celecoxib has been researched along with Carcinoma, Pancreatic Ductal in 7 studies
Carcinoma, Pancreatic Ductal: Carcinoma that arises from the PANCREATIC DUCTS. It accounts for the majority of cancers derived from the PANCREAS.
Excerpt | Relevance | Reference |
---|---|---|
"Capecitabine was administered at a dose of 1,000 mg/m(2) b." | 6.74 | Capecitabine and celecoxib as second-line treatment of advanced pancreatic and biliary tract cancers. ( Bria, E; Carlini, P; Carpanese, L; Cognetti, F; De Marco, S; Gelibter, A; Milella, M; Nuzzo, C; Pino, MS; Ruggeri, EM; Sperduti, I, 2009) |
"Celecoxib treatment suppressed FGF-2 and FGFR-2 expression and decreased MMP-2, MMP-9 and p-ERK expression in the PANC-1 cells." | 5.43 | Celecoxib suppresses fibroblast growth factor-2 expression in pancreatic ductal adenocarcinoma PANC-1 cells. ( Dong, L; Li, J; Luo, M; Shang, B; Wang, Y, 2016) |
" In this study, we show that a novel Mucin-1 (MUC1)-based vaccine in combination with a cyclooxygenase-2 inhibitor (celecoxib), and low-dose chemotherapy (gemcitabine) was effective in preventing the progression of preneoplastic intraepithelial lesions to invasive pancreatic ductal adenocarcinomas." | 3.75 | Progression of pancreatic adenocarcinoma is significantly impeded with a combination of vaccine and COX-2 inhibition. ( Arefayene, M; Basu, GD; Bradley, JM; De Petris, G; Mukherjee, P; Skaar, T; Subramani, DB; Tinder, TL, 2009) |
"Capecitabine was administered at a dose of 1,000 mg/m(2) b." | 2.74 | Capecitabine and celecoxib as second-line treatment of advanced pancreatic and biliary tract cancers. ( Bria, E; Carlini, P; Carpanese, L; Cognetti, F; De Marco, S; Gelibter, A; Milella, M; Nuzzo, C; Pino, MS; Ruggeri, EM; Sperduti, I, 2009) |
"The progression and metastasis of pancreatic ductal adenocarcinoma (PDAC) is highly dependent on the tumour microenvironment." | 1.51 | Tumour cell-derived debris and IgG synergistically promote metastasis of pancreatic cancer by inducing inflammation via tumour-associated macrophages. ( Bai, X; Chen, Q; Chen, Y; Dang, X; Fu, Q; Liang, T; Lou, Y; Wang, J; Wei, T; Yang, J; Ye, M; Zhang, J; Zhang, Q; Zhang, X, 2019) |
"Celecoxib treatment suppressed FGF-2 and FGFR-2 expression and decreased MMP-2, MMP-9 and p-ERK expression in the PANC-1 cells." | 1.43 | Celecoxib suppresses fibroblast growth factor-2 expression in pancreatic ductal adenocarcinoma PANC-1 cells. ( Dong, L; Li, J; Luo, M; Shang, B; Wang, Y, 2016) |
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 0 (0.00) | 18.2507 |
2000's | 4 (57.14) | 29.6817 |
2010's | 3 (42.86) | 24.3611 |
2020's | 0 (0.00) | 2.80 |
Authors | Studies |
---|---|
Chen, Q | 1 |
Wang, J | 1 |
Zhang, Q | 1 |
Zhang, J | 2 |
Lou, Y | 1 |
Yang, J | 1 |
Chen, Y | 1 |
Wei, T | 1 |
Fu, Q | 1 |
Ye, M | 1 |
Zhang, X | 1 |
Dang, X | 1 |
Liang, T | 1 |
Bai, X | 1 |
Li, J | 1 |
Luo, M | 1 |
Wang, Y | 2 |
Shang, B | 1 |
Dong, L | 1 |
Colby, JK | 1 |
Klein, RD | 1 |
McArthur, MJ | 1 |
Conti, CJ | 1 |
Kiguchi, K | 1 |
Kawamoto, T | 1 |
Riggs, PK | 1 |
Pavone, AI | 1 |
Sawicki, J | 1 |
Fischer, SM | 1 |
Mukherjee, P | 1 |
Basu, GD | 1 |
Tinder, TL | 1 |
Subramani, DB | 1 |
Bradley, JM | 1 |
Arefayene, M | 1 |
Skaar, T | 1 |
De Petris, G | 1 |
Pino, MS | 1 |
Milella, M | 1 |
Gelibter, A | 1 |
Sperduti, I | 1 |
De Marco, S | 1 |
Nuzzo, C | 1 |
Bria, E | 1 |
Carpanese, L | 1 |
Ruggeri, EM | 1 |
Carlini, P | 1 |
Cognetti, F | 1 |
Hill, R | 1 |
Li, Y | 1 |
Tran, LM | 1 |
Dry, S | 1 |
Calvopina, JH | 1 |
Garcia, A | 1 |
Kim, C | 1 |
Donahue, TR | 1 |
Herschman, HR | 1 |
Wu, H | 1 |
Gregor, JI | 1 |
Kilian, M | 1 |
Heukamp, I | 1 |
Kiewert, C | 1 |
Kristiansen, G | 1 |
Schimke, I | 1 |
Walz, MK | 1 |
Jacobi, CA | 1 |
Wenger, FA | 1 |
1 trial available for celecoxib and Carcinoma, Pancreatic Ductal
Article | Year |
---|---|
Capecitabine and celecoxib as second-line treatment of advanced pancreatic and biliary tract cancers.
Topics: Adenocarcinoma; Adult; Aged; Antineoplastic Combined Chemotherapy Protocols; Biliary Tract Neoplasms | 2009 |
6 other studies available for celecoxib and Carcinoma, Pancreatic Ductal
Article | Year |
---|---|
Tumour cell-derived debris and IgG synergistically promote metastasis of pancreatic cancer by inducing inflammation via tumour-associated macrophages.
Topics: Animals; Antineoplastic Combined Chemotherapy Protocols; Carcinoma, Pancreatic Ductal; Celecoxib; Ce | 2019 |
Celecoxib suppresses fibroblast growth factor-2 expression in pancreatic ductal adenocarcinoma PANC-1 cells.
Topics: Adenocarcinoma; Apoptosis; Carcinoma, Pancreatic Ductal; Celecoxib; Cell Line, Tumor; Cell Prolifera | 2016 |
Progressive metaplastic and dysplastic changes in mouse pancreas induced by cyclooxygenase-2 overexpression.
Topics: Animals; Biomarkers, Tumor; Carcinoma, Pancreatic Ductal; Celecoxib; Cell Transformation, Neoplastic | 2008 |
Progression of pancreatic adenocarcinoma is significantly impeded with a combination of vaccine and COX-2 inhibition.
Topics: Adenocarcinoma; Animals; Antibodies; Cancer Vaccines; Carcinoma, Pancreatic Ductal; Celecoxib; Cyclo | 2009 |
Cell intrinsic role of COX-2 in pancreatic cancer development.
Topics: Animals; Carcinoma, Pancreatic Ductal; Celecoxib; Cell Membrane; Cyclooxygenase 2; Disease Models, A | 2012 |
Effects of selective COX-2 and 5-LOX inhibition on prostaglandin and leukotriene synthesis in ductal pancreatic cancer in Syrian hamster.
Topics: Animals; Carcinoma, Pancreatic Ductal; Celecoxib; Cricetinae; Cyclooxygenase Inhibitors; Dinoproston | 2005 |