Page last updated: 2024-10-21

cgp 52411 and Brain Edema

cgp 52411 has been researched along with Brain Edema in 2 studies

4,5-dianilinophthalimide: structure given in first source
4,5-dianilinophthalimide : Phthalimide substituted at the 4- and 5-positions by anilino groups.

Brain Edema: Increased intracellular or extracellular fluid in brain tissue. Cytotoxic brain edema (swelling due to increased intracellular fluid) is indicative of a disturbance in cell metabolism, and is commonly associated with hypoxic or ischemic injuries (see HYPOXIA, BRAIN). An increase in extracellular fluid may be caused by increased brain capillary permeability (vasogenic edema), an osmotic gradient, local blockages in interstitial fluid pathways, or by obstruction of CSF flow (e.g., obstructive HYDROCEPHALUS). (From Childs Nerv Syst 1992 Sep; 8(6):301-6)

Research

Studies (2)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's1 (50.00)18.2507
2000's0 (0.00)29.6817
2010's1 (50.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Cipolla, MJ1
Huang, Q1
Sweet, JG1
Justicia, C1
Planas, AM1

Other Studies

2 other studies available for cgp 52411 and Brain Edema

ArticleYear
Inhibition of protein kinase Cβ reverses increased blood-brain barrier permeability during hyperglycemic stroke and prevents edema formation in vivo.
    Stroke, 2011, Volume: 42, Issue:11

    Topics: Animals; Blood-Brain Barrier; Brain Edema; Capillary Permeability; Hyperglycemia; Male; Phthalimides

2011
Transforming growth factor-alpha acting at the epidermal growth factor receptor reduces infarct volume after permanent middle cerebral artery occlusion in rats.
    Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism, 1999, Volume: 19, Issue:2

    Topics: Animals; Arterial Occlusive Diseases; Brain Edema; Cerebral Arteries; Cerebral Infarction; Dose-Resp

1999