Page last updated: 2024-10-17

chlorine and Ciliary Motility Disorders

chlorine has been researched along with Ciliary Motility Disorders in 2 studies

chloride : A halide anion formed when chlorine picks up an electron to form an an anion.

Ciliary Motility Disorders: Conditions caused by abnormal CILIA movement in the body, usually causing KARTAGENER SYNDROME, chronic respiratory disorders, chronic SINUSITIS, and chronic OTITIS. Abnormal ciliary beating is likely due to defects in any of the 200 plus ciliary proteins, such as missing motor enzyme DYNEIN arms.

Research Excerpts

ExcerptRelevanceReference
"Activation of the inositol cycle by a factor capable of by-passing the normal controls on exocrine secretion by an interaction with a coupling protein could produce effects similar to a calcium ionophore or the ciliary dyskinesia factor."7.67The end organ defect in cystic fibrosis; a hypothesis: disinhibited inositol cycle activation? ( van Woerkom, AE, 1987)
"Activation of the inositol cycle by a factor capable of by-passing the normal controls on exocrine secretion by an interaction with a coupling protein could produce effects similar to a calcium ionophore or the ciliary dyskinesia factor."3.67The end organ defect in cystic fibrosis; a hypothesis: disinhibited inositol cycle activation? ( van Woerkom, AE, 1987)

Research

Studies (2)

TimeframeStudies, this research(%)All Research%
pre-19901 (50.00)18.7374
1990's0 (0.00)18.2507
2000's0 (0.00)29.6817
2010's1 (50.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Shim, JW1
Territo, PR1
Simpson, S1
Watson, JC1
Jiang, L1
Riley, AA1
McCarthy, B1
Persohn, S1
Fulkerson, D1
Blazer-Yost, BL1
van Woerkom, AE1

Other Studies

2 other studies available for chlorine and Ciliary Motility Disorders

ArticleYear
Hydrocephalus in a rat model of Meckel Gruber syndrome with a TMEM67 mutation.
    Scientific reports, 2019, 01-31, Volume: 9, Issue:1

    Topics: Animals; Brain; Chlorides; Choroid Plexus; Ciliary Motility Disorders; Encephalocele; Female; Hydroc

2019
The end organ defect in cystic fibrosis; a hypothesis: disinhibited inositol cycle activation?
    Medical hypotheses, 1987, Volume: 23, Issue:4

    Topics: Blood Proteins; Calcium; Cell Membrane Permeability; Chlorides; Ciliary Motility Disorders; Cystic F

1987