chlorine has been researched along with Absence Status in 13 studies
chloride : A halide anion formed when chlorine picks up an electron to form an an anion.
| Excerpt | Relevance | Reference |
|---|---|---|
| " In this study, we aimed to investigate the long-term expression profiles of NKCC1 and KCC2 in CA1 region in the mice model of lithium-pilocarpine induced status epilepticus (PISE) and their relationship with epileptogenesis." | 7.74 | Long-term expressional changes of Na+ -K+ -Cl- co-transporter 1 (NKCC1) and K+ -Cl- co-transporter 2 (KCC2) in CA1 region of hippocampus following lithium-pilocarpine induced status epilepticus (PISE). ( Chen, S; Chen, Z; Li, X; Zhou, J; Zhou, L; Zhu, F, 2008) |
| "Functional modulation of gamma-aminobutyric acid(A) (GABA(A)) receptors by Zn(2+), pentobarbital, neuroactive steroid alphaxalone, and flunitrazepam was studied in the cerebral cortex and cerebellum of rats undergoing status epilepticus induced by pilocarpine." | 7.70 | Zinc inhibition of gamma-aminobutyric acid(A) receptor function is decreased in the cerebral cortex during pilocarpine-induced status epilepticus. ( Banerjee, PK; Olsen, RW; Snead, OC, 1999) |
| " after they had been assigned to one of 8 groups in a 3-way analysis of variance design that involved (1) induction of limbic seizures by a systemic injection of lithium/pilocarpine, (2) physical restraint, and (3) administration of acepromazine." | 7.68 | Extreme hypothermia induced by a synergism of acute limbic seizures, physical restraint, and acepromazine: implications for survival following brain injury. ( Bureau, YR; Persinger, MA, 1993) |
| "Lithium is known to potentiate the ability of pilocarpine to induce status epilepticus in rats." | 7.68 | Ontogenic study of lithium-pilocarpine-induced status epilepticus in rats. ( Baram, TZ; Hirsch, E; Snead, OC, 1992) |
| "The specific binding of [3H]hemicholinium-3 ([3H]HCh-3) and high-affinity [3H]choline uptake were measured in rats with status epilepticus induced by lithium and pilocarpine." | 7.68 | [3H]hemicholinium-3 binding in rats with status epilepticus induced by lithium chloride and pilocarpine. ( Coyle, JT; Saltarelli, MD; Yamada, K, 1991) |
| " These results demonstrated that both acute and chronic administration of lithium enhance cholinergic function in vivo." | 5.27 | Status epilepticus is produced by administration of cholinergic agonists to lithium-treated rats: comparison with kainic acid. ( Jope, RS; Morrisett, RA; Snead, OC, 1987) |
| " Using the kainic acid model of status epilepticus, we have studied the effects of repetitive neonatal episodes of status epilepticus on the expression of cation chloride cotransporter KCC2 in the neonatal hippocampus." | 4.84 | Developmental patterns in the regulation of chloride homeostasis and GABA(A) receptor signaling by seizures. ( Galanopoulou, AS, 2007) |
| " In this study, we aimed to investigate the long-term expression profiles of NKCC1 and KCC2 in CA1 region in the mice model of lithium-pilocarpine induced status epilepticus (PISE) and their relationship with epileptogenesis." | 3.74 | Long-term expressional changes of Na+ -K+ -Cl- co-transporter 1 (NKCC1) and K+ -Cl- co-transporter 2 (KCC2) in CA1 region of hippocampus following lithium-pilocarpine induced status epilepticus (PISE). ( Chen, S; Chen, Z; Li, X; Zhou, J; Zhou, L; Zhu, F, 2008) |
