celastrol has been researched along with Carcinoma, Epidermoid in 6 studies
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 0 (0.00) | 18.2507 |
2000's | 2 (33.33) | 29.6817 |
2010's | 2 (33.33) | 24.3611 |
2020's | 2 (33.33) | 2.80 |
Authors | Studies |
---|---|
Feng, W; Li, X; Mou, Y; Wang, X; Wang, Y; Zhang, Y | 1 |
Ahmad, F; Al-Tamimi, M; Alam, M; Anver, R; Buddenkotte, J; Khan, AQ; M Mateo, J; Raza, SS; Steinhoff, M; Uddin, S | 1 |
Pan, J; Tan, F; Tu, Y; Zhou, J | 1 |
Brownell, AL; Cai, P; Callaghan, MU; Fribley, AM; Garshott, DM; Kaufman, RJ; Kodali, V; Miller, JR; Narula, N; Reist, TE; Xi, Y; Zeng, Q | 1 |
Dannenberg, AJ; Guttenplan, JB; Hughes, D; Marcus, CB; Subbaramaiah, K | 1 |
Chen, W; Duan, W; He, D; Xu, Q; Yan, M; Ye, D; Zhang, P; Zhang, Z; Zhong, L; Zhou, X | 1 |
1 review(s) available for celastrol and Carcinoma, Epidermoid
Article | Year |
---|---|
The multifaceted mechanisms of pristimerin in the treatment of tumors state-of-the-art.
Topics: Carcinoma, Squamous Cell; Humans; Male; Mouth Neoplasms; Pentacyclic Triterpenes; Triterpenes; Tumor Microenvironment | 2022 |
5 other study(ies) available for celastrol and Carcinoma, Epidermoid
Article | Year |
---|---|
Pristimerin mediated anticancer effects and sensitization of human skin cancer cells through modulation of MAPK signaling pathways.
Topics: Carcinoma, Squamous Cell; Humans; MAP Kinase Signaling System; Reactive Oxygen Species; Skin Neoplasms | 2022 |
Pristimerin targeting NF-κB pathway inhibits proliferation, migration, and invasion in esophageal squamous cell carcinoma cells.
Topics: Animals; Antineoplastic Agents; Apoptosis; Carcinoma, Squamous Cell; Cell Movement; Cell Proliferation; Cell Survival; Dose-Response Relationship, Drug; Drug Screening Assays, Antitumor; Esophageal Neoplasms; Esophageal Squamous Cell Carcinoma; Humans; Male; Mice; Mice, Nude; Molecular Conformation; Neoplasm Invasiveness; NF-kappa B; Pentacyclic Triterpenes; Structure-Activity Relationship; Triterpenes; Tumor Cells, Cultured | 2018 |
Celastrol induces unfolded protein response-dependent cell death in head and neck cancer.
Topics: Activating Transcription Factor 4; Animals; Apoptosis; Carcinoma, Squamous Cell; Cell Line, Tumor; Cell Proliferation; CHO Cells; Cricetulus; DNA-Binding Proteins; eIF-2 Kinase; Endoplasmic Reticulum Stress; Eukaryotic Initiation Factor-2; Humans; Mice; Mouth Neoplasms; Pentacyclic Triterpenes; Plant Extracts; Regulatory Factor X Transcription Factors; RNA, Messenger; Signal Transduction; Transcription Factor CHOP; Transcription Factors; Tripterygium; Triterpenes; Ubiquitination; Unfolded Protein Response; X-Box Binding Protein 1 | 2015 |
Heat shock protein 90 inhibitors suppress aryl hydrocarbon receptor-mediated activation of CYP1A1 and CYP1B1 transcription and DNA adduct formation.
Topics: Antineoplastic Agents; Aryl Hydrocarbon Hydroxylases; Benzoquinones; Carcinoma, Squamous Cell; Cells, Cultured; Chemoprevention; Cytochrome P-450 CYP1A1; Cytochrome P-450 CYP1B1; DNA Adducts; Drug Evaluation, Preclinical; Enzyme Activation; Gene Expression Regulation, Enzymologic; Head and Neck Neoplasms; HSP90 Heat-Shock Proteins; Humans; Lactams, Macrocyclic; Models, Biological; Pentacyclic Triterpenes; Precancerous Conditions; Receptors, Aryl Hydrocarbon; Transcription, Genetic; Triterpenes | 2008 |
The NF-kappa B inhibitor, celastrol, could enhance the anti-cancer effect of gambogic acid on oral squamous cell carcinoma.
Topics: Antineoplastic Agents; Apoptosis; Carcinoma, Squamous Cell; Cell Line, Tumor; Cell Proliferation; Humans; Mouth Neoplasms; NF-kappa B; Pentacyclic Triterpenes; Signal Transduction; Triterpenes; Xanthones | 2009 |