Zhu, Y | 1 |
4 other study(ies) available for chrysophanic acid and Cirrhosis
| Article | Year |
|---|---|
Chrysophanol, a main anthraquinone from Rheum palmatum L. (rhubarb), protects against renal fibrosis by suppressing NKD2/NF-κB pathway.
Topics: Adaptor Proteins, Signal Transducing; Animals; Anthraquinones; Calcium-Binding Proteins; Fibrosis; Humans; Inflammation; Kidney; Mice; Molecular Docking Simulation; NF-kappa B; Renal Insufficiency, Chronic; Rheum; Transforming Growth Factor beta1; Ureteral Obstruction | 2022 |
Chrysophanol ameliorates renal interstitial fibrosis by inhibiting the TGF-β/Smad signaling pathway.
Topics: Animals; Anthraquinones; Cell Line; Cell Survival; Disease Models, Animal; Epithelial Cells; Fibrosis; Humans; Kidney; Kidney Tubules, Proximal; Male; Mice; Mice, Inbred C57BL; Nephritis, Interstitial; Signal Transduction; Smad Proteins; Transforming Growth Factor beta | 2020 |
Chrysophanol attenuates hepatitis B virus X protein-induced hepatic stellate cell fibrosis by regulating endoplasmic reticulum stress and ferroptosis.
Topics: Animals; Anthraquinones; Cell Line; Endoplasmic Reticulum Stress; Ferroptosis; Fibrosis; Hepatic Stellate Cells; Liver Cirrhosis; Phytotherapy; Rats; Reactive Oxygen Species; Trans-Activators; Viral Regulatory and Accessory Proteins | 2020 |
The potential of chrysophanol in protecting against high fat-induced cardiac injury through Nrf2-regulated anti-inflammation, anti-oxidant and anti-fibrosis in Nrf2 knockout mice.
Topics: Animals; Anthraquinones; Anti-Inflammatory Agents; Antioxidants; Diabetic Cardiomyopathies; Diet, High-Fat; Fibrosis; Heart Injuries; Inflammation; Mice; Mice, Knockout; NF-E2-Related Factor 2; Oxidants; Oxidative Stress; Protective Agents; Signal Transduction | 2017 |