| "Functional modulation of gamma-aminobutyric acid(A) (GABA(A)) receptors by Zn(2+), pentobarbital, neuroactive steroid alphaxalone, and flunitrazepam was studied in the cerebral cortex and cerebellum of rats undergoing status epilepticus induced by pilocarpine." | 3.70 | Zinc inhibition of gamma-aminobutyric acid(A) receptor function is decreased in the cerebral cortex during pilocarpine-induced status epilepticus. ( Banerjee, PK; Olsen, RW; Snead, OC, 1999) |
| " after they had been assigned to one of 8 groups in a 3-way analysis of variance design that involved (1) induction of limbic seizures by a systemic injection of lithium/pilocarpine, (2) physical restraint, and (3) administration of acepromazine." | 3.68 | Extreme hypothermia induced by a synergism of acute limbic seizures, physical restraint, and acepromazine: implications for survival following brain injury. ( Bureau, YR; Persinger, MA, 1993) |
| "Lithium is known to potentiate the ability of pilocarpine to induce status epilepticus in rats." | 3.68 | Ontogenic study of lithium-pilocarpine-induced status epilepticus in rats. ( Baram, TZ; Hirsch, E; Snead, OC, 1992) |
| "The specific binding of [3H]hemicholinium-3 ([3H]HCh-3) and high-affinity [3H]choline uptake were measured in rats with status epilepticus induced by lithium and pilocarpine." | 3.68 | [3H]hemicholinium-3 binding in rats with status epilepticus induced by lithium chloride and pilocarpine. ( Coyle, JT; Saltarelli, MD; Yamada, K, 1991) |
| "Occurrence of spontaneous epileptic seizures was analyzed at 2 months post-SE using continuous video-EEG monitoring." | 1.35 | Manganese enhanced MRI detects mossy fiber sprouting rather than neurodegeneration, gliosis or seizure-activity in the epileptic rat hippocampus. ( Einula, C; Gröhn, OH; Immonen, RJ; Kharatishvili, I; Pitkänen, A; Sierra, A, 2008) |
| " These results demonstrated that both acute and chronic administration of lithium enhance cholinergic function in vivo." | 1.27 | Status epilepticus is produced by administration of cholinergic agonists to lithium-treated rats: comparison with kainic acid. ( Jope, RS; Morrisett, RA; Snead, OC, 1987) |
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 2 (15.38) | 18.7374 |
| 1990's | 5 (38.46) | 18.2507 |
| 2000's | 3 (23.08) | 29.6817 |
| 2010's | 3 (23.08) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Puskarjov, M | 1 |
| Ahmad, F | 1 |
| Khirug, S | 1 |
| Sivakumaran, S | 1 |
| Kaila, K | 1 |
| Blaesse, P | 1 |
| Silayeva, L | 1 |
| Deeb, TZ | 1 |
| Hines, RM | 1 |
| Kelley, MR | 1 |
| Munoz, MB | 1 |
| Lee, HH | 1 |
| Brandon, NJ | 1 |
| Dunlop, J | 1 |
| Maguire, J | 1 |
| Davies, PA | 1 |
| Moss, SJ | 1 |
| Li, X | 1 |
| Zhou, J | 1 |
| Chen, Z | 1 |
| Chen, S | 1 |
| Zhu, F | 1 |
| Zhou, L | 1 |
| Barmashenko, G | 1 |
| Hefft, S | 1 |
| Aertsen, A | 1 |
| Kirschstein, T | 1 |
| Köhling, R | 1 |
| Galanopoulou, AS | 1 |
| Immonen, RJ | 1 |
| Kharatishvili, I | 1 |
| Sierra, A | 1 |
| Einula, C | 1 |
| Pitkänen, A | 1 |
| Gröhn, OH | 1 |
| Brivet, F | 1 |
| Bernardin, M | 1 |
| Cherin, P | 1 |
| Chalas, J | 1 |
| Galanaud, P | 1 |
| Dormont, J | 1 |
| Bureau, YR | 1 |
| Persinger, MA | 1 |
| Banerjee, PK | 1 |
| Olsen, RW | 1 |
| Snead, OC | 3 |
| Hirsch, E | 1 |
| Baram, TZ | 1 |
| Yamada, K | 1 |
| Saltarelli, MD | 1 |
| Coyle, JT | 1 |
| Morrisett, RA | 1 |
| Jope, RS | 1 |
| Lux, HD | 1 |
| Heinemann, U | 1 |
| Dietzel, I | 1 |
2 reviews available for chlorine and Absence Status
| Article | Year |
|---|---|
Developmental patterns in the regulation of chloride homeostasis and GABA(A) receptor signaling by seizures.
