chenodeoxycholic acid has been researched along with Kidney Diseases in 3 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 1 (33.33) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 0 (0.00) | 29.6817 |
| 2010's | 2 (66.67) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Gai, Z; Gui, T; Hiller, C; Kullak-Ublick, GA | 1 |
| Kratzer, A; Levi, M; Lewis, LB; Miyazaki, M; Miyazaki-Anzai, S; Ting, TC | 1 |
| Heymans, HS; Schutgens, RB; Wanders, RJ | 1 |
3 other study(ies) available for chenodeoxycholic acid and Kidney Diseases
| Article | Year |
|---|---|
Farnesoid X Receptor Protects against Kidney Injury in Uninephrectomized Obese Mice.
Topics: Aldehydes; Animals; Bile Acids and Salts; CCAAT-Enhancer-Binding Proteins; Chenodeoxycholic Acid; Diet, High-Fat; Endoplasmic Reticulum Chaperone BiP; Endoplasmic Reticulum Stress; Fatty Acids, Nonesterified; Female; Glutathione; Heat-Shock Proteins; Humans; Kidney; Kidney Diseases; Kidney Tubules; Lipid Metabolism; Lipid Peroxidation; Lipids; Male; Mice; Mice, Inbred C57BL; Mice, Obese; Middle Aged; Nephrectomy; Non-alcoholic Fatty Liver Disease; Obesity; Oxidative Stress; Reactive Oxygen Species; Receptors, Cytoplasmic and Nuclear | 2016 |
Farnesoid X receptor activation prevents the development of vascular calcification in ApoE-/- mice with chronic kidney disease.
Topics: Animals; Aorta; Apolipoproteins E; Calcinosis; Cattle; Cell Differentiation; Cells, Cultured; Chenodeoxycholic Acid; Chronic Disease; Disease Models, Animal; Kidney Diseases; Male; Mice; Mice, Knockout; Osteogenesis; Receptors, Cytoplasmic and Nuclear; Signal Transduction; Triglycerides; Vascular Diseases | 2010 |
Deficient cholesterol side chain oxidation in patients without peroxisomes (Zellweger syndrome): evidence for the involvement of peroxisomes in bile acid synthesis in man.
Topics: Bile Acids and Salts; Brain Diseases; Chenodeoxycholic Acid; Cholesterol; Cholic Acid; Cholic Acids; Humans; Kidney Diseases; Liver; Liver Diseases; Microbodies; Oxidation-Reduction; Protein Processing, Post-Translational; Syndrome | 1987 |