celastrol has been researched along with Libman-Sacks Disease in 4 studies
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 0 (0.00) | 18.2507 |
2000's | 1 (25.00) | 29.6817 |
2010's | 1 (25.00) | 24.3611 |
2020's | 2 (50.00) | 2.80 |
Authors | Studies |
---|---|
Shi, K; Wang, J; Xiang, G | 1 |
Erqin, D; Hongtao, D; Lei, W; Ningning, Y; Xinqiang, S; Yu, Z | 1 |
Hearth-Holmes, MP; Klassen, LW; Koehn, CD; Li, S; Mikuls, TR; O'Dell, JR; Su, K; Thiele, GM; Yu, Y; Yue, Y; Zhang, Z | 1 |
Huang, XY; Jia, YF; Li, D; Li, H; Sun, YN; Zhang, YY | 1 |
4 other study(ies) available for celastrol and Libman-Sacks Disease
Article | Year |
---|---|
Celastrol alleviates murine lupus nephritis via inducting CD4+Foxp3+ regulatory T cells.
Topics: Animals; Creatinine; Cytokines; Eosine Yellowish-(YS); Forkhead Transcription Factors; Hematoxylin; Immunosuppressive Agents; Lupus Erythematosus, Systemic; Lupus Nephritis; Mice; Pentacyclic Triterpenes; T-Lymphocytes, Regulatory | 2022 |
Molecular mechanism of celastrol in the treatment of systemic lupus erythematosus based on network pharmacology and molecular docking technology.
Topics: Cytokines; Drug Delivery Systems; Humans; Hydrogen Bonding; Lupus Erythematosus, Systemic; Molecular Docking Simulation; Pentacyclic Triterpenes; Receptors, Cytokine; Signal Transduction; Triterpenes | 2020 |
Celastrol inhibits inflammatory stimuli-induced neutrophil extracellular trap formation.
Topics: Arthritis, Rheumatoid; Cells, Cultured; Enzyme Activation; Extracellular Signal-Regulated MAP Kinases; Extracellular Traps; Humans; I-kappa B Proteins; Immunoglobulin G; Inflammation; Intracellular Signaling Peptides and Proteins; Lupus Erythematosus, Systemic; MAP Kinase Kinase Kinases; Neutrophils; NF-KappaB Inhibitor alpha; Ovalbumin; Pentacyclic Triterpenes; Phosphorylation; Protein-Tyrosine Kinases; Respiratory Burst; Syk Kinase; Tripterygium; Triterpenes; Tumor Necrosis Factor-alpha | 2015 |
Beneficial effect of tripterine on systemic lupus erythematosus induced by active chromatin in BALB/c mice.
Topics: Analysis of Variance; Animals; Autoantibodies; Cell Proliferation; Chromatin; Female; Immunoassay; Immunoglobulin G; Interferon-gamma; Interleukin-10; Kidney Glomerulus; Lupus Erythematosus, Systemic; Lymphocytes; Mice; Mice, Inbred BALB C; Nitric Oxide; Pentacyclic Triterpenes; Proteinuria; Spleen; Triterpenes | 2005 |