Topics: Animals; Animals, Newborn; Brain; Chlorides; Disease Models, Animal; Fluoresceins; Hippocampus; Home | 2007 |
Ionic changes and alterations in the size of the extracellular space during epileptic activity.
Topics: Action Potentials; Aluminum Oxide; Animals; Chlorides; Epilepsy; Extracellular Space; Forecasting; H | 1986 |
11 other studies available for chlorine and Absence Status
| Article | Year |
|---|---|
BDNF is required for seizure-induced but not developmental up-regulation of KCC2 in the neonatal hippocampus.
Topics: Animals; Animals, Newborn; Blotting, Western; Brain-Derived Neurotrophic Factor; Calpain; Chlorides; | 2015 |
KCC2 activity is critical in limiting the onset and severity of status epilepticus.
Topics: Animals; Chlorides; Endocytosis; gamma-Aminobutyric Acid; Gene Knock-In Techniques; Glutamates; K Cl | 2015 |
Long-term expressional changes of Na+ -K+ -Cl- co-transporter 1 (NKCC1) and K+ -Cl- co-transporter 2 (KCC2) in CA1 region of hippocampus following lithium-pilocarpine induced status epilepticus (PISE).
Topics: Animals; Chloride Channels; Chlorides; Disease Models, Animal; Epilepsy; Gene Expression Regulation; | 2008 |
Positive shifts of the GABAA receptor reversal potential due to altered chloride homeostasis is widespread after status epilepticus.
Topics: 2-Amino-5-phosphonovalerate; 6-Cyano-7-nitroquinoxaline-2,3-dione; Analysis of Variance; Animals; Bi | 2011 |
Manganese enhanced MRI detects mossy fiber sprouting rather than neurodegeneration, gliosis or seizure-activity in the epileptic rat hippocampus.
Topics: Algorithms; Animals; Blood-Brain Barrier; Chlorides; Chronic Disease; Contrast Media; Data Interpret | 2008 |
Hyperchloremic acidosis during grand mal seizure lactic acidosis.
Topics: Acid-Base Equilibrium; Acidosis, Lactic; Acute Disease; Bicarbonates; Blood Gas Analysis; Chlorides; | 1994 |
Extreme hypothermia induced by a synergism of acute limbic seizures, physical restraint, and acepromazine: implications for survival following brain injury.
Topics: Acepromazine; Animals; Body Temperature Regulation; Brain Injuries; Chlorides; Hypothermia; Limbic S | 1993 |
Zinc inhibition of gamma-aminobutyric acid(A) receptor function is decreased in the cerebral cortex during pilocarpine-induced status epilepticus.
Topics: Animals; Biological Transport; Cerebellum; Cerebral Cortex; Chlorides; GABA Agonists; GABA-A Recepto | 1999 |
Ontogenic study of lithium-pilocarpine-induced status epilepticus in rats.
Topics: Aging; Animals; Animals, Newborn; Cerebral Cortex; Chlorides; Drug Administration Schedule; Electroe | 1992 |
[3H]hemicholinium-3 binding in rats with status epilepticus induced by lithium chloride and pilocarpine.
Topics: Acetylcholine; Animals; Cerebral Cortex; Chlorides; Choline; Hemicholinium 3; Hippocampus; Kinetics; | 1991 |
Status epilepticus is produced by administration of cholinergic agonists to lithium-treated rats: comparison with kainic acid.
Topics: Animals; Arecoline; Chlorides; Dose-Response Relationship, Drug; Drug Synergism; Electroencephalogra | 1987 |