celecoxib has been researched along with Primary Ovarian Insufficiency in 1 studies
Primary Ovarian Insufficiency: Cessation of ovarian function after MENARCHE but before the age of 40, without or with OVARIAN FOLLICLE depletion. It is characterized by the presence of OLIGOMENORRHEA or AMENORRHEA, elevated GONADOTROPINS, and low ESTRADIOL levels. It is a state of female HYPERGONADOTROPIC HYPOGONADISM. Etiologies include genetic defects, autoimmune processes, chemotherapy, radiation, and infections. The most commonly known genetic cause is the expansion of a CGG repeat to 55 to 199 copies in the 5' untranslated region in the X-linked FMR1 gene.
Excerpt | Relevance | Reference |
---|---|---|
"Menopause is the permanent cessation of menstruation that results from depletion of ovarian germ cells and follicles." | 1.39 | Cyclooxygenase-1 inhibition prolongs postnatal ovarian follicle lifespan in mice. ( Cai, KQ; Smedberg, J; Smith, ER; Xu, XX; Yang, WL; Yeasky, T, 2013) |
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 0 (0.00) | 18.2507 |
2000's | 0 (0.00) | 29.6817 |
2010's | 1 (100.00) | 24.3611 |
2020's | 0 (0.00) | 2.80 |
Authors | Studies |
---|---|
Smith, ER | 1 |
Yang, WL | 1 |
Yeasky, T | 1 |
Smedberg, J | 1 |
Cai, KQ | 1 |
Xu, XX | 1 |
1 other study available for celecoxib and Primary Ovarian Insufficiency
Article | Year |
---|---|
Cyclooxygenase-1 inhibition prolongs postnatal ovarian follicle lifespan in mice.
Topics: Aging; Animals; Celecoxib; Crosses, Genetic; Cyclooxygenase 1; Cyclooxygenase 2; Cyclooxygenase 2 In | 2013 |