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chloral hydrate and Epilepsy, Temporal Lobe

chloral hydrate has been researched along with Epilepsy, Temporal Lobe in 2 studies

Epilepsy, Temporal Lobe: A localization-related (focal) form of epilepsy characterized by recurrent seizures that arise from foci within the TEMPORAL LOBE, most commonly from its mesial aspect. A wide variety of psychic phenomena may be associated, including illusions, hallucinations, dyscognitive states, and affective experiences. The majority of complex partial seizures (see EPILEPSY, COMPLEX PARTIAL) originate from the temporal lobes. Temporal lobe seizures may be classified by etiology as cryptogenic, familial, or symptomatic. (From Adams et al., Principles of Neurology, 6th ed, p321).

Research Excerpts

ExcerptRelevanceReference
" Finally, we examined the effects of different types of anesthesia (chloral hydrate, isoflurane) on kainate-induced status epilepticus (SE) and epileptogenesis."3.83Significant effects of sex, strain, and anesthesia in the intrahippocampal kainate mouse model of mesial temporal lobe epilepsy. ( Brandt, C; Löscher, W; Töllner, K; Twele, F, 2016)

Research

Studies (2)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's1 (50.00)18.2507
2000's0 (0.00)29.6817
2010's1 (50.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Twele, F1
Töllner, K1
Brandt, C1
Löscher, W1
Magloczky, Z1
Freund, TF1

Other Studies

2 other studies available for chloral hydrate and Epilepsy, Temporal Lobe

ArticleYear
Significant effects of sex, strain, and anesthesia in the intrahippocampal kainate mouse model of mesial temporal lobe epilepsy.
    Epilepsy & behavior : E&B, 2016, Volume: 55

    Topics: Anesthesia; Anesthetics; Animals; Chloral Hydrate; Electroencephalography; Epilepsy, Temporal Lobe;

2016
Selective neuronal death in the contralateral hippocampus following unilateral kainate injections into the CA3 subfield.
    Neuroscience, 1993, Volume: 56, Issue:2

    Topics: Anesthetics; Animals; Biomarkers; Calbindin 2; Cell Death; Chloral Hydrate; Disease Models, Animal;

1993