chlorine has been researched along with von Willebrand Diseases in 2 studies
chloride : A halide anion formed when chlorine picks up an electron to form an an anion.
von Willebrand Diseases: Group of hemorrhagic disorders in which the VON WILLEBRAND FACTOR is either quantitatively or qualitatively abnormal. They are usually inherited as an autosomal dominant trait though rare kindreds are autosomal recessive. Symptoms vary depending on severity and disease type but may include prolonged bleeding time, deficiency of factor VIII, and impaired platelet adhesion.
| Excerpt | Relevance | Reference |
|---|---|---|
| " A ferric chloride injury model of thrombosis was also evaluated." | 3.78 | Use of a mouse model to elucidate the phenotypic effects of the von Willebrand factor cleavage mutants, Y1605A/M1606A and R1597W. ( Bryant, A; Golder, M; Haberichter, S; Hegadorn, C; Lillicrap, D; Pruss, CM, 2012) |
| "Gene therapy for severe von Willebrand disease (vWD) seems an interesting treatment alternative with long-term therapeutic potential." | 1.35 | Restoration of plasma von Willebrand factor deficiency is sufficient to correct thrombus formation after gene therapy for severe von Willebrand disease. ( Chuah, MK; De Meyer, SF; Deckmyn, H; Lenting, PJ; Pareyn, I; Petrus, I; VandenDriessche, T; Vandeputte, N; Vanhoorelbeke, K, 2008) |
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 1 (50.00) | 29.6817 |
| 2010's | 1 (50.00) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| De Meyer, SF | 1 |
| Vandeputte, N | 1 |
| Pareyn, I | 1 |
| Petrus, I | 1 |
| Lenting, PJ | 1 |
| Chuah, MK | 1 |
| VandenDriessche, T | 1 |
| Deckmyn, H | 1 |
| Vanhoorelbeke, K | 1 |
| Pruss, CM | 1 |
| Golder, M | 1 |
| Bryant, A | 1 |
| Hegadorn, C | 1 |
| Haberichter, S | 1 |
| Lillicrap, D | 1 |
2 other studies available for chlorine and von Willebrand Diseases
| Article | Year |
|---|---|
Restoration of plasma von Willebrand factor deficiency is sufficient to correct thrombus formation after gene therapy for severe von Willebrand disease.
Topics: alpha 1-Antitrypsin; Animals; Bleeding Time; Chlorides; Cytomegalovirus; Disease Models, Animal; Fac | 2008 |
Use of a mouse model to elucidate the phenotypic effects of the von Willebrand factor cleavage mutants, Y1605A/M1606A and R1597W.
Topics: ADAMTS13 Protein; Animals; Blood Coagulation; Blotting, Western; Chlorides; Disease Models, Animal; | 2012 |