celastrol has been researched along with Osteosarcoma in 4 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 0 (0.00) | 29.6817 |
| 2010's | 4 (100.00) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Chen, Y; He, B; Liu, H; Ou, Y; Tao, Y; Yin, H; Yu, H; Zhao, Z; Zhong, S | 1 |
| Gao, HL; Li, BH; Li, HY; Sun, LL; Xie, T; Ye, ZM; Zhang, J; Zhang, N | 1 |
| Dai, M; Fu, C; Guo, R; Liu, H; Nie, T; Wang, Q; Yu, X; Zhang, B; Zhou, X; Zou, F | 1 |
| Li, Z; Tang, J; Wang, R; Zhang, J | 1 |
4 other study(ies) available for celastrol and Osteosarcoma
| Article | Year |
|---|---|
Effect and mechanisms of celastrol on the apoptosis of HOS osteosarcoma cells.
Topics: Apoptosis; Apoptosis Regulatory Proteins; Autophagy; Biomarkers, Tumor; Bone Neoplasms; Cell Proliferation; eIF-2 Kinase; Endoplasmic Reticulum Stress; Gene Expression Regulation, Neoplastic; Humans; Mitochondria; Osteosarcoma; Pentacyclic Triterpenes; Reactive Oxygen Species; Signal Transduction; Triterpenes; Tumor Cells, Cultured | 2018 |
Celastrol induces apoptosis and autophagy via the ROS/JNK signaling pathway in human osteosarcoma cells: an in vitro and in vivo study.
Topics: Animals; Apoptosis; Autophagy; Caspases; Cell Cycle Checkpoints; Cell Cycle Proteins; Cell Line, Tumor; Cell Proliferation; Enzyme Activation; Female; Fibroblasts; Humans; JNK Mitogen-Activated Protein Kinases; MAP Kinase Signaling System; Mice, Inbred BALB C; Mice, Nude; Mitochondria; Models, Biological; Osteosarcoma; Pentacyclic Triterpenes; Phagosomes; Reactive Oxygen Species; Triterpenes; Xenograft Model Antitumor Assays | 2015 |
Celastrol induces apoptosis of human osteosarcoma cells via the mitochondrial apoptotic pathway.
Topics: Antineoplastic Agents; Apoptosis; Blotting, Western; Bone Neoplasms; Cell Line, Tumor; Cell Proliferation; Cell Survival; Flow Cytometry; Humans; Membrane Potential, Mitochondrial; Mitochondria; Osteosarcoma; Pentacyclic Triterpenes; Triterpenes | 2015 |
Celastrol increases osteosarcoma cell lysis by γδ T cells through up-regulation of death receptors.
Topics: Cell Line, Tumor; Cells, Cultured; Coculture Techniques; Cytotoxicity, Immunologic; Gene Expression Regulation, Neoplastic; Humans; Osteosarcoma; Pentacyclic Triterpenes; Receptors, Antigen, T-Cell, gamma-delta; Receptors, Death Domain; Receptors, TNF-Related Apoptosis-Inducing Ligand; T-Lymphocytes; Triterpenes; Up-Regulation | 2016 |