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chlorine and Absence Status

chlorine has been researched along with Absence Status in 13 studies

chloride : A halide anion formed when chlorine picks up an electron to form an an anion.

Research Excerpts

ExcerptRelevanceReference
" In this study, we aimed to investigate the long-term expression profiles of NKCC1 and KCC2 in CA1 region in the mice model of lithium-pilocarpine induced status epilepticus (PISE) and their relationship with epileptogenesis."7.74Long-term expressional changes of Na+ -K+ -Cl- co-transporter 1 (NKCC1) and K+ -Cl- co-transporter 2 (KCC2) in CA1 region of hippocampus following lithium-pilocarpine induced status epilepticus (PISE). ( Chen, S; Chen, Z; Li, X; Zhou, J; Zhou, L; Zhu, F, 2008)
"Functional modulation of gamma-aminobutyric acid(A) (GABA(A)) receptors by Zn(2+), pentobarbital, neuroactive steroid alphaxalone, and flunitrazepam was studied in the cerebral cortex and cerebellum of rats undergoing status epilepticus induced by pilocarpine."7.70Zinc inhibition of gamma-aminobutyric acid(A) receptor function is decreased in the cerebral cortex during pilocarpine-induced status epilepticus. ( Banerjee, PK; Olsen, RW; Snead, OC, 1999)
" after they had been assigned to one of 8 groups in a 3-way analysis of variance design that involved (1) induction of limbic seizures by a systemic injection of lithium/pilocarpine, (2) physical restraint, and (3) administration of acepromazine."7.68Extreme hypothermia induced by a synergism of acute limbic seizures, physical restraint, and acepromazine: implications for survival following brain injury. ( Bureau, YR; Persinger, MA, 1993)
"Lithium is known to potentiate the ability of pilocarpine to induce status epilepticus in rats."7.68Ontogenic study of lithium-pilocarpine-induced status epilepticus in rats. ( Baram, TZ; Hirsch, E; Snead, OC, 1992)
"The specific binding of [3H]hemicholinium-3 ([3H]HCh-3) and high-affinity [3H]choline uptake were measured in rats with status epilepticus induced by lithium and pilocarpine."7.68[3H]hemicholinium-3 binding in rats with status epilepticus induced by lithium chloride and pilocarpine. ( Coyle, JT; Saltarelli, MD; Yamada, K, 1991)
" These results demonstrated that both acute and chronic administration of lithium enhance cholinergic function in vivo."5.27Status epilepticus is produced by administration of cholinergic agonists to lithium-treated rats: comparison with kainic acid. ( Jope, RS; Morrisett, RA; Snead, OC, 1987)
" Using the kainic acid model of status epilepticus, we have studied the effects of repetitive neonatal episodes of status epilepticus on the expression of cation chloride cotransporter KCC2 in the neonatal hippocampus."4.84Developmental patterns in the regulation of chloride homeostasis and GABA(A) receptor signaling by seizures. ( Galanopoulou, AS, 2007)
" In this study, we aimed to investigate the long-term expression profiles of NKCC1 and KCC2 in CA1 region in the mice model of lithium-pilocarpine induced status epilepticus (PISE) and their relationship with epileptogenesis."3.74Long-term expressional changes of Na+ -K+ -Cl- co-transporter 1 (NKCC1) and K+ -Cl- co-transporter 2 (KCC2) in CA1 region of hippocampus following lithium-pilocarpine induced status epilepticus (PISE). ( Chen, S; Chen, Z; Li, X; Zhou, J; Zhou, L; Zhu, F, 2008)
"Functional modulation of gamma-aminobutyric acid(A) (GABA(A)) receptors by Zn(2+), pentobarbital, neuroactive steroid alphaxalone, and flunitrazepam was studied in the cerebral cortex and cerebellum of rats undergoing status epilepticus induced by pilocarpine."3.70Zinc inhibition of gamma-aminobutyric acid(A) receptor function is decreased in the cerebral cortex during pilocarpine-induced status epilepticus. ( Banerjee, PK; Olsen, RW; Snead, OC, 1999)
" after they had been assigned to one of 8 groups in a 3-way analysis of variance design that involved (1) induction of limbic seizures by a systemic injection of lithium/pilocarpine, (2) physical restraint, and (3) administration of acepromazine."3.68Extreme hypothermia induced by a synergism of acute limbic seizures, physical restraint, and acepromazine: implications for survival following brain injury. ( Bureau, YR; Persinger, MA, 1993)
"Lithium is known to potentiate the ability of pilocarpine to induce status epilepticus in rats."3.68Ontogenic study of lithium-pilocarpine-induced status epilepticus in rats. ( Baram, TZ; Hirsch, E; Snead, OC, 1992)
"The specific binding of [3H]hemicholinium-3 ([3H]HCh-3) and high-affinity [3H]choline uptake were measured in rats with status epilepticus induced by lithium and pilocarpine."3.68[3H]hemicholinium-3 binding in rats with status epilepticus induced by lithium chloride and pilocarpine. ( Coyle, JT; Saltarelli, MD; Yamada, K, 1991)
"Occurrence of spontaneous epileptic seizures was analyzed at 2 months post-SE using continuous video-EEG monitoring."1.35Manganese enhanced MRI detects mossy fiber sprouting rather than neurodegeneration, gliosis or seizure-activity in the epileptic rat hippocampus. ( Einula, C; Gröhn, OH; Immonen, RJ; Kharatishvili, I; Pitkänen, A; Sierra, A, 2008)
" These results demonstrated that both acute and chronic administration of lithium enhance cholinergic function in vivo."1.27Status epilepticus is produced by administration of cholinergic agonists to lithium-treated rats: comparison with kainic acid. ( Jope, RS; Morrisett, RA; Snead, OC, 1987)

