celecoxib has been researched along with Infarction, Middle Cerebral Artery in 2 studies
Infarction, Middle Cerebral Artery: NECROSIS occurring in the MIDDLE CEREBRAL ARTERY distribution system which brings blood to the entire lateral aspects of each CEREBRAL HEMISPHERE. Clinical signs include impaired cognition; APHASIA; AGRAPHIA; weak and numbness in the face and arms, contralaterally or bilaterally depending on the infarction.
Excerpt | Relevance | Reference |
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"Celecoxib treatment decreased polyubiquitinated protein load and ER stress marker expression such as glucose-related protein 78 (GRP78), C/EBP (CCAAT/enhancer-binding protein) homologous protein (CHOP), and caspase 12 after 48 h of reperfusion." | 1.62 | Celecoxib-Dependent Neuroprotection in a Rat Model of Transient Middle Cerebral Artery Occlusion (tMCAO) Involves Modifications in Unfolded Protein Response (UPR) and Proteasome. ( Anuncibay-Soto, B; Fernández-López, A; Font-Belmonte, E; González-Rodríguez, P; Pérez-Rodríguez, D; Santos-Galdiano, M; Ugidos, IF, 2021) |
"Celecoxib treatment improved the neurologic deficit, reduced the infarct volume by 50% after 48 hours of reperfusion, and resulted in a reduced percentage of SNL areas and microglia and astroglia reactivity after 48 hours of reperfusion." | 1.48 | Celecoxib Treatment Improves Neurologic Deficit and Reduces Selective Neuronal Loss and Glial Response in Rats after Transient Middle Cerebral Artery Occlusion. ( Anuncibay-Soto, B; Fernández-López, A; Font-Belmonte, E; Pérez-García, CC; Pérez-Rodríguez, D; Santos-Galdiano, M; Ugidos, IF, 2018) |
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 0 (0.00) | 18.2507 |
2000's | 0 (0.00) | 29.6817 |
2010's | 1 (50.00) | 24.3611 |
2020's | 1 (50.00) | 2.80 |
Authors | Studies |
---|---|
Santos-Galdiano, M | 2 |
González-Rodríguez, P | 1 |
Font-Belmonte, E | 2 |
Ugidos, IF | 2 |
Anuncibay-Soto, B | 2 |
Pérez-Rodríguez, D | 2 |
Fernández-López, A | 2 |
Pérez-García, CC | 1 |
2 other studies available for celecoxib and Infarction, Middle Cerebral Artery
Article | Year |
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Celecoxib-Dependent Neuroprotection in a Rat Model of Transient Middle Cerebral Artery Occlusion (tMCAO) Involves Modifications in Unfolded Protein Response (UPR) and Proteasome.
Topics: Animals; Apoptosis; Autophagy; Biomarkers; Brain Ischemia; Celecoxib; Disease Models, Animal; eIF-2 | 2021 |
Celecoxib Treatment Improves Neurologic Deficit and Reduces Selective Neuronal Loss and Glial Response in Rats after Transient Middle Cerebral Artery Occlusion.
Topics: Animals; Astrocytes; Celecoxib; Disease Models, Animal; Infarction, Middle Cerebral Artery; Male; Ne | 2018 |