Page last updated: 2024-09-03

celastrol and Carcinoma, Epidermoid

celastrol has been researched along with Carcinoma, Epidermoid in 6 studies

Research

Studies (6)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's2 (33.33)29.6817
2010's2 (33.33)24.3611
2020's2 (33.33)2.80

Authors

AuthorsStudies
Feng, W; Li, X; Mou, Y; Wang, X; Wang, Y; Zhang, Y1
Ahmad, F; Al-Tamimi, M; Alam, M; Anver, R; Buddenkotte, J; Khan, AQ; M Mateo, J; Raza, SS; Steinhoff, M; Uddin, S1
Pan, J; Tan, F; Tu, Y; Zhou, J1
Brownell, AL; Cai, P; Callaghan, MU; Fribley, AM; Garshott, DM; Kaufman, RJ; Kodali, V; Miller, JR; Narula, N; Reist, TE; Xi, Y; Zeng, Q1
Dannenberg, AJ; Guttenplan, JB; Hughes, D; Marcus, CB; Subbaramaiah, K1
Chen, W; Duan, W; He, D; Xu, Q; Yan, M; Ye, D; Zhang, P; Zhang, Z; Zhong, L; Zhou, X1

Reviews

1 review(s) available for celastrol and Carcinoma, Epidermoid

ArticleYear
The multifaceted mechanisms of pristimerin in the treatment of tumors state-of-the-art.
    Biomedicine & pharmacotherapy = Biomedecine & pharmacotherapie, 2022, Volume: 154

    Topics: Carcinoma, Squamous Cell; Humans; Male; Mouth Neoplasms; Pentacyclic Triterpenes; Triterpenes; Tumor Microenvironment

2022

Other Studies

5 other study(ies) available for celastrol and Carcinoma, Epidermoid

ArticleYear
Pristimerin mediated anticancer effects and sensitization of human skin cancer cells through modulation of MAPK signaling pathways.
    Biomedicine & pharmacotherapy = Biomedecine & pharmacotherapie, 2022, Volume: 156

    Topics: Carcinoma, Squamous Cell; Humans; MAP Kinase Signaling System; Reactive Oxygen Species; Skin Neoplasms

2022
Pristimerin targeting NF-κB pathway inhibits proliferation, migration, and invasion in esophageal squamous cell carcinoma cells.
    Cell biochemistry and function, 2018, Volume: 36, Issue:4

    Topics: Animals; Antineoplastic Agents; Apoptosis; Carcinoma, Squamous Cell; Cell Movement; Cell Proliferation; Cell Survival; Dose-Response Relationship, Drug; Drug Screening Assays, Antitumor; Esophageal Neoplasms; Esophageal Squamous Cell Carcinoma; Humans; Male; Mice; Mice, Nude; Molecular Conformation; Neoplasm Invasiveness; NF-kappa B; Pentacyclic Triterpenes; Structure-Activity Relationship; Triterpenes; Tumor Cells, Cultured

2018
Celastrol induces unfolded protein response-dependent cell death in head and neck cancer.
    Experimental cell research, 2015, Jan-15, Volume: 330, Issue:2

    Topics: Activating Transcription Factor 4; Animals; Apoptosis; Carcinoma, Squamous Cell; Cell Line, Tumor; Cell Proliferation; CHO Cells; Cricetulus; DNA-Binding Proteins; eIF-2 Kinase; Endoplasmic Reticulum Stress; Eukaryotic Initiation Factor-2; Humans; Mice; Mouth Neoplasms; Pentacyclic Triterpenes; Plant Extracts; Regulatory Factor X Transcription Factors; RNA, Messenger; Signal Transduction; Transcription Factor CHOP; Transcription Factors; Tripterygium; Triterpenes; Ubiquitination; Unfolded Protein Response; X-Box Binding Protein 1

2015
Heat shock protein 90 inhibitors suppress aryl hydrocarbon receptor-mediated activation of CYP1A1 and CYP1B1 transcription and DNA adduct formation.
    Cancer prevention research (Philadelphia, Pa.), 2008, Volume: 1, Issue:6

    Topics: Antineoplastic Agents; Aryl Hydrocarbon Hydroxylases; Benzoquinones; Carcinoma, Squamous Cell; Cells, Cultured; Chemoprevention; Cytochrome P-450 CYP1A1; Cytochrome P-450 CYP1B1; DNA Adducts; Drug Evaluation, Preclinical; Enzyme Activation; Gene Expression Regulation, Enzymologic; Head and Neck Neoplasms; HSP90 Heat-Shock Proteins; Humans; Lactams, Macrocyclic; Models, Biological; Pentacyclic Triterpenes; Precancerous Conditions; Receptors, Aryl Hydrocarbon; Transcription, Genetic; Triterpenes

2008
The NF-kappa B inhibitor, celastrol, could enhance the anti-cancer effect of gambogic acid on oral squamous cell carcinoma.
    BMC cancer, 2009, Sep-25, Volume: 9

    Topics: Antineoplastic Agents; Apoptosis; Carcinoma, Squamous Cell; Cell Line, Tumor; Cell Proliferation; Humans; Mouth Neoplasms; NF-kappa B; Pentacyclic Triterpenes; Signal Transduction; Triterpenes; Xanthones

2009