Research

Studies (13)

TimeframeStudies, this research(%)All Research%
pre-19902 (15.38)18.7374
1990's5 (38.46)18.2507
2000's3 (23.08)29.6817
2010's3 (23.08)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Puskarjov, M1
Ahmad, F1
Khirug, S1
Sivakumaran, S1
Kaila, K1
Blaesse, P1
Silayeva, L1
Deeb, TZ1
Hines, RM1
Kelley, MR1
Munoz, MB1
Lee, HH1
Brandon, NJ1
Dunlop, J1
Maguire, J1
Davies, PA1
Moss, SJ1
Li, X1
Zhou, J1
Chen, Z1
Chen, S1
Zhu, F1
Zhou, L1
Barmashenko, G1
Hefft, S1
Aertsen, A1
Kirschstein, T1
Köhling, R1
Galanopoulou, AS1
Immonen, RJ1
Kharatishvili, I1
Sierra, A1
Einula, C1
Pitkänen, A1
Gröhn, OH1
Brivet, F1
Bernardin, M1
Cherin, P1
Chalas, J1
Galanaud, P1
Dormont, J1
Bureau, YR1
Persinger, MA1
Banerjee, PK1
Olsen, RW1
Snead, OC3
Hirsch, E1
Baram, TZ1
Yamada, K1
Saltarelli, MD1
Coyle, JT1
Morrisett, RA1
Jope, RS1
Lux, HD1
Heinemann, U1
Dietzel, I1

Reviews

2 reviews available for chlorine and Absence Status

ArticleYear
Developmental patterns in the regulation of chloride homeostasis and GABA(A) receptor signaling by seizures.
    Epilepsia, 2007, Volume: 48 Suppl 5

    Topics: Animals; Animals, Newborn; Brain; Chlorides; Disease Models, Animal; Fluoresceins; Hippocampus; Home

2007
Ionic changes and alterations in the size of the extracellular space during epileptic activity.
    Advances in neurology, 1986, Volume: 44

    Topics: Action Potentials; Aluminum Oxide; Animals; Chlorides; Epilepsy; Extracellular Space; Forecasting; H

1986

Other Studies

11 other studies available for chlorine and Absence Status

ArticleYear
BDNF is required for seizure-induced but not developmental up-regulation of KCC2 in the neonatal hippocampus.
    Neuropharmacology, 2015, Volume: 88

    Topics: Animals; Animals, Newborn; Blotting, Western; Brain-Derived Neurotrophic Factor; Calpain; Chlorides;

2015
KCC2 activity is critical in limiting the onset and severity of status epilepticus.
    Proceedings of the National Academy of Sciences of the United States of America, 2015, Mar-17, Volume: 112, Issue:11

    Topics: Animals; Chlorides; Endocytosis; gamma-Aminobutyric Acid; Gene Knock-In Techniques; Glutamates; K Cl

2015
Long-term expressional changes of Na+ -K+ -Cl- co-transporter 1 (NKCC1) and K+ -Cl- co-transporter 2 (KCC2) in CA1 region of hippocampus following lithium-pilocarpine induced status epilepticus (PISE).
    Brain research, 2008, Jul-24, Volume: 1221

    Topics: Animals; Chloride Channels; Chlorides; Disease Models, Animal; Epilepsy; Gene Expression Regulation;

2008
Positive shifts of the GABAA receptor reversal potential due to altered chloride homeostasis is widespread after status epilepticus.
    Epilepsia, 2011, Volume: 52, Issue:9

    Topics: 2-Amino-5-phosphonovalerate; 6-Cyano-7-nitroquinoxaline-2,3-dione; Analysis of Variance; Animals; Bi

2011
Manganese enhanced MRI detects mossy fiber sprouting rather than neurodegeneration, gliosis or seizure-activity in the epileptic rat hippocampus.
    NeuroImage, 2008, May-01, Volume: 40, Issue:4

    Topics: Algorithms; Animals; Blood-Brain Barrier; Chlorides; Chronic Disease; Contrast Media; Data Interpret

2008
Hyperchloremic acidosis during grand mal seizure lactic acidosis.
    Intensive care medicine, 1994, Volume: 20, Issue:1

    Topics: Acid-Base Equilibrium; Acidosis, Lactic; Acute Disease; Bicarbonates; Blood Gas Analysis; Chlorides;

1994
Extreme hypothermia induced by a synergism of acute limbic seizures, physical restraint, and acepromazine: implications for survival following brain injury.
    Psychological reports, 1993, Volume: 72, Issue:1

    Topics: Acepromazine; Animals; Body Temperature Regulation; Brain Injuries; Chlorides; Hypothermia; Limbic S

1993
Zinc inhibition of gamma-aminobutyric acid(A) receptor function is decreased in the cerebral cortex during pilocarpine-induced status epilepticus.
    The Journal of pharmacology and experimental therapeutics, 1999, Volume: 291, Issue:1

    Topics: Animals; Biological Transport; Cerebellum; Cerebral Cortex; Chlorides; GABA Agonists; GABA-A Recepto

1999
Ontogenic study of lithium-pilocarpine-induced status epilepticus in rats.
    Brain research, 1992, Jun-26, Volume: 583, Issue:1-2

    Topics: Aging; Animals; Animals, Newborn; Cerebral Cortex; Chlorides; Drug Administration Schedule; Electroe

1992
[3H]hemicholinium-3 binding in rats with status epilepticus induced by lithium chloride and pilocarpine.
    European journal of pharmacology, 1991, Apr-03, Volume: 195, Issue:3

    Topics: Acetylcholine; Animals; Cerebral Cortex; Chlorides; Choline; Hemicholinium 3; Hippocampus; Kinetics;

1991
Status epilepticus is produced by administration of cholinergic agonists to lithium-treated rats: comparison with kainic acid.
    Experimental neurology, 1987, Volume: 98, Issue:3

    Topics: Animals; Arecoline; Chlorides; Dose-Response Relationship, Drug; Drug Synergism; Electroencephalogra